Signaling crosstalk between RANKL and interferons in osteoclast differentiation. - Wikidata - awgldk - TriplyDB

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

http://www.wikidata.org/entity/Q34731222

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Enhanced osteoclast development in collagen-induced arthritis in interferon-gamma receptor knock-out mice as related to increased splenic CD11b+ myelopoiesis Osteoimmunology and the influence of pro-inflammatory cytokines on osteoclasts Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia Structural basis of a unique interferon-β signaling axis mediated via the receptor IFNAR1 Dynamin forms a Src kinase-sensitive complex with Cbl and regulates podosomes and osteoclast activity.TRP14 inhibits osteoclast differentiation via its catalytic activity T cells: critical bone regulators in health and disease.Osteoclasts and odontoclasts: signaling pathways to development and disease.Subchondral bone microstructural damage by increased remodelling aggravates experimental osteoarthritis preceded by osteoporosis.Cytokine-mediated bone destruction in rheumatoid arthritis.Osteoclast activated FoxP3+ CD8+ T-cells suppress bone resorption in vitro.Gene therapy for arthritis.New aspects of IFN-alpha/beta signalling in immunity, oncogenesis and bone metabolism.IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.Stauntonia hexaphylla (Lardizabalaceae) leaf methanol extract inhibits osteoclastogenesis and bone resorption activity via proteasome-mediated degradation of c-Fos protein and suppression of NFATc1 expression.Interactions between the immune system and bone.Stat1 functions as a cytoplasmic attenuator of Runx2 in the transcriptional program of osteoblast differentiation.Type 1 interferons suppress accelerated osteoclastogenesis and prevent loss of bone mass during systemic inflammatory responses to Pneumocystis lung infection.Bone turnover and nutritional status in Crohn's disease: relationship to circulating mononuclear cell function and response to fish oil and antioxidants.Inflammatory bone destruction and osteoimmunology.In vivo impact of a 4 bp deletion mutation in the DLX3 gene on bone development.Interplay between interferon and other cytokine systems in bone metabolism.The role of the immune system in the physiopathology of osteoporosis Direct and indirect estrogen actions on osteoblasts and osteoclasts.Functions of RANKL/RANK/OPG in bone modeling and remodeling.Osteoclasts - the innate immune cells of the bone.Association of polymorphisms in the Interleukin 23 receptor gene with osteonecrosis of femoral head in Korean population Dynamin reduces Pyk2 Y402 phosphorylation and SRC binding in osteoclasts Green tea and bone metabolism.NADPH oxidase gp91(phox) contributes to RANKL-induced osteoclast differentiation by upregulating NFATc1 Selective induction of P2Y14 receptor by RANKL promotes osteoclast formation.Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis: a longitudinal study of gene expression in bone tissue from glucocorticoid-treated mice.Role of T cells in ovariectomy induced bone loss--revisited.Immunological reaction in TNF-α-mediated osteoclast formation and bone resorption in vitro and in vivo.Immunoglobulin class switch recombination deficiency type 1 or CD40 ligand deficiency: from bedside to bench and back again.Microarray profile of gene expression during osteoclast differentiation in modelled microgravity.Prostaglandin E(2) is a main mediator in receptor activator of nuclear factor-kappaB ligand-dependent osteoclastogenesis induced by Porphyromonas gingivalis, Treponema denticola, and Treponema socranskii.Role of FK506 binding protein 5 (FKBP5) in osteoclast differentiation.A RANKL-inducible gene Znf216 in osteoclast differentiation.Elevated Response to Type I IFN Enhances RANKL-Mediated Osteoclastogenesis in Usp18-Knockout Mice.

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P2860

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

http://www.wikidata.org/entity/Q34731222

description

2002 nî lūn-bûn

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2002 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2002 թվականի մայիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@ast

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@en

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@nl

type

label

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@ast

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@en

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@nl

prefLabel

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@ast

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@en

Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

@nl

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P1433

Arthritis Research and Therapy

Arthritis Research

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Signaling crosstalk between RANKL and interferons in osteoclast differentiation.

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P2093

Hiroshi Takayanagi

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Sunhwa Kim

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Tadatsugu Taniguchi

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P2860

Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

How cells respond to interferons

Severe osteopetrosis, defective interleukin-1 signalling and lymph node organogenesis in TRAF6-deficient mice

A new mechanism of bone destruction in rheumatoid arthritis: synovial fibroblasts induce osteoclastogenesis

The TRAF family of signal transducers mediates NF-kappaB activation by the TRANCE receptor

TRAF6 deficiency results in osteopetrosis and defective interleukin-1, CD40, and LPS signaling

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10.1186/AR581

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4 Suppl 3

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2002-05-09T00:00:00Z

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11268667

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transport protein

membrane proteins

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