Activation of mitochondrial energy metabolism protects against cardiac failure. - Wikidata - awgldk - TriplyDB

Activation of mitochondrial energy metabolism protects against cardiac failure.

Activation of mitochondrial energy metabolism protects against cardiac failure.

http://www.wikidata.org/entity/Q34424919

about

Transition metals and mitochondrial metabolism in the heart Hormesis in aging and neurodegeneration-a prodigy awaiting dissection Optimization of cardiac metabolism in heart failure Potential biomarkers in mouse myocardium of doxorubicin-induced cardiomyopathy: a metabonomic method and its application Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels RhTFAM treatment stimulates mitochondrial oxidative metabolism and improves memory in aged mice High-fat feeding-induced hyperinsulinemia increases cardiac glucose uptake and mitochondrial function despite peripheral insulin resistance.Mitochondrial Fe-S cluster biogenesis, frataxin and the modulation of susceptibility to drug-induced cardiomyopathy.The functions of cardiolipin in cellular metabolism-potential modifiers of the Barth syndrome phenotype Integromics network meta-analysis on cardiac aging offers robust multi-layer modular signatures and reveals micronome synergism.Doxorubicin and NRG-1/erbB4-Deficiency Affect Gene Expression Profile: Involving Protein Homeostasis in Mouse.Loss of cardiolipin leads to perturbation of mitochondrial and cellular iron homeostasis NRF2 and the Phase II Response in Acute Stress Resistance Induced by Dietary Restriction.Mechanistic or mammalian target of rapamycin (mTOR) may determine robustness in young male mice at the cost of accelerated aging.A proteomic view of isoproterenol induced cardiac hypertrophy: prohibitin identified as a potential biomarker in rats.Mitochondrial hormesis links low-dose arsenite exposure to lifespan extension.Oxidative stress and anti-oxidant enzyme activities in the trophocytes and fat cells of queen honeybees (Apis mellifera)Mitochondria in cardiac hypertrophy and heart failure.Oxidative stress and β-amyloid protein in Alzheimer's disease.Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integration Heme Oxygenase-1 and Carbon Monoxide in the Heart: The Balancing Act Between Danger Signaling and Pro-Survival.Diabetes Increases Cryoinjury Size with Associated Effects on Cx43 Gap Junction Function and Phosphorylation in the Mouse Heart.Cardioprotective mIGF-1/SIRT1 signaling induces hypertension, leukocytosis and fear response in mice Mössbauer Spectra of Mouse Hearts Reveal Age-dependent Changes in Mitochondrial and Ferritin Iron Levels.Overexpression of Drosophila frataxin triggers cell death in an iron-dependent manner.Transcriptional response of skeletal muscle to a low-protein gestation diet in porcine offspring accumulates in growth- and cell cycle-regulating pathways.A Review on the Effect of Traditional Chinese Medicine Against Anthracycline-Induced Cardiac Toxicity.Drosophila melanogaster Models of Friedreich's Ataxia.Frataxin overexpression in Müller cells protects retinal ganglion cells in a mouse model of ischemia/reperfusion injury in vivo.Tissue-dependent changes in oxidative damage with male reproductive effort in house mice

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P2860

Activation of mitochondrial energy metabolism protects against cardiac failure.

http://www.wikidata.org/entity/Q34424919

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Activation of mitochondrial energy metabolism protects against cardiac failure.

@ast

Activation of mitochondrial energy metabolism protects against cardiac failure.

@en

Activation of mitochondrial energy metabolism protects against cardiac failure.

@nl

type

label

Activation of mitochondrial energy metabolism protects against cardiac failure.

@ast

Activation of mitochondrial energy metabolism protects against cardiac failure.

@en

Activation of mitochondrial energy metabolism protects against cardiac failure.

@nl

prefLabel

Activation of mitochondrial energy metabolism protects against cardiac failure.

@ast

Activation of mitochondrial energy metabolism protects against cardiac failure.

@en

Activation of mitochondrial energy metabolism protects against cardiac failure.

@nl

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Activation of mitochondrial energy metabolism protects against cardiac failure.

@en

P2093

Andreas F H Pfeiffer

http://www.w3.org/2001/XMLSchema#string

Anja Voigt

http://www.w3.org/2001/XMLSchema#string

Beate Laube

http://www.w3.org/2001/XMLSchema#string

Carsten Tschöpe

http://www.w3.org/2001/XMLSchema#string

Dirk Westermann

http://www.w3.org/2001/XMLSchema#string

Frank Isken

http://www.w3.org/2001/XMLSchema#string

Kim Zarse

http://www.w3.org/2001/XMLSchema#string

Lutz Schomburg

http://www.w3.org/2001/XMLSchema#string

Tim J Schulz

http://www.w3.org/2001/XMLSchema#string

P2860

Studies of human, mouse and yeast homologues indicate a mitochondrial function for frataxin

How increased oxidative stress promotes longevity and metabolic health: The concept of mitochondrial hormesis (mitohormesis)

Induction of oxidative metabolism by mitochondrial frataxin inhibits cancer growth: Otto Warburg revisited

Adriamycin-induced cardiac damage in the mouse: a small-animal model of cardiotoxicity

Mouse models for Friedreich ataxia exhibit cardiomyopathy, sensory nerve defect and Fe-S enzyme deficiency followed by intramitochondrial iron deposits

Reduced expression of mitochondrial frataxin in mice exacerbates diet-induced obesity

Targeted disruption of hepatic frataxin expression causes impaired mitochondrial function, decreased life span and tumor growth in mice

The failing heart--an engine out of fuel

Insulin-resistant heart exhibits a mitochondrial biogenic response driven by the peroxisome proliferator-activated receptor-alpha/PGC-1alpha gene regulatory pathway.

Doxorubicin cardiotoxicity may be caused by its metabolite, doxorubicinol.

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843-853

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scholarly article

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10.18632/AGING.100234

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11

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2

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René Thierbach

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2010-11-01T00:00:00Z

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21084725

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3006026

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