Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study.
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Dispersion of cardiac action potential duration and the initiation of re-entry: a computational study.Computational Approaches to Understanding the Role of Fibroblast-Myocyte Interactions in Cardiac ArrhythmogenesisModel study of ATP and ADP buffering, transport of Ca(2+) and Mg(2+), and regulation of ion pumps in ventricular myocyteImpairment of Excitation-Contraction Coupling in Right Ventricular Hypertrophied Muscle with Fibrosis Induced by Pulmonary Artery BandingAcute effects of sex steroid hormones on susceptibility to cardiac arrhythmias: a simulation studySimulation of the undiseased human cardiac ventricular action potential: model formulation and experimental validationSimulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failureSex, age, and regional differences in L-type calcium current are important determinants of arrhythmia phenotype in rabbit hearts with drug-induced long QT type 2Cardiac models in drug discovery and development: a review.Bradycardia alters Ca(2+) dynamics enhancing dispersion of repolarization and arrhythmia riskReduced Na⁺ current density underlies impaired propagation in the diabetic rabbit ventricleA computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na+ current.Inherited and acquired vulnerability to ventricular arrhythmias: cardiac Na+ and K+ channels.Subepicardial phase 0 block and discontinuous transmural conduction underlie right precordial ST-segment elevation by a SCN5A loss-of-function mutation.Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutationA Combined Approach Using Patch-Clamp Study and Computer Simulation Study for Understanding Long QT Syndrome and TdP in Women.Reentry in survived subepicardium coupled to depolarized and inexcitable midmyocardium: insights into arrhythmogenesis in ischemia phase 1BContributions of ion channel currents to ventricular action potential changes and induction of early afterdepolarizations during acute hypoxia.Mechanisms for initiation of reentry in acute regional ischemia phase 1B.Increased interstitial loading reduces the effect of microstructural variations in cardiac tissue.Computational biology of cardiac myocytes: proposed standards for the physiome.Mechanisms underlying the antifibrillatory action of hyperkalemia in Guinea pig hearts.A multiscale computational modelling approach predicts mechanisms of female sex risk in the setting of arousal-induced arrhythmias.An integrative model of the cardiac ventricular myocyte incorporating local control of Ca2+ release.Mathematical modeling of physiological systems: an essential tool for discovery.Determinants of excitability in cardiac myocytes: mechanistic investigation of memory effect.Ionic charge conservation and long-term steady state in the Luo-Rudy dynamic cell modelModel of intracellular calcium cycling in ventricular myocytesA computational model of the human left-ventricular epicardial myocyte.Multiphysics simulation of left ventricular filling dynamics using fluid-structure interaction finite element method.Asymmetry in membrane responses to electric shocks: insights from bidomain simulations.A simplified local control model of calcium-induced calcium release in cardiac ventricular myocytesDepolarization of cardiac membrane potential synchronizes calcium sparks and waves in tissueData-based theoretical identification of subcellular calcium compartments and estimation of calcium dynamics in cardiac myocytes.Ischemia-related subcellular redistribution of sodium channels enhances the proarrhythmic effect of class I antiarrhythmic drugs: a simulation study.Computational modeling of inhibition of voltage-gated Ca channels: identification of different effects on uterine and cardiac action potentials.Mechanisms of excitation-contraction coupling in an integrative model of the cardiac ventricular myocyteSpontaneous Ca2+ sparks and Ca2+ homeostasis in a minimal model of permeabilized ventricular myocytes.A 3D Monte Carlo analysis of the role of dyadic space geometry in spark generation.Defibrillation of the heart: insights into mechanisms from modelling studies.
P2860
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P2860
Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study.
description
2000 nî lūn-bûn
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2000 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2000 թվականի մայիսին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
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2000年论文
@wuu
name
Action potential and contractility changes in [Na
@nl
Action potential and contracti ...... myocytes: a simulation study.
@ast
Action potential and contracti ...... myocytes: a simulation study.
@en
type
label
Action potential and contractility changes in [Na
@nl
Action potential and contracti ...... myocytes: a simulation study.
@ast
Action potential and contracti ...... myocytes: a simulation study.
@en
prefLabel
Action potential and contractility changes in [Na
@nl
Action potential and contracti ...... myocytes: a simulation study.
@ast
Action potential and contracti ...... myocytes: a simulation study.
@en
P2860
P1433
P1476
Action potential and contracti ...... myocytes: a simulation study.
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(00)76783-X
P407
P577
2000-05-01T00:00:00Z