Destabilization of simple repetitive DNA sequences by transcription in yeast. - Wikidata - awgldk - TriplyDB

Destabilization of simple repetitive DNA sequences by transcription in yeast.

Destabilization of simple repetitive DNA sequences by transcription in yeast.

http://www.wikidata.org/entity/Q33967667

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New insights into repeat instability: role of RNA•DNA hybrids Inactivation of Exonuclease 1 in mice results in DNA mismatch repair defects, increased cancer susceptibility, and male and female sterility Identification of a mutant DNA polymerase delta in Saccharomyces cerevisiae with an antimutator phenotype for frameshift mutations High frequency of microsatellites in S. cerevisiae meiotic recombination hotspots The peculiar landscape of repetitive sequences in the olive (Olea europaea L.) genome.Increased instability of human CTG repeat tracts on yeast artificial chromosomes during gametogenesis.Destabilization of yeast micro- and minisatellite DNA sequences by mutations affecting a nuclease involved in Okazaki fragment processing (rad27) and DNA polymerase delta (pol3-t).Determining mutation rates in bacterial populations Variation in efficiency of DNA mismatch repair at different sites in the yeast genome.Regulation of base excision repair: Ntg1 nuclear and mitochondrial dynamic localization in response to genotoxic stress Microsatellite instability in yeast: dependence on the length of the microsatellite.Isolation and characterization of point mutations in mismatch repair genes that destabilize microsatellites in yeast.Identification of a distinctive mutation spectrum associated with high levels of transcription in yeast Fine-tuning gene networks using simple sequence repeats.Base composition of mononucleotide runs affects DNA polymerase slippage and removal of frameshift intermediates by mismatch repair in Saccharomyces cerevisiae A mutation of the yeast gene encoding PCNA destabilizes both microsatellite and minisatellite DNA sequences Genetic analysis of transcription-associated mutation in Saccharomyces cerevisiae.Regulation of genome stability by TEL1 and MEC1, yeast homologs of the mammalian ATM and ATR genes.Delineating the requirements for spontaneous DNA damage resistance pathways in genome maintenance and viability in Saccharomyces cerevisiae.Genomic instability induced by mutations in Saccharomyces cerevisiae POL1.Transcription increases the deletion frequency of long CTG.CAG triplet repeats from plasmids in Escherichia coli.Triplet repeats form secondary structures that escape DNA repair in yeast.Selection and analysis of spontaneous reciprocal mitotic cross-overs in Saccharomyces cerevisiae Relative rates of insertion and deletion mutations in a microsatellite sequence in cultured cells Genome of Cnaphalocrocis medinalis Granulovirus, the First Crambidae-Infecting Betabaculovirus Isolated from Rice Leaffolder to Sequenced.Genetic regulation of telomere-telomere fusions in the yeast Saccharomyces cerevisae.Oligonucleotide transformation of yeast reveals mismatch repair complexes to be differentially active on DNA replication strands.Ninety-six haploid yeast strains with individual disruptions of open reading frames between YOR097C and YOR192C, constructed for the Saccharomyces genome deletion project, have an additional mutation in the mismatch repair gene MSH3 The rate and spectrum of microsatellite mutation in Caenorhabditis elegans and Daphnia pulex.Dual roles for DNA sequence identity and the mismatch repair system in the regulation of mitotic crossing-over in yeast.Transcription destabilizes triplet repeats A versatile microsatellite instability reporter system in human cells.Analysis of microsatellite mutations in the mitochondrial DNA of Saccharomyces cerevisiae.Recombinogenic conditions influence partner choice in spontaneous mitotic recombination.Beyond junk-variable tandem repeats as facilitators of rapid evolution of regulatory and coding sequences Transcription influences the types of deletion and expansion products in an orientation-dependent manner from GAC*GTC repeats A mononucleotide repeat in PRRT2 is an important, frequent target of mismatch repair deficiency in cancer.Expanded complexity of unstable repeat diseases.Transcription-induced DNA toxicity at trinucleotide repeats: double bubble is trouble.Mismatch repair enhances convergent transcription-induced cell death at trinucleotide repeats by activating ATR.

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P2860

Destabilization of simple repetitive DNA sequences by transcription in yeast.

http://www.wikidata.org/entity/Q33967667

description

1996 nî lūn-bûn

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1996 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի հունիսին հրատարակված գիտական հոդված

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1996年の論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年论文

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name

Destabilization of simple repetitive DNA sequences by transcription in yeast.

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Destabilization of simple repetitive DNA sequences by transcription in yeast.

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Destabilization of simple repetitive DNA sequences by transcription in yeast.

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Destabilization of simple repetitive DNA sequences by transcription in yeast.

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Destabilization of simple repetitive DNA sequences by transcription in yeast.

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Destabilization of simple repetitive DNA sequences by transcription in yeast.

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P2860

A system of shuttle vectors and yeast host strains designed for efficient manipulation of DNA in Saccharomyces cerevisiae

Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer

Sequences that regulate the divergent GAL1-GAL10 promoter in Saccharomyces cerevisiae

Destabilization of tracts of simple repetitive DNA in yeast by mutations affecting DNA mismatch repair

Mutations in the MSH3 gene preferentially lead to deletions within tracts of simple repetitive DNA in Saccharomyces cerevisiae.

A new type of fusion analysis applicable to many organisms: protein fusions to the URA3 gene of yeast.

Frameshift mutations and the genetic code. This paper is dedicated to Professor Theodosius Dobzhansky on the occasion of his 66th birthday.

Z-DNA-forming sequences are spontaneous deletion hot spots.

Genetic instability in human ovarian cancer cell lines.

Triplet repeat mutations in human disease.

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