Cytolytic pathways in haematopoietic stem-cell transplantation. - Wikidata - awgldk - TriplyDB

Cytolytic pathways in haematopoietic stem-cell transplantation.

Cytolytic pathways in haematopoietic stem-cell transplantation.

http://www.wikidata.org/entity/Q34632083

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Diagnosis and differential diagnosis of hepatic graft versus host disease (GVHD)Bone Marrow GvHD after Allogeneic Hematopoietic Stem Cell Transplantation Therapeutics for Graft-versus-Host Disease: From Conventional Therapies to Novel Virotherapeutic Strategies The complex and central role of interferon-γ in graft-versus-host disease and graft-versus-tumor activity Disease severity and mortality can be independently regulated in a mouse model of experimental graft versus host disease Murine and human myogenic cells identified by elevated aldehyde dehydrogenase activity: implications for muscle regeneration and repair Graft-versus-host disease in paediatric solid organ transplantation: A review of the literature.The cytolytic molecules Fas ligand and TRAIL are required for murine thymic graft-versus-host disease Current and future approaches for control of graft-versus-host disease.IL-17 genetic and immunophenotypic evaluation in chronic graft-versus-host disease.Pathogenesis and management of graft-versus-host disease.Notch signaling is a critical regulator of allogeneic CD4+ T-cell responses mediating graft-versus-host disease.Graft-versus-host disease is enhanced by selective CD73 blockade in mice HDAC inhibition and graft versus host disease Abrogation of donor T-cell IL-21 signaling leads to tissue-specific modulation of immunity and separation of GVHD from GVL Immunotherapy of cancer by active vaccination: does allogeneic bone marrow transplantation after non-myeloablative conditioning provide a new option?IL-35 mitigates murine acute graft-versus-host disease with retention of graft-versus-leukemia effects Homeostatic expansion and repertoire regeneration of donor T cells during graft versus host disease is constrained by the host environment.CCR2 is required for CD8-induced graft-versus-host disease.Remodeling specific immunity by use of MHC tetramers: demonstration in a graft-versus-host disease model.Inhibition of histone methylation arrests ongoing graft-versus-host disease in mice by selectively inducing apoptosis of alloreactive effector T cells.A long road of T-cells to cure cancer: from adoptive immunotherapy with unspecific cellular products to donor lymphocyte infusions and transfer of engineered tumor-specific T-cells.Pathophysiology of acute graft-versus-host disease: recent advances.Differential Effect of MyD88 Signal in Donor T Cells on Graft-versus-Leukemia Effect and Graft-versus-Host Disease after Experimental Allogeneic Stem Cell Transplantation.IL-17-deficient allogeneic bone marrow transplantation prevents the induction of collagen-induced arthritis in DBA/1J mice Effector memory CD4+ T cells mediate graft-versus-leukemia without inducing graft-versus-host disease.CD8+ but not CD4+ T cells require cognate interactions with target tissues to mediate GVHD across only minor H antigens, whereas both CD4+ and CD8+ T cells require direct leukemic contact to mediate GVL.Relapse after allogeneic hematopoietic cell therapy.A critical role for the retinoic acid signaling pathway in the pathophysiology of gastrointestinal graft-versus-host disease.Differential effects of donor T-cell cytokines on outcome with continuous bortezomib administration after allogeneic bone marrow transplantation.Host-derived CD8+ dendritic cells are required for induction of optimal graft-versus-tumor responses after experimental allogeneic bone marrow transplantation.Targeting deacetylases to improve outcomes after allogeneic bone marrow transplantation.Mouse models of bone marrow transplantation IL-17 contributes to CD4-mediated graft-versus-host disease.Interleukin-23 secretion by donor antigen-presenting cells is critical for organ-specific pathology in graft-versus-host disease.GVHD: a continuing barrier to the safety of allogeneic transplantation The histone methyltransferase Ezh2 is a crucial epigenetic regulator of allogeneic T-cell responses mediating graft-versus-host disease Cytolytic T cells induce ceramide-rich platforms in target cell membranes to initiate graft-versus-host disease.Acute graft-versus-host disease: from the bench to the bedside.Cytomegalovirus (CMV)-Specific Perforin and Granzyme B ELISPOT Assays Detect Reactivation of CMV Infection in Inflammatory Bowel Disease.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

http://www.wikidata.org/entity/Q34632083

description

2002 nî lūn-bûn

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2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2002 թվականի ապրիլին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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type

label

Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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prefLabel

Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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Cytolytic pathways in haematopoietic stem-cell transplantation.

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Marcel R M van den Brink

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P2860

Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations

TRAIL (Apo2 ligand) and TWEAK (Apo3 ligand) mediate CD4+ T cell killing of antigen-presenting macrophages

CD95's deadly mission in the immune system

Apoptosis by death factor

Cytotoxicity mediated by T cells and natural killer cells is greatly impaired in perforin-deficient mice

Pathophysiologic mechanisms of acute graft-vs.-host disease.

IL-11 separates graft-versus-leukemia effects from graft-versus-host disease after bone marrow transplantation.

A domain in TNF receptors that mediates ligand-independent receptor assembly and signaling.

A fresh look at tumor immunosurveillance and immunotherapy.

The development of lymphomas in families with autoimmune lymphoproliferative syndrome with germline Fas mutations and defective lymphocyte apoptosis.

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