Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression. - Wikidata - awgldk - TriplyDB

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

http://www.wikidata.org/entity/Q34574452

about

The T-box gene family: emerging roles in development, stem cells and cancer Control of Hes7 expression by Tbx6, the Wnt pathway and the chemical Gsk3 inhibitor LiCl in the mouse segmentation clock A multi-cell, multi-scale model of vertebrate segmentation and somite formation The roles of Mesp family proteins: functional diversity and redundancy in differentiation of pluripotent stem cells and mammalian mesodermal development Tbx6, Mesp-b and Ripply1 regulate the onset of skeletal myogenesis in zebrafish Differentiation of pluripotent stem cells to muscle fiber to model Duchenne muscular dystrophy Mammalian TBX1 preferentially binds and regulates downstream targets via a tandem T-site repeat Tbx protein level critical for clock-mediated somite positioning is regulated through interaction between Tbx and Ripply Ciona intestinalis as a model for cardiac development.Wnt-regulated dynamics of positional information in zebrafish somitogenesis.Signaling gradients during paraxial mesoderm development.LTRs of Endogenous Retroviruses as a Source of Tbx6 Binding Sites.Autosomal dominant spondylocostal dysostosis is caused by mutation in TBX6.Scoliosis and segmentation defects of the vertebrae.A novel gain-of-function mutation of the proneural IRX1 and IRX2 genes disrupts axis elongation in the Araucana rumpless chicken.Tbx6-dependent Sox2 regulation determines neural or mesodermal fate in axial stem cells.Noncyclic Notch activity in the presomitic mesoderm demonstrates uncoupling of somite compartmentalization and boundary formation.Divergent mechanisms regulate conserved cardiopharyngeal development and gene expression in distantly related ascidians.Novel ENU-Induced Mutation in Tbx6 Causes Dominant Spondylocostal Dysostosis-Like Vertebral Malformations in the Rat.A genetic screen for modifiers of the delta1-dependent notch signaling function in the mouse.The synchrony and cyclicity of developmental events Bone cell interactions through Eph/ephrin: bone modeling, remodeling and associated diseases.Activator-to-repressor conversion of T-box transcription factors by the Ripply family of Groucho/TLE-associated mediators From segment to somite: segmentation to epithelialization analyzed within quantitative frameworks.Dynamic CREB family activity drives segmentation and posterior polarity specification in mammalian somitogenesis EphrinB2 coordinates the formation of a morphological boundary and cell epithelialization during somite segmentation.Teasing out T-box targets in early mesoderm Smad6 inhibits the transcriptional activity of Tbx6 by mediating its degradation.Progress and perspective of TBX6 gene in congenital vertebral malformations.Congenic fine-mapping identifies a major causal locus for variation in the native collateral circulation and ischemic injury in brain and lower extremity.The mouse notches up another success: understanding the causes of human vertebral malformation.A fluorescence spotlight on the clockwork development and metabolism of bone.Oscillatory gene expression and somitogenesis.Signalling dynamics in vertebrate segmentation.A cis-regulatory module upstream of deltaC regulated by Ntla and Tbx16 drives expression in the tailbud, presomitic mesoderm and somites Molecular mechanism for cyclic generation of somites: Lessons from mice and zebrafish.Molecular diagnosis of vertebral segmentation disorders in humans.Response to Lefebvre et al.Supt20 is required for development of the axial skeleton.Compound heterozygous mutations in RIPPLY2 associated with vertebral segmentation defects.

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P2860

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

http://www.wikidata.org/entity/Q34574452

description

2006 nî lūn-bûn

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2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

name

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@ast

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@en

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@nl

type

label

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@ast

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@en

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@nl

prefLabel

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@ast

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@en

Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

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Tbx6-mediated Notch signaling controls somite-specific Mesp2 expression.

@en

P2093

Jun Kanno

http://www.w3.org/2001/XMLSchema#string

Satoshi Kitajima

http://www.w3.org/2001/XMLSchema#string

Seiki Haraguchi

http://www.w3.org/2001/XMLSchema#string

Yu Takahashi

http://www.w3.org/2001/XMLSchema#string

Yukuto Yasuhiko

http://www.w3.org/2001/XMLSchema#string

Yumiko Saga

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P2860

The T-box family

Crystallographic structure of the T domain-DNA complex of the Brachyury transcription factor

Notch signaling: from the outside in

A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

Characterization of size-fractionated cDNA libraries generated by the in vitro recombination-assisted method

Notch-1 signalling requires ligand-induced proteolytic release of intracellular domain

Signalling downstream of activated mammalian Notch

The retinoic acid-inactivating enzyme CYP26 is essential for establishing an uneven distribution of retinoic acid along the anterio-posterior axis within the mouse embryo

The Mesp2 transcription factor establishes segmental borders by suppressing Notch activity

Mga, a dual-specificity transcription factor that interacts with Max and contains a T-domain DNA-binding motif.

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3651-3656

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10

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103

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16505380

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1450137

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