Early treatment suppresses the development of spike-wave epilepsy in a rat model - Wikidata - awgldk - TriplyDB

Early treatment suppresses the development of spike-wave epilepsy in a rat model

Early treatment suppresses the development of spike-wave epilepsy in a rat model

http://www.wikidata.org/entity/Q34723299

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Clinical risk factors associated with anti-epileptic drug responsiveness in canine epilepsy Novel frontiers in epilepsy treatments: preventing epileptogenesis by targeting inflammation Treatment in canine epilepsy--a systematic review Issues related to symptomatic and disease-modifying treatments affecting cognitive and neuropsychiatric comorbidities of epilepsy Mechanisms of Action of Antiseizure Drugs and the Ketogenic Diet Evidence for a role of Nav1.6 in facilitating increases in neuronal hyperexcitability during epileptogenesis.Absence epilepsy in apathetic, a spontaneous mutant mouse lacking the h channel subunit, HCN2 Increased persistent Na+ current contributes to seizure in the slamdance bang-sensitive Drosophila mutant.The potential of antiseizure drugs and agents that act on novel molecular targets as antiepileptogenic treatments.Searching for the ideal antiepileptogenic agent in experimental models: single treatment versus combinatorial treatment strategies Neuroimaging biomarkers of epileptogenesis.Rhythm and blues: animal models of epilepsy and depression comorbidity.Where fMRI and electrophysiology agree to disagree: corticothalamic and striatal activity patterns in the WAG/Rij rat.Finding a better drug for epilepsy: preclinical screening strategies and experimental trial design.Prevention or modification of epileptogenesis after brain insults: experimental approaches and translational research.Mammalian target of rapamycin (mTOR) inhibition as a potential antiepileptogenic therapy: From tuberous sclerosis to common acquired epilepsies Antidepressants but not antipsychotics have antiepileptogenic effects with limited effects on comorbid depressive-like behaviour in the WAG/Rij rat model of absence epilepsy An N-terminal deletion variant of HCN1 in the epileptic WAG/Rij strain modulates HCN current densities.Long-term outcomes of generalized tonic-clonic seizures in a childhood absence epilepsy trial.Development of spike-wave seizures in C3H/HeJ mice.Neurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures.Increased resting functional connectivity in spike-wave epilepsy in WAG/Rij rats.2014 Epilepsy Benchmarks Area II: Prevent Epilepsy and Its Progression.Computer modelling of epilepsy The challenge and promise of anti-epileptic therapy development in animal models.DTI abnormalities in anterior corpus callosum of rats with spike-wave epilepsy Hyperpolarization activated cyclic-nucleotide gated (HCN) channels in developing neuronal networks.Early life stress as an influence on limbic epilepsy: an hypothesis whose time has come?Past and present definitions of epileptogenesis and its biomarkers.Long-term seizure remission in childhood absence epilepsy: might initial treatment matter?Is epilepsy a preventable disorder? New evidence from animal models.Therapeutic approaches to epileptogenesis--hope on the horizon.Human clinical trails in antiepileptogenesis.Low threshold T-type calcium channels as targets for novel epilepsy treatments.Development of new treatment approaches for epilepsy: unmet needs and opportunities.Anti-epileptogenesis: Electrophysiology, diffusion tensor imaging and behavior in a genetic absence model.Dynamics of networks during absence seizure's on- and offset in rodents and man.Fingolimod Exerts only Temporary Antiepileptogenic Effects but Longer-Lasting Positive Effects on Behavior in the WAG/Rij Rat Absence Epilepsy Model.Animal Models of Seizures and Epilepsy: Past, Present, and Future Role for the Discovery of Antiseizure Drugs.Advancing pharmacologic treatment options for pharmacologic treatment options for children with epilepsy.

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P2860

Early treatment suppresses the development of spike-wave epilepsy in a rat model

http://www.wikidata.org/entity/Q34723299

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2007 nî lūn-bûn

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2007 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2007年論文

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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J.1528-1167.2007.01458.X

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Early treatment suppresses the development of spike-wave epilepsy in a rat model

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Anuradha Phadke

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April R Levin

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Chhitij Bashyal

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Crystal Paul-Laughinghouse

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Dario J Englot

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Davender S Khera

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Frederick Wang

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Hal Blumenfeld

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Hrachya Nersesyan

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John Mission

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P2860

Enhanced expression of a specific hyperpolarization-activated cyclic nucleotide-gated cation channel (HCN) in surviving dentate gyrus granule cells of human and experimental epileptic hippocampus

The neurobiology of antiepileptic drugs

Characterization of ethosuximide reduction of low-threshold calcium current in thalamic neurons

Environmental manipulations early in development alter seizure activity, Ih and HCN1 protein expression later in life

Consciousness and epilepsy: why are patients with absence seizures absent?

Brain imaging in idiopathic generalized epilepsies.

Relative changes in cerebral blood flow and neuronal activity in local microdomains during generalized seizures.

Cellular and network mechanisms of spike-wave seizures.

The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons.

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400-409

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3

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49

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2007-12-06T00:00:00Z

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