Transcription increases the deletion frequency of long CTG.CAG triplet repeats from plasmids in Escherichia coli. - Wikidata - awgldk - TriplyDB

Transcription increases the deletion frequency of long CTG.CAG triplet repeats from plasmids in Escherichia coli.

Transcription increases the deletion frequency of long CTG.CAG triplet repeats from plasmids in Escherichia coli.

http://www.wikidata.org/entity/Q34631670

about

Engineering BioBrick vectors from BioBrick parts New insights into repeat instability: role of RNA•DNA hybrids Processing of double-R-loops in (CAG)·(CTG) and C9orf72 (GGGGCC)·(GGCCCC) repeats causes instability.Diverse effects of individual mismatch repair components on transcription-induced CAG repeat instability in human cells.R loops stimulate genetic instability of CTG.CAG repeats.Methylation mosaicism of 5'-(CGG)(n)-3' repeats in fragile X, premutation and normal individuals.Lack of consistent short sequence repeat polymorphisms in genetically homologous colonizing and invasive Candida albicans strains Transcription through a simple DNA repeat blocks replication elongation Xpa deficiency reduces CAG trinucleotide repeat instability in neuronal tissues in a mouse model of SCA1.Potassium bromate, a potent DNA oxidizing agent, exacerbates germline repeat expansion in a fragile X premutation mouse model Bidirectional transcription stimulates expansion and contraction of expanded (CTG)*(CAG) repeats.Determinants of R-loop formation at convergent bidirectionally transcribed trinucleotide repeats Long CTG tracts from the myotonic dystrophy gene induce deletions and rearrangements during recombination at the APRT locus in CHO cells.Double-strand breaks in the myotonic dystrophy type 1 and the fragile X syndrome triplet repeat sequences induce different types of mutations in DNA flanking sequences in Escherichia coli.Repeat instability during DNA repair: Insights from model systems Repeat instability: mechanisms of dynamic mutations.ATR protects the genome against CGG.CCG-repeat expansion in Fragile X premutation mice.Cell-free cloning of highly expanded CTG repeats by amplification of dimerized expanded repeats Transcription destabilizes triplet repeats Abundance and length of simple repeats in vertebrate genomes are determined by their structural properties.Long intronic GAA*TTC repeats induce epigenetic changes and reporter gene silencing in a molecular model of Friedreich ataxia.Features of trinucleotide repeat instability in vivo.Transcription influences the types of deletion and expansion products in an orientation-dependent manner from GAC*GTC repeats Expanded complexity of unstable repeat diseases.Transcription-induced DNA toxicity at trinucleotide repeats: double bubble is trouble.Mismatch repair enhances convergent transcription-induced cell death at trinucleotide repeats by activating ATR.R Loops and Links to Human Disease.Single-stranded DNA-binding protein in vitro eliminates the orientation-dependent impediment to polymerase passage on CAG/CTG repeats.Selectable system for monitoring the instability of CTG/CAG triplet repeats in mammalian cells.Transcription-induced CAG repeat contraction in human cells is mediated in part by transcription-coupled nucleotide excision repair.Proofreading and secondary structure processing determine the orientation dependence of CAG x CTG trinucleotide repeat instability in Escherichia coli.Mechanisms of dinucleotide repeat instability in Escherichia coli.Inhibitory effects of expanded GAA.TTC triplet repeats from intron I of the Friedreich ataxia gene on transcription and replication in vivo.The Chromatin Remodeler Isw1 Prevents CAG Repeat Expansions During Transcription in Saccharomyces cerevisiae.The myotonic dystrophy type 1 triplet repeat sequence induces gross deletions and inversions.Increased negative superhelical density in vivo enhances the genetic instability of triplet repeat sequences.

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P2860

Transcription increases the deletion frequency of long CTG.CAG triplet repeats from plasmids in Escherichia coli.

http://www.wikidata.org/entity/Q34631670

description

1997 nî lūn-bûn

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1997 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

1997 թվականի հուլիսին հրատարակված գիտական հոդված

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1997年の論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年论文

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name

Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

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type

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Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

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Transcription increases the de ...... plasmids in Escherichia coli.

@en

Transcription increases the de ...... plasmids in Escherichia coli.

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Nucleic Acids Research

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Transcription increases the de ...... plasmids in Escherichia coli.

@en

P2093

A Jaworski

http://www.w3.org/2001/XMLSchema#string

J E Larson

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P Parniewski

http://www.w3.org/2001/XMLSchema#string

R D Wells

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R P Bowater

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P2860

Simple tandem DNA repeats and human genetic disease

Simple sequences

Molecular basis of genetic instability of triplet repeats

Slipped-strand mispairing: a major mechanism for DNA sequence evolution

Microsatellite instability in cancer of the proximal colon

Consequences of replication fork movement through transcription units in vivo.

Destabilization of simple repetitive DNA sequences by transcription in yeast.

The stationary phase of the bacterial life cycle.

GASPing for life in stationary phase.

Dynamic mutations: a new class of mutations causing human disease.

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2861-2868

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10.1093/NAR/25.14.2861

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14

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25

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277443956

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9207036

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Escherichia coli

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146811

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