Endogenous human papillomavirus E6 and E7 proteins differentially regulate proliferation, senescence, and apoptosis in HeLa cervical carcinoma cells
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Immortalization of human mammary epithelial cells is associated with inactivation of the p14ARF-p53 pathwayThe E7 oncoprotein is translated from spliced E6*I transcripts in high-risk human papillomavirus type 16- or type 18-positive cervical cancer cell lines via translation reinitiationPresence of papillomavirus sequences in condylomatous lesions of the mamillae and in invasive carcinoma of the breast.Noninvasive assessment of mitochondrial organization in three-dimensional tissues reveals changes associated with cancer development.Sonoporation delivery of monoclonal antibodies against human papillomavirus 16 E6 restores p53 expression in transformed cervical keratinocytesFurther Stimulation of Cellular Immune Responses through Association of HPV-16 E6, E7 and L1 Genes in order to produce more Effective Therapeutic DNA Vaccines in Cervical Cancer ModelImproved assays for determining the cytosolic access of peptides, proteins, and their mimetics.Analysis of The Cancer Genome Atlas sequencing data reveals novel properties of the human papillomavirus 16 genome in head and neck squamous cell carcinoma.Depletion of the cdk inhibitor p16INK4a differentially affects proliferation of established cervical carcinoma cells.The mRNA decay factor tristetraprolin (TTP) induces senescence in human papillomavirus-transformed cervical cancer cells by targeting E6-AP ubiquitin ligase.The potential role of infectious agents and pelvic inflammatory disease in ovarian carcinogenesisGenome-wide siRNA screen identifies SMCX, EP400, and Brd4 as E2-dependent regulators of human papillomavirus oncogene expressionThe role of viral and bacterial pathogens in gastrointestinal cancer.Fowlpox virus recombinants expressing HPV-16 E6 and E7 oncogenes for the therapy of cervical carcinoma elicit humoral and cell-mediated responses in rabbits.Distinct human papillomavirus type 16 methylomes in cervical cells at different stages of premalignancy.Lessons from Australia: human papillomavirus is not a major risk factor for esophageal squamous cell carcinoma.Radiation pharmacogenomics: a genome-wide association approach to identify radiation response biomarkers using human lymphoblastoid cell linesInactivation of the human papillomavirus E6 or E7 gene in cervical carcinoma cells by using a bacterial CRISPR/Cas RNA-guided endonuclease.Highly potent and specific siRNAs against E6 or E7 genes of HPV16- or HPV18-infected cervical cancersPersistence of high-grade cervical dysplasia and cervical cancer requires the continuous expression of the human papillomavirus type 16 E7 oncogene.miR-29 and miR-30 regulate B-Myb expression during cellular senescence.RBP1 family proteins exhibit SUMOylation-dependent transcriptional repression and induce cell growth inhibition reminiscent of senescence.Vaccination of rabbits with an adenovirus vector expressing the papillomavirus E2 protein leads to clearance of papillomas and infection.Regulation of apoptosis by the papillomavirus E6 oncogene.Complementation for an essential ancillary non-structural protein function across parvovirus genera.Docosahexaenoic acid induces the degradation of HPV E6/E7 oncoproteins by activating the ubiquitin-proteasome system.Analysis of mutations in the E6 oncogene of human papillomavirus 16 in cervical cancer isolates from Moroccan women.BiP and multiple DNAJ molecular chaperones in the endoplasmic reticulum are required for efficient simian virus 40 infection.Antiproliferative activity of methanolic extracts from two green algae, Enteromorpha intestinalis and Rizoclonium riparium on HeLa cells.PIK3CA-mediated PI3-kinase signalling is essential for HPV-induced transformation in vitroSmall molecule inhibitors of human papillomavirus protein - protein interactions.Re-expression of HPV16 E2 in SiHa (human cervical cancer) cells potentiates NF-κB activation induced by TNF-α concurrently increasing senescence and survivalTranscriptome signature of irreversible senescence in human papillomavirus-positive cervical cancer cellsB-Myb, cancer, senescence, and microRNAs.Immunization with mutant HPV16 E7 protein inhibits the growth of TC-1 cells in tumor-bearing mice.Dependence of intracellular and exosomal microRNAs on viral E6/E7 oncogene expression in HPV-positive tumor cells.Targeting the human papillomavirus E6 and E7 oncogenes through expression of the bovine papillomavirus type 1 E2 protein stimulates cellular motility.Modulation of apoptosis by human papillomavirus (HPV) oncoproteins.Primary human cervical carcinoma cells require human papillomavirus E6 and E7 expression for ongoing proliferation.Molecular Mechanism and Potential Targets for Blocking HPV-Induced Lesion Development
P2860
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P2860
Endogenous human papillomavirus E6 and E7 proteins differentially regulate proliferation, senescence, and apoptosis in HeLa cervical carcinoma cells
description
2003 nî lūn-bûn
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2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年學術文章
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name
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@ast
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@en
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@nl
type
label
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@ast
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@en
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@nl
prefLabel
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@ast
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@en
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@nl
P2093
P2860
P1433
P1476
Endogenous human papillomaviru ...... HeLa cervical carcinoma cells
@en
P2093
Daniel DiMaio
Edward C Goodwin
Lingling Wu
Rosa Anna DeFilippis
P2860
P304
P356
10.1128/JVI.77.2.1551-1563.2003
P407
P577
2003-01-01T00:00:00Z