Depletion of SMN by RNA interference in HeLa cells induces defects in Cajal body formation.
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WRAP53 is essential for Cajal body formation and for targeting the survival of motor neuron complex to Cajal bodiesTwo distinct arginine methyltransferases are required for biogenesis of Sm-class ribonucleoproteinsCajal-body formation correlates with differential coilin phosphorylation in primary and transformed cell linesDephosphorylation of survival motor neurons (SMN) by PPM1G/PP2Cgamma governs Cajal body localization and stability of the SMN complexA stochastic view of spliceosome assembly and recycling in the nucleusCoilin: The first 25 yearsTDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy.Genetic analysis of nuclear bodies: from nondeterministic chaos to deterministic order.The role of nuclear bodies in gene expression and disease.The Cajal body and the nucleolus: "In a relationship" or "It's complicated"?Whole-genome screening identifies proteins localized to distinct nuclear bodiesCajal body proteins differentially affect the processing of box C/D scaRNPs.TDRD6 mediates early steps of spliceosome maturation in primary spermatocytes.The Hsp90 chaperone controls the biogenesis of L7Ae RNPs through conserved machineryGemin3 is an essential gene required for larval motor function and pupation in Drosophila.The loss of the snoRNP chaperone Nopp140 from Cajal bodies of patient fibroblasts correlates with the severity of spinal muscular atrophy.Fibroblast growth factor-2 regulates the stability of nuclear bodies.Phosphorylation and the Cajal body: modification in search of function.Fam118B, a newly identified component of Cajal bodies, is required for Cajal body formation, snRNP biogenesis and cell viability.Cajal bodies: where form meets function.Gem depletion: amyotrophic lateral sclerosis and spinal muscular atrophy crossover.Spliceosomal small nuclear ribonucleoprotein particles repeatedly cycle through Cajal bodies.UsnRNP biogenesis: mechanisms and regulation.SMN - A chaperone for nuclear RNP social occasions?The SMN protein is a key regulator of nuclear architecture in differentiating neuroblastoma cells.Post-transcriptional modification of spliceosomal RNAs is normal in SMN-deficient cells.Diabetic polyneuropathy, sensory neurons, nuclear structure and spliceosome alterations: a role for CWC22.Changes in intranuclear mobility of mature snRNPs provide a mechanism for splicing defects in spinal muscular atrophy.CBC-ARS2 stimulates 3'-end maturation of multiple RNA families and favors cap-proximal processing.Impaired minor tri-snRNP assembly generates differential splicing defects of U12-type introns in lymphoblasts derived from a type I SMA patient.
P2860
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P2860
Depletion of SMN by RNA interference in HeLa cells induces defects in Cajal body formation.
description
2006 nî lūn-bûn
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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@ast
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@en
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@nl
type
label
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@ast
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@en
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@nl
prefLabel
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@ast
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@en
Depletion of SMN by RNA interf ...... fects in Cajal body formation.
@nl
P2093
P2860
P356
P1476
Depletion of SMN by RNA interf ...... efects in Cajal body formation
@en
P2093
Cyrille Girard
Henry Neel
Rémy Bordonné
P2860
P304
P356
10.1093/NAR/GKL374
P407
P577
2006-05-31T00:00:00Z