IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation. - Wikidata - awgldk - TriplyDB

IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.

IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.

http://www.wikidata.org/entity/Q35219971

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Interleukin-32 promotes osteoclast differentiation but not osteoclast activation T lymphocytes amplify the anabolic activity of parathyroid hormone through Wnt10b signaling Osteoimmunology: interactions of the bone and immune system Osteoimmune Interactions in Inflammatory Bowel Disease: Central Role of Bone Marrow Th17 TNFα Cells in Osteoclastogenesis The role of "bone immunological niche" for a new pathogenetic paradigm of osteoporosis Host response mechanisms in periodontal diseases Osteoimmunology and the influence of pro-inflammatory cytokines on osteoclasts Immune regulation of osteoclast function in postmenopausal osteoporosis: a critical interdisciplinary perspective HIV: inflammation and bone T cells induce pre-metastatic osteolytic disease and help bone metastases establishment in a mouse model of metastatic breast cancer Bone Cells Dynamics during Peri-Implantitis: a Theoretical Analysis Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia Systems genetics: a novel approach to dissect the genetic basis of osteoporosis.Identification of a gene module associated with BMD through the integration of network analysis and genome-wide association data.Lymphocytes and the Dap12 adaptor are key regulators of osteoclast activation associated with gonadal failure.Interferon (IFN)-γ-mediated inflammation and the kynurenine pathway in relation to bone mineral density: the Hordaland Health Study.A bone-protective role for IL-17 receptor signaling in ovariectomy-induced bone loss.Elevated suppressor of cytokine signaling-1 (SOCS-1): a mechanism for dysregulated osteoclastogenesis in HIV transgenic rats Vitamin D and immunomodulation in early rheumatoid arthritis: A randomized double-blind placebo-controlled study.Interferon gamma (IFN-γ)-mediated inflammation and the kynurenine pathway in relation to risk of hip fractures: the Hordaland Health Study Oxidative stress and gamma radiation-induced cancellous bone loss with musculoskeletal disuse.Commensal Gut Microbiota Immunomodulatory Actions in Bone Marrow and Liver have Catabolic Effects on Skeletal Homeostasis in Health.Circulating osteogenic cells: characterization and relationship to rates of bone loss in postmenopausal women.The oxytocin-bone axis Simultaneous analysis of T helper subsets (Th1, Th2, Th9, Th17, Th22, Tfh, Tr1 and Tregs) markers expression in periapical lesions reveals multiple cytokine clusters accountable for lesions activity and inactivity status.T cells: critical bone regulators in health and disease.The immune system and bone.Osteoporosis and gastrointestinal disease.Targeted overexpression of osteoactivin in cells of osteoclastic lineage promotes osteoclastic resorption and bone loss in mice.Cytokine-mediated bone destruction in rheumatoid arthritis.Oxytocin and bone Transplantation of SHED prevents bone loss in the early phase of ovariectomy-induced osteoporosis.Ovariectomy disregulates osteoblast and osteoclast formation through the T-cell receptor CD40 ligand.Human osteoclasts are inducible immunosuppressive cells in response to T cell-derived IFN-γ and CD40 ligand in vitro Effects of a structured weight-bearing exercise program on bone metabolism among breast cancer survivors: a feasibility trial The polyphenol fisetin protects bone by repressing NF-κB and MKP-1-dependent signaling pathways in osteoclasts Novel immunostimulatory effects of osteoclasts and macrophages on human γδ T cells.The sclerostin-independent bone anabolic activity of intermittent PTH treatment is mediated by T-cell-produced Wnt10b TLR2-dependent modulation of osteoclastogenesis by Porphyromonas gingivalis through differential induction of NFATc1 and NF-kappaB.Inflammation and uncoupling as mechanisms of periodontal bone loss

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IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.

http://www.wikidata.org/entity/Q35219971

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

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IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

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type

label

IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

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IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

@en

prefLabel

IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

@ast

IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

@en

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Journal of Clinical Investigation

P1476

IFN-gamma stimulates osteoclas ...... igen-driven T cell activation.

@en

P2093

Francesco Grassi

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Karen Page

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M Neale Weitzmann

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Masakazu Terauchi

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Michaela Robbie Ryan

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Roberto Pacifici

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Xiaoying Yang

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Yuhao Gao

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P2860

Positive regulatory domain I-binding factor 1 mediates repression of the MHC class II transactivator (CIITA) type IV promoter

IFN-gamma-producing human T cells directly induce osteoclastogenesis from human monocytes via the expression of RANKL

Exposure to receptor-activator of NFkappaB ligand renders pre-osteoclasts resistant to IFN-gamma by inducing terminal differentiation

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor kappa B ligand and tumor necrosis factor alpha from T cells

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

Gamma interferon positively modulates Actinobacillus actinomycetemcomitans-specific RANKL+ CD4+ Th-cell-mediated alveolar bone destruction in vivo.

Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.

CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice

An IL-7-dependent rebound in thymic T cell output contributes to the bone loss induced by estrogen deficiency.

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122-132

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10.1172/JCI30074

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1

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117

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2006-12-14T00:00:00Z

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17173138

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1697800

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