Mcl-1 overexpression in hepatocellular carcinoma: a potential target for antisense therapy. - Wikidata - awgldk - TriplyDB

Mcl-1 overexpression in hepatocellular carcinoma: a potential target for antisense therapy.

Mcl-1 overexpression in hepatocellular carcinoma: a potential target for antisense therapy.

http://www.wikidata.org/entity/Q34467880

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Role of bile salt in regulating Mcl-1 phosphorylation and chemoresistance in hepatocellular carcinoma cells Targeting Cell Survival Proteins for Cancer Cell Death Targeting the Bcl-2 family for cancer therapy Targeting Mcl-1 for the therapy of cancer Hepatitis C Virus Proteins Modulate MicroRNA Expression and Chemosensitivity in Malignant Hepatocytes A novel BH3 ligand that selectively targets Mcl-1 reveals that apoptosis can proceed without Mcl-1 degradation Apoptotic Regulation by MCL-1 through Heterodimerization Mitochondrial apoptosis and BH3 mimetics Regulation of Mcl-1 by SRSF1 and SRSF5 in cancer cells Pan-Bcl-2 inhibitor obatoclax delays cell cycle progression and blocks migration of colorectal cancer cells Suppression of Mcl-1 via RNA interference sensitizes human hepatocellular carcinoma cells towards apoptosis induction.3-Substituted-N-(4-hydroxynaphthalen-1-yl)arylsulfonamides as a novel class of selective Mcl-1 inhibitors: structure-based design, synthesis, SAR, and biological evaluation.Hedgehog inhibition reduces angiogenesis by downregulation of tumoral VEGF-A expression in hepatocellular carcinoma.Hepatocyte IKKbeta/NF-kappaB inhibits tumor promotion and progression by preventing oxidative stress-driven STAT3 activation Anti-apoptotic effects of osteopontin through the up-regulation of Mcl-1 in gastrointestinal stromal tumors.Targeting MCL-1 sensitizes human esophageal squamous cell carcinoma cells to cisplatin-induced apoptosis Hepatocyte-specific deletion of the antiapoptotic protein myeloid cell leukemia-1 triggers proliferation and hepatocarcinogenesis in mice.Therapeutics Based on microRNA: A New Approach for Liver Cancer Correlation analysis of liver tumor-associated genes with liver regeneration Synthesis and evaluation of substituted hexahydronaphthalenes as novel inhibitors of the Mcl-1/BimBH3 interaction Overexpression of Mcl-1L splice variant is associated with poor prognosis and chemoresistance in oral cancers.The stress protein BAG3 stabilizes Mcl-1 protein and promotes survival of cancer cells and resistance to antagonist ABT-737 Synergistic interaction between the HDAC inhibitor, MPT0E028, and sorafenib in liver cancer cells in vitro and in vivo HBx sensitizes cells to oxidative stress-induced apoptosis by accelerating the loss of Mcl-1 protein via caspase-3 cascade Sorafenib sensitizes solid tumors to Apo2L/TRAIL and Apo2L/TRAIL receptor agonist antibodies by the Jak2-Stat3-Mcl1 axis Cytosolic and nuclear caspase-8 have opposite impact on survival after liver resection for hepatocellular carcinoma.A New Player in the Development of TRAIL Based Therapies for Hepatocarcinoma Treatment: ATM Kinase.Branched-chain amino acids ameliorate fibrosis and suppress tumor growth in a rat model of hepatocellular carcinoma with liver cirrhosis.Identification of SLAMF3 (CD229) as an inhibitor of hepatocellular carcinoma cell proliferation and tumour progression.Regulation of Mcl-1 by constitutive activation of NF-κB contributes to cell viability in human esophageal squamous cell carcinoma cells.Mitochondrion-dependent N-terminal processing of outer membrane Mcl-1 protein removes an essential Mule/Lasu1 protein-binding site.Dihydroartemisinin induces apoptosis preferentially via a Bim-mediated intrinsic pathway in hepatocarcinoma cells.Enhanced expression of cohesin loading factor NIPBL confers poor prognosis and chemotherapy resistance in non-small cell lung cancer Reduced ubiquitin-specific protease 9X expression induced by RNA interference inhibits the bioactivity of hepatocellular carcinoma cells Cell death and cell death responses in liver disease: mechanisms and clinical relevance Deubiquitinase USP9x confers radioresistance through stabilization of Mcl-1 Hepatitis C virus and cellular stress response: implications to molecular pathogenesis of liver diseases.Mcl-1 involvement in mitochondrial dynamics is associated with apoptotic cell death Overexpression of Mcl-1 confers resistance to BRAFV600E inhibitors alone and in combination with MEK1/2 inhibitors in melanoma.Mcl-1 and Bcl-xL cooperatively maintain integrity of hepatocytes in developing and adult murine liver

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Mcl-1 overexpression in hepatocellular carcinoma: a potential target for antisense therapy.

http://www.wikidata.org/entity/Q34467880

description

2005 nî lūn-bûn

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2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Mcl-1 overexpression in hepato ...... target for antisense therapy.

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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type

label

Mcl-1 overexpression in hepato ...... target for antisense therapy.

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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prefLabel

Mcl-1 overexpression in hepato ...... target for antisense therapy.

@ast

Mcl-1 overexpression in hepato ...... target for antisense therapy.

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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J.JHEP.2005.09.010

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Journal of Hepatology

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Mcl-1 overexpression in hepato ...... target for antisense therapy.

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P2093

Brett P Monia

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Daniel Cejka

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Doris Losert

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Katharina Schmid

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Martin Bodingbauer

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Richard Crevenna

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Sabine Strommer

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Susanne Rasoul-Rockenschaub

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Volker Wacheck

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Wolfgang Sieghart

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151-157

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scholarly article

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10.1016/J.JHEP.2005.09.010

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1

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44

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Markus Peck-Radosavljevic

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2005-10-25T00:00:00Z

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16289418

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