Specific therapy based on the genotype and cellular mechanism in inherited cardiac arrhythmias. Long QT syndrome and Brugada syndrome
about
Congenital long QT syndromes: clinical features, molecular genetics and genetic testing.Successful elimination of significant arrhythmia burden with flecainide in an adolescent with long QT syndrome type 3.Novel calmodulin mutations associated with congenital arrhythmia susceptibility.Abnormal repolarization as the basis for late potentials and fractionated electrograms recorded from epicardium in experimental models of Brugada syndrome.Partially dominant mutant channel defect corresponding with intermediate LQT2 phenotype.Pharmacogenetics and anti-arrhythmic drug therapy: a theoretical investigation.Role of sodium channels in propagation in heart muscle: how subtle genetic alterations result in major arrhythmic disorders.Mechanisms of drug-induced proarrhythmia in clinical practice.A new hERG allosteric modulator rescues genetic and drug-induced long-QT syndrome phenotypes in cardiomyocytes from isogenic pairs of patient induced pluripotent stem cells.Malignant perinatal variant of long-QT syndrome caused by a profoundly dysfunctional cardiac sodium channelCilostazol ameliorates atrial ionic remodeling in long-term rapid atrial pacing dogs.Delayed and indirect effects of antiarrhythmic drugs in reducing sudden cardiac death.Late sodium current inhibition in acquired and inherited ventricular (dys)function and arrhythmias.Congenital long QT syndromes: prevalence, pathophysiology and management.Antiarrhythmic Drug Therapy to Avoid Implantable Cardioverter Defibrillator Shocks.Reduction of repolarization reserve unmasks the proarrhythmic role of endogenous late Na(+) current in the heart.Drug Therapy For Preventing Ventricular Arrhythmia In Brugada syndrome: Do We Have The Answers Yet?A rare association of long QT syndrome and syndactyly: Timothy syndrome (LQT 8).Evidence for a crucial modulating role of the sodium channel in the QTc prolongation related to antipsychotics.Effects of L-type Ca2+ channel antagonism on ventricular arrhythmogenesis in murine hearts containing a modification in the Scn5a gene modelling human long QT syndrome 3.Calcineurin Increases Cardiac Transient Outward K+Currents via Transcriptional Up-regulation of Kv4.2 Channel Subunits
P2860
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P2860
Specific therapy based on the genotype and cellular mechanism in inherited cardiac arrhythmias. Long QT syndrome and Brugada syndrome
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Specific therapy based on the ...... syndrome and Brugada syndrome
@ast
Specific therapy based on the ...... syndrome and Brugada syndrome
@en
Specific therapy based on the ...... syndrome and Brugada syndrome
@nl
type
label
Specific therapy based on the ...... syndrome and Brugada syndrome
@ast
Specific therapy based on the ...... syndrome and Brugada syndrome
@en
Specific therapy based on the ...... syndrome and Brugada syndrome
@nl
prefLabel
Specific therapy based on the ...... syndrome and Brugada syndrome
@ast
Specific therapy based on the ...... syndrome and Brugada syndrome
@en
Specific therapy based on the ...... syndrome and Brugada syndrome
@nl
P2860
P356
P1476
Specific therapy based on the ...... syndrome and Brugada syndrome
@en
P2093
Takeshi Aiba
Wataru Shimizu
P2860
P304
P356
10.2174/1381612053764823
P577
2005-01-01T00:00:00Z