Reducing the fitness cost of antibiotic resistance by amplification of initiator tRNA genes. - Wikidata - awgldk - TriplyDB

Reducing the fitness cost of antibiotic resistance by amplification of initiator tRNA genes.

Reducing the fitness cost of antibiotic resistance by amplification of initiator tRNA genes.

http://www.wikidata.org/entity/Q34598420

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The tandem inversion duplication in Salmonella enterica: selection drives unstable precursors to final mutation types Mutations in MTFMT underlie a human disorder of formylation causing impaired mitochondrial translation Lateral transfer of genes and gene fragments in Staphylococcus extends beyond mobile elements Costs of antibiotic resistance - separating trait effects and selective effects Adaptive copy number evolution in malaria parasites Fmt bypass in Pseudomonas aeruginosa causes induction of MexXY efflux pump expression Role of 16S ribosomal RNA methylations in translation initiation in Escherichia coli Reduced susceptibility of Haemophilus influenzae to the peptide deformylase inhibitor LBM415 can result from target protein overexpression due to amplified chromosomal def gene copy number.Cj0440c Affects Flagella Formation and In Vivo Colonization of Erythromycin-Susceptible and -Resistant Campylobacter jejuni Reversibility of antibiotic resistance Challenges of antibacterial discovery A tradeoff drives the evolution of reduced metal resistance in natural populations of yeast Origins of a 350-kilobase genomic duplication in Mycobacterium tuberculosis and its impact on virulence Biochemical characterization of pathogenic mutations in human mitochondrial methionyl-tRNA formyltransferase Extensive amplification of GI-VII-6, a multidrug resistance genomic island of Salmonella enterica serovar Typhimurium, increases resistance to extended-spectrum cephalosporins Multiple pathways of selected gene amplification during adaptive mutation.A Comparison of Methods to Measure Fitness in Escherichia coli.Caenorhabditis elegans as a model to determine fitness of antibiotic-resistant Salmonella enterica serovar typhimurium.A fitness cost associated with the antibiotic resistance enzyme SME-1 beta-lactamase A longitudinal study of antimicrobial resistant faecal bacteria in sediments collected from a hospital wastewater system Drug forecast - the peptide deformylase inhibitors as antibacterial agents.Antimicrobial resistance and virulence: a successful or deleterious association in the bacterial world?Plasmid selection in Escherichia coli using an endogenous essential gene marker.Double trouble: medical implications of genetic duplication and amplification in bacteria.Mutations in three distinct loci cause resistance to peptide deformylase inhibitors in Bacillus subtilis.A unique peptide deformylase platform to rationally design and challenge novel active compounds.Formyl-methionine as a degradation signal at the N-termini of bacterial proteins.The Origin of Mutants Under Selection: How Natural Selection Mimics Mutagenesis (Adaptive Mutation)Lack of formylated methionyl-tRNA has pleiotropic effects on Bacillus subtilis.FabR regulates Salmonella biofilm formation via its direct target FabB.Error-prone initiation factor 2 mutations reduce the fitness cost of antibiotic resistance.A novel gene amplification causes upregulation of the PatAB ABC transporter and fluoroquinolone resistance in Streptococcus pneumoniae Frequency of Spontaneous Resistance to Peptide Deformylase Inhibitor GSK1322322 in Haemophilus influenzae, Staphylococcus aureus, Streptococcus pyogenes, and Streptococcus pneumoniae.Staphylococcus aureus formyl-methionyl transferase mutants demonstrate reduced virulence factor production and pathogenicity How many initiator tRNA genes does Escherichia coli need?Crucial contribution of the multiple copies of the initiator tRNA genes in the fidelity of tRNA(fMet) selection on the ribosomal P-site in Escherichia coli Ligand and Structure-Based Approaches for the Identification of Peptide Deformylase Inhibitors as Antibacterial Drugs.The joys and terrors of fast adaptation: new findings elucidate antibiotic resistance and natural selection.Contribution of gene amplification to evolution of increased antibiotic resistance in Salmonella typhimurium.No fitness cost of glyphosate resistance endowed by massive EPSPS gene amplification in Amaranthus palmeri.

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P2860

Reducing the fitness cost of antibiotic resistance by amplification of initiator tRNA genes.

http://www.wikidata.org/entity/Q34598420

description

2006 nî lūn-bûn

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2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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PNAS.0602171103

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Proceedings of the National Academy of Sciences of the United States of America

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Reducing the fitness cost of a ...... ation of initiator tRNA genes.

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Anna Kanth

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Anna Zorzet

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Annika I Nilsson

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Dan I Andersson

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Otto G Berg

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Sabina Dahlström

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P2860

Tight regulation, modulation, and high-level expression by vectors containing the arabinose PBAD promoter

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

The evolutionary fate and consequences of duplicate genes

Peptide deformylase in Staphylococcus aureus: resistance to inhibition is mediated by mutations in the formyltransferase gene

The role of peptide deformylase in protein biosynthesis: a proteomic study.

The biological cost of antibiotic resistance.

Evolutionary preservation of redundant duplicated genes.

Actinonin, a naturally occurring antibacterial agent, is a potent deformylase inhibitor.

Peptide deformylase as a target for new generation, broad spectrum antimicrobial agents.

Fusidic acid-resistant EF-G perturbs the accumulation of ppGpp.

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6976-6981

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10.1073/PNAS.0602171103

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18

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103

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7145103

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16636273

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antibiotic resistance

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1459004

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