Reactive oxygen species produced by NAD(P)H oxidase inhibit apoptosis in pancreatic cancer cells.
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Upsides and downsides of reactive oxygen species for cancer: the roles of reactive oxygen species in tumorigenesis, prevention, and therapyRedox-directed cancer therapeutics: molecular mechanisms and opportunitiesThe pancreatic stellate cell: a star on the rise in pancreatic diseasesGenetics and biology of pancreatic ductal adenocarcinomaReactive Oxygen Species and Targeted Therapy for Pancreatic CancerThe Role of Reactive Oxygen Species in Myelofibrosis and Related NeoplasmsOxidative stress and cytokines in the pathogenesis of pancreatic cancerMechanisms of superoxide signaling in epigenetic processes: relation to aging and cancerNADPH oxidases in lung health and diseaseOxidative stress in cancer and fibrosis: Opportunity for therapeutic intervention with antioxidant compounds, enzymes, and nanoparticlesDNA binding suppresses human AIF-M2 activity and provides a connection between redox chemistry, reactive oxygen species, and apoptosisNADPH oxidase activation is required in reactive oxygen species generation and cell transformation induced by hexavalent chromiumIntracellular coenzymes as natural biomarkers for metabolic activities and mitochondrial anomaliesGeneration of reactive oxygen species by polyenylpyrroles derivatives causes DNA damage leading to G2/M arrest and apoptosis in human oral squamous cell carcinoma cellsSoluble factors from stellate cells induce pancreatic cancer cell proliferation via Nrf2-activated metabolic reprogramming and ROS detoxificationRole of Rac1-dependent NADPH oxidase in the growth of pancreatic cancer.Regulation of pancreatic cancer growth by superoxideSelective detection of superoxide anion radicals generated from macrophages by using a novel fluorescent probe.Nuclear factor (erythroid-derived 2)-like 2 regulates drug resistance in pancreatic cancer cells.MAOA-a novel decision maker of apoptosis and autophagy in hormone refractory neuroendocrine prostate cancer cells.Dual effect of fetal bovine serum on early development depends on stage-specific reactive oxygen species demands in pigs.The quest for selective nox inhibitors and therapeutics: challenges, triumphs and pitfallsNOX enzymes and pulmonary disease.Redox regulation in cancer: a double-edged sword with therapeutic potential.Expression of NADPH oxidase homologues and accessory genes in human cancer cell lines, tumours and adjacent normal tissuesTumorigenic poxviruses up-regulate intracellular superoxide to inhibit apoptosis and promote cell proliferationSTAT3 down regulates LC3 to inhibit autophagy and pancreatic cancer cell growth.Regulation of NADPH oxidase in vascular endothelium: the role of phospholipases, protein kinases, and cytoskeletal proteins.Role of Nox4 and Nox2 in hyperoxia-induced reactive oxygen species generation and migration of human lung endothelial cells.The NADPH oxidase subunit p22phox inhibits the function of the tumor suppressor protein tuberinThe complex landscape of pancreatic cancer metabolismNADPH oxidase NOX4 supports renal tumorigenesis by promoting the expression and nuclear accumulation of HIF2α.Akt kinase mediates the prosurvival effect of smoking compounds in pancreatic ductal cells.AIF suppresses chemical stress-induced apoptosis and maintains the transformed state of tumor cells.NOX4 promotes non-small cell lung cancer cell proliferation and metastasis through positive feedback regulation of PI3K/Akt signaling.Up-regulation and sustained activation of Stat1 are essential for interferon-gamma (IFN-gamma)-induced dual oxidase 2 (Duox2) and dual oxidase A2 (DuoxA2) expression in human pancreatic cancer cell lines.NADPH oxidase-4 mediates protection against chronic load-induced stress in mouse hearts by enhancing angiogenesis.AMP-activated protein kinase (AMPK) negatively regulates Nox4-dependent activation of p53 and epithelial cell apoptosis in diabetes.Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells.NADPH oxidase activation in pancreatic cancer cells is mediated through Akt-dependent up-regulation of p22phox
P2860
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P2860
Reactive oxygen species produced by NAD(P)H oxidase inhibit apoptosis in pancreatic cancer cells.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Reactive oxygen species produced by NAD
@nl
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@ast
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@en
type
label
Reactive oxygen species produced by NAD
@nl
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@ast
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@en
prefLabel
Reactive oxygen species produced by NAD
@nl
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@ast
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@en
P2093
P2860
P356
P1476
Reactive oxygen species produc ...... is in pancreatic cancer cells.
@en
P2093
Anna S Gukovskaya
Eva C Vaquero
Ilya Gukovsky
Mouad Edderkaoui
Stephen J Pandol
P2860
P304
34643-34654
P356
10.1074/JBC.M400078200
P407
P577
2004-05-23T00:00:00Z