Colorectal cancer intrinsic subtypes predict chemotherapy benefit, deficient mismatch repair and epithelial-to-mesenchymal transition - Wikidata - awgldk - TriplyDB

Colorectal cancer intrinsic subtypes predict chemotherapy benefit, deficient mismatch repair and epithelial-to-mesenchymal transition

Colorectal cancer intrinsic subtypes predict chemotherapy benefit, deficient mismatch repair and epithelial-to-mesenchymal transition

http://www.wikidata.org/entity/Q34522228

about

Molecular pathological classification of colorectal cancer Molecular Subtypes and Personalized Therapy in Metastatic Colorectal Cancer Treatment Individualization in Colorectal Cancer Clinical development of galunisertib (LY2157299 monohydrate), a small molecule inhibitor of transforming growth factor-beta signaling pathway Colorectal Cancer Classification and Cell Heterogeneity: A Systems Oncology Approach Translating genomic profiling to gastrointestinal cancer treatment.Imatinib treatment of poor prognosis mesenchymal-type primary colon cancer: a proof-of-concept study in the preoperative window period (ImPACCT).Prediction of overall survival in stage II and III colon cancer beyond TNM system: a retrospective, pooled biomarker study.EMT and inflammation: inseparable actors of cancer progression.Prognosis of stage III colorectal carcinomas with FOLFOX adjuvant chemotherapy can be predicted by molecular subtype Molecular landscape and sub-classification of gastrointestinal cancers: a review of literature.Epithelial-mesenchymal transition and somatic alteration in colorectal cancer with and without peritoneal carcinomatosis A 5-gene classifier from the carcinoma-associated fibroblast transcriptomic profile and clinical outcome in colorectal cancer.How Can Next-Generation Sequencing (Genomics) Help Us in Treating Colorectal Cancer?Emerging understanding of multiscale tumor heterogeneity Implications of epithelial-mesenchymal plasticity for heterogeneity in colorectal cancer.Cancer in silico drug discovery: a systems biology tool for identifying candidate drugs to target specific molecular tumor subtypes Altered pathways and colorectal cancer prognosis Impact of the Choice of Normalization Method on Molecular Cancer Class Discovery Using Nonnegative Matrix Factorization.Exome Sequencing Reveals AMER1 as a Frequently Mutated Gene in Colorectal Cancer The consensus molecular subtypes of colorectal cancer MicroRNA-mRNA interactions underlying colorectal cancer molecular subtypes.Fra-1 is a key driver of colon cancer metastasis and a Fra-1 classifier predicts disease-free survival.Molecular Biomarkers in the Personalized Treatment of Colorectal Cancer.A multigene mutation classification of 468 colorectal cancers reveals a prognostic role for APC.Genomic markers of panitumumab resistance including ERBB2/ HER2 in a phase II study of KRAS wild-type (wt) metastatic colorectal cancer (mCRC).A multidimensional network approach reveals microRNAs as determinants of the mesenchymal colorectal cancer subtype.Twist1-induced epithelial-mesenchymal transition according to microsatellite instability status in colon cancer cells Transcriptional upregulation of c-MET is associated with invasion and tumor budding in colorectal cancer.Immune regulation by low doses of the DNA methyltransferase inhibitor 5-azacitidine in common human epithelial cancers.Accuracy of TNM staging in colorectal cancer: a review of current culprits, the modern role of morphology and stepping-stones for improvements in the molecular era.Hurdles on the road to personalized medicine.Personalized treatment for colorectal cancer: novel developments and putative therapeutic strategies.Personalized treatment for patients with colorectal cancer: role of biomarkers.Clinical Outcome From Oxaliplatin Treatment in Stage II/III Colon Cancer According to Intrinsic Subtypes: Secondary Analysis of NSABP C-07/NRG Oncology Randomized Clinical Trial.The rationale for liquid biopsy in colorectal cancer: a focus on circulating tumor cells.Current controversies in the management of metastatic colorectal cancer.FOXO3a and the MAPK p38 are activated by cetuximab to induce cell death and inhibit cell proliferation and their expression predicts cetuximab efficacy in colorectal cancer.Translating colorectal cancer genetics into clinically useful biomarkers.Collagen-rich stroma in aggressive colon tumors induces mesenchymal gene expression and tumor cell invasion.

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Colorectal cancer intrinsic subtypes predict chemotherapy benefit, deficient mismatch repair and epithelial-to-mesenchymal transition

http://www.wikidata.org/entity/Q34522228

description

2013 nî lūn-bûn

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2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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type

label

Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

@nl

prefLabel

Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

@ast

Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

@en

Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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International Journal of Cancer

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Colorectal cancer intrinsic su ...... lial-to-mesenchymal transition

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Andreas Schlicker

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Christine M Chresta

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George Orphanides

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Iris M Simon

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Lodewyk F A Wessels

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Mireille H Snel

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Paul Roepman

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Ramon Salazar

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Robert Rosenberg

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Sun Tian

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P2860

Ubiquitous somatic mutations in simple repeated sequences reveal a new mechanism for colonic carcinogenesis

Tumor microsatellite-instability status as a predictor of benefit from fluorouracil-based adjuvant chemotherapy for colon cancer

Comprehensive molecular characterization of human colon and rectal cancer

A genetic model for colorectal tumorigenesis

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

Lessons from hereditary colorectal cancer

AZD2171: a highly potent, orally bioavailable, vascular endothelial growth factor receptor-2 tyrosine kinase inhibitor for the treatment of cancer

The role of chemotherapy in microsatellite unstable (MSI-H) colorectal cancer

EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

Methylation of the hMLH1 promoter correlates with lack of expression of hMLH1 in sporadic colon tumors and mismatch repair-defective human tumor cell lines

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552-562

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10.1002/IJC.28387

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3

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134

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Josep Tabernero

Gabriel Capella

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2013-09-13T00:00:00Z

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249321535

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23852808

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colorectal cancer

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4234005

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