Altered titin expression, myocardial stiffness, and left ventricular function in patients with dilated cardiomyopathy. - Wikidata - awgldk - TriplyDB

Altered titin expression, myocardial stiffness, and left ventricular function in patients with dilated cardiomyopathy.

Altered titin expression, myocardial stiffness, and left ventricular function in patients with dilated cardiomyopathy.

http://www.wikidata.org/entity/Q34331630

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The muscle ankyrin repeat proteins CARP, Ankrd2, and DARP are not essential for normal cardiac development and function at basal conditions and in response to pressure overload Induction of Ankrd1 in Dilated Cardiomyopathy Correlates with the Heart Failure Progression Mutations in the ANKRD1 gene encoding CARP are responsible for human dilated cardiomyopathy A Review of the Giant Protein Titin in Clinical Molecular Diagnostics of Cardiomyopathies Cardiac thin filament regulation and the Frank-Starling mechanism Titin-based tension in the cardiac sarcomere: molecular origin and physiological adaptations Heart failure with preserved ejection fraction Pathophysiological defects and transcriptional profiling in the RBM20-/- rat model Platform technology for scalable assembly of instantaneously functional mosaic tissues.CARP interacts with titin at a unique helical N2A sequence and at the domain Ig81 to form a structured complex Questioning the preclinical paradigm: natural, extreme biology as an alternative discovery platform What global diastolic function is, what it is not, and how to measure it Sarcomeric protein isoform transitions in cardiac muscle: a journey to heart failure EH-myomesin splice isoform is a novel marker for dilated cardiomyopathy Length-dependent activation is modulated by cardiac troponin I bisphosphorylation at Ser23 and Ser24 but not by Thr143 phosphorylation Small and large animal models in cardiac contraction research: advantages and disadvantages Novex-3, the tiny titin of muscle.Genetic determinants of myocardial dysfunction.Shear Wave Imaging of Passive Diastolic Myocardial Stiffness: Stunned Versus Infarcted Myocardium Single molecule force spectroscopy on titin implicates immunoglobulin domain stability as a cardiac disease mechanism.Mutation that dramatically alters rat titin isoform expression and cardiomyocyte passive tension Modeling structural and functional deficiencies of RBM20 familial dilated cardiomyopathy using human induced pluripotent stem cells Poly(3-hydroxyoctanoate), a promising new material for cardiac tissue engineering.Hypoxia induces dilated cardiomyopathy in the chick embryo: mechanism, intervention, and long-term consequences Titin-mediated control of cardiac myofibrillar function.Experimentally increasing titin compliance in a novel mouse model attenuates the Frank-Starling mechanism but has a beneficial effect on diastole.Transmural heterogeneity of cellular level power output is reduced in human heart failure.Extensible behavior of titin in the miniswine left ventricle.Titin diversity--alternative splicing gone wild Excision of titin's cardiac PEVK spring element abolishes PKCalpha-induced increases in myocardial stiffness Splice variants of enigma homolog, differentially expressed during heart development, promote or prevent hypertrophy.A Tissue Engineered Model of Aging: Interdependence and Cooperative Effects in Failing Tissues.Genotype-specific pathogenic effects in human dilated cardiomyopathy.Physiologic basis and pathophysiologic implications of the diastolic properties of the cardiac muscle Cytosolic CARP promotes angiotensin II- or pressure overload-induced cardiomyocyte hypertrophy through calcineurin accumulation Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction.Cardiac ankyrin repeat protein attenuates cardiac hypertrophy by inhibition of ERK1/2 and TGF-β signaling pathways.More severe cellular phenotype in human idiopathic dilated cardiomyopathy compared to ischemic heart disease.Probing muscle ankyrin-repeat protein (MARP) structure and function Overexpression of ankyrin repeat domain 1 enhances cardiomyocyte apoptosis by promoting p53 activation and mitochondrial dysfunction in rodents.

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P2860

Altered titin expression, myocardial stiffness, and left ventricular function in patients with dilated cardiomyopathy.

http://www.wikidata.org/entity/Q34331630

description

2004 nî lūn-bûn

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2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի հուլիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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type

label

Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Altered titin expression, myoc ...... s with dilated cardiomyopathy.

@nl

prefLabel

Altered titin expression, myoc ...... s with dilated cardiomyopathy.

@ast

Altered titin expression, myoc ...... s with dilated cardiomyopathy.

@en

Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Altered titin expression, myoc ...... s with dilated cardiomyopathy.

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Christian C Witt

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Gopi Shah

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Guillermo Torre-Amione

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Henk L Granzier

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Katy Becker

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Nicholas M P King

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Sherif F Nagueh

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Siegfried Labeit

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Sunshine Lahmers

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Yiming Wu

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