Acetylation of MEK2 and I kappa B kinase (IKK) activation loop residues by YopJ inhibits signaling.
about
Cell death programs in Yersinia immunity and pathogenesisA Pseudomonas aeruginosa toxin that hijacks the host ubiquitin proteolytic systemStructural requirements for Yersinia YopJ inhibition of MAP kinase pathwaysMetabolism, cytoskeleton and cellular signalling in the grip of protein Nepsilon - and O-acetylationRo52-mediated monoubiquitination of IKK{beta} down-regulates NF-{kappa}B signallingThe acetyltransferase activity of the bacterial toxin YopJ of Yersinia is activated by eukaryotic host cell inositol hexakisphosphateYersinia virulence factors - a sophisticated arsenal for combating host defencesYersinia type III effectors perturb host innate immune responsesSubverting Toll-Like Receptor Signaling by Bacterial PathogensMLL5 maintains spindle bipolarity by preventing aberrant cytosolic aggregation of PLK1Proteomic discovery of host kinase signaling in bacterial infectionsModulation of the host innate immune and inflammatory response by translocated bacterial proteinsThe effects of anthrax lethal toxin on host barrier functionAnthrax lethal toxin disrupts intestinal barrier function and causes systemic infections with enteric bacteriaAvrBsT acetylates Arabidopsis ACIP1, a protein that associates with microtubules and is required for immunityModulation of NF-κB signalling by microbial pathogensThe intrinsic disorder alphabet. III. Dual personality of serine.Cytotoxic necrotizing factor-Y boosts Yersinia effector translocation by activating Rac protein.Innate immune recognition of Yersinia pseudotuberculosis type III secretion.Reduced secretion of YopJ by Yersinia limits in vivo cell death but enhances bacterial virulence.Yersinia pestis endowed with increased cytotoxicity is avirulent in a bubonic plague model and induces rapid protection against pneumonic plague.Selective inhibition of type III secretion activated signaling by the Salmonella effector AvrA.Neutrophils are resistant to Yersinia YopJ/P-induced apoptosis and are protected from ROS-mediated cell death by the type III secretion systemAgent-based modeling of endotoxin-induced acute inflammatory response in human blood leukocytes.Allele-specific virulence attenuation of the Pseudomonas syringae HopZ1a type III effector via the Arabidopsis ZAR1 resistance protein.Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.Yersinia virulence factor YopM induces sustained RSK activation by interfering with dephosphorylation.Autoacetylation of the Ralstonia solanacearum effector PopP2 targets a lysine residue essential for RRS1-R-mediated immunity in Arabidopsis.YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infectionA Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophagesUnrestrictive identification of non-phosphorylation PTMs in yeast kinases by MS and PTMap.Delineation of regions of the Yersinia YopM protein required for interaction with the RSK1 and PRK2 host kinases and their requirement for interleukin-10 production and virulenceA bacterial acetyltransferase destroys plant microtubule networks and blocks secretionThe Listeria monocytogenes InlC protein interferes with innate immune responses by targeting the I{kappa}B kinase subunit IKK{alpha}.YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense.Translocated effectors of Yersinia.Bacterial effector activates jasmonate signaling by directly targeting JAZ transcriptional repressors.c-KIT signaling is targeted by pathogenic Yersinia to suppress the host immune response.Post-translational modifications are key players of the Legionella pneumophila infection strategyHost response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways.
P2860
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P2860
Acetylation of MEK2 and I kappa B kinase (IKK) activation loop residues by YopJ inhibits signaling.
description
2006 nî lūn-bûn
@nan
2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
name
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@ast
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@en
Acetylation of MEK2 and I kappa B kinase
@nl
type
label
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@ast
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@en
Acetylation of MEK2 and I kappa B kinase
@nl
prefLabel
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@ast
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@en
Acetylation of MEK2 and I kappa B kinase
@nl
P2093
P2860
P356
P1476
Acetylation of MEK2 and I kapp ...... es by YopJ inhibits signaling.
@en
P2093
Harvey T McMahon
Rohit Mittal
Sew-Yeu Peak-Chew
P2860
P304
18574-18579
P356
10.1073/PNAS.0608995103
P407
P577
2006-11-20T00:00:00Z