Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells. - Wikidata - awgldk - TriplyDB

Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells.

Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells.

http://www.wikidata.org/entity/Q34603387

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NADPH oxidase 4 mediates insulin-stimulated HIF-1α and VEGF expression, and angiogenesis in vitro Adiponectin regulates albuminuria and podocyte function in mice Suppression of NADPH Oxidase Activity May Slow the Expansion of Osteolytic Bone Metastases Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Therapeutic targeting of redox signaling in myofibroblast differentiation and age-related fibrotic disease Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system Cardiac defects contribute to the pathology of spinal muscular atrophy models Oxidant Mechanisms in Renal Injury and Disease Obesity and Diabetic Kidney Disease: Role of Oxidant Stress and Redox Balance Oxidative stress and glutathione in TGF-beta-mediated fibrogenesis Bi-module sensing device to in situ quantitatively detect hydrogen peroxide released from migrating tumor cells Subcellular localization of Nox4 and regulation in diabetes Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice Wild-type and mutant p53 differentially regulate NADPH oxidase 4 in TGF-β-mediated migration of human lung and breast epithelial cells C5a alters blood-brain barrier integrity in experimental lupus.Oxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts.Constant or fluctuating hyperglycemias increases cytomembrane stiffness of human umbilical vein endothelial cells in culture: roles of cytoskeletal rearrangement and nitric oxide synthesis.Stiffness memory of EA.hy926 endothelial cells in response to chronic hyperglycemia Therapeutic targets for treating fibrotic kidney diseases Inhibitory role of the KEAP1-NRF2 pathway in TGFβ1-stimulated renal epithelial transition to fibroblastic cells: a modulatory effect on SMAD signaling.Transforming growth factor-β1 requires NADPH oxidase 4 for angiogenesis in vitro and in vivo Generation of reactive oxygen species in adipose-derived stem cells: friend or foe?Decorin deficiency enhances progressive nephropathy in diabetic mice.Nox4 involvement in TGF-beta and SMAD3-driven induction of the epithelial-to-mesenchymal transition and migration of breast epithelial cells.TGF-β and Hypoxia/Reoxygenation Promote Radioresistance of A549 Lung Cancer Cells through Activation of Nrf2 and EGFR.The Endoplasmic Reticulum Chaperone Calnexin Is a NADPH Oxidase NOX4 Interacting Protein.Impaired pressure natriuresis resulting in salt-sensitive hypertension is caused by tubulointerstitial immune cell infiltration in the kidney Regulation of transforming growth factor beta in diabetic nephropathy: implications for treatment.Vitreous induces heme oxygenase-1 expression mediated by transforming growth factor-beta and reactive oxygen species generation in human retinal pigment epithelial cells Endothelin-1 contributes to the progression of renal injury in sickle cell disease via reactive oxygen species Glutathione and catalase suppress TGFbeta-induced cataract-related changes in cultured rat lenses and lens epithelial explants The angiotensin II-AT1 receptor stimulates reactive oxygen species within the cell nucleus.Trichostatin A, a histone deacetylase inhibitor suppresses NADPH Oxidase 4-Derived Redox Signalling and Angiogenesis Pirfenidone is renoprotective in diabetic kidney disease.N-acetylcysteine inhibits alveolar epithelial-mesenchymal transition.Role of NADPH oxidase in atherosclerosis.Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1.SIRT3 is attenuated in systemic sclerosis skin and lungs, and its pharmacologic activation mitigates organ fibrosis.Myofibroblast differentiation during fibrosis: role of NAD(P)H oxidases.

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P2860

Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells.

http://www.wikidata.org/entity/Q34603387

description

2005 nî lūn-bûn

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2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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Reactive oxygen species produc ...... erations in endothelial cells.

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American Journal of Physiology - Renal Physiology

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Reactive oxygen species produc ...... erations in endothelial cells.

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Barry J Goldstein

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Cathryn Valancius

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Irene Toh

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Kalyankar Mahadev

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Kumar Sharma

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Marilyn Woolkalis

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Satish P Ramachandrarao

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Senthuran Siva

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Taishan Hu

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Yanqing Zhu

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P2860

Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

The NAD(P)H oxidase homolog Nox4 modulates insulin-stimulated generation of H2O2 and plays an integral role in insulin signal transduction

Biochemistry and molecular cell biology of diabetic complications

Homologs of gp91phox: cloning and tissue expression of Nox3, Nox4, and Nox5

Direct interaction of the novel Nox proteins with p22phox is required for the formation of a functionally active NADPH oxidase

Involvement of transforming growth factor-beta in regulation of calcium transients in diabetic vascular smooth muscle cells

Transcriptional control by the TGF-beta/Smad signaling system

How cells read TGF-beta signals

Restricted access chromatographic sample preparation of low mass proteins expressed in human fibroblast cells for proteomics analysis.

Angiotensin II-induced superoxide anion generation in human vascular endothelial cells: role of membrane-bound NADH-/NADPH-oxidases.

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