IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
about
Meta-analysis of associations of interleukin-28B polymorphisms rs8099917 and rs12979860 with development of hepatitis virus-related hepatocellular carcinomaHepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress ResponseInterferon Response in Hepatitis C Virus (HCV) Infection: Lessons from Cell Culture Systems of HCV InfectionNatural history and clinical response: "it's the virus, stupid, or is it the host?"STAT proteins - key regulators of anti-viral responses, inflammation, and tumorigenesis in the liverFuture classes of hepatitis C virus therapeutic agentsComparison of antiviral activity of lambda-interferons against HIV replication in macrophagesPrevalence of IFNL3 rs4803217 single nucleotide polymorphism and clinical course of chronic hepatitis C.IL28B SNP rs12979860 is a critical predictor for on-treatment and sustained virologic response in patients with hepatitis C virus genotype-1 infectionHepatitis C virus NS5A hijacks ARFGAP1 to maintain a phosphatidylinositol 4-phosphate-enriched microenvironmentHepatitis C virus in the new era: perspectives in epidemiology, prevention, diagnostics and predictors of response to therapy.A functional genomic screen reveals novel host genes that mediate interferon-alpha's effects against hepatitis C virus.IL28B alleles exert an additive dose effect when applied to HCV-HIV coinfected persons undergoing peginterferon and ribavirin therapyIFN-λ3 inhibits HIV infection of macrophages through the JAK-STAT pathway.IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infectionClinical utility of pharmacogenomics in the management of hepatitis C.IFN-λ Inhibits MiR-122 Transcription through a Stat3-HNF4α Inflammatory Feedback Loop in an IFN-α Resistant HCV Cell Culture System.The association of IL28B polymorphism and graft survival in patients with hepatitis C undergoing liver transplantationDendritic cells in hepatitis C virus infection: key players in the IFNL3-genotype response.Albumin fusion of interleukin-28B: production and characterization of its biological activities and protein stability.Hepatitis C virus-mediated enhancement of microRNA miR-373 impairs the JAK/STAT signaling pathwayNetwork-based study reveals potential infection pathways of hepatitis-C leading to various diseasesIFNL4 ss469415590 variant shows similar performance to rs12979860 as predictor of response to treatment against Hepatitis C Virus genotype 1 or 4 in CaucasiansRole of IFN-ks, IFN-ks related genes and the DEPDC5 gene in Hepatitis B virus-related liver disease.Detection of allele specific differences in IFNL3 (IL28B) mRNA expressionEFTUD2 Is a Novel Innate Immune Regulator Restricting Hepatitis C Virus Infection through the RIG-I/MDA5 Pathway.KPP: KEGG Pathway PainterChaperone-Mediated Autophagy Targets IFNAR1 for Lysosomal Degradation in Free Fatty Acid Treated HCV Cell Culture.EPSTI1 Is Involved in IL-28A-Mediated Inhibition of HCV InfectionThe impact of interferon lambda 3 gene polymorphism on natural course and treatment of hepatitis CRecent advances in the anti-HCV mechanisms of interferon.Rs12979860 and rs8099917 single nucleotide polymorphisms of interleukin-28B gene: simultaneous genotyping in caucasian patients infected with hepatitis C virus.PharmGKB summary: peginterferon-α pathway.Modeling Ribavirin-Induced Anemia in Patients with Chronic Hepatitis C VirusInterferon-lambda (IFN-λ) induces signal transduction and gene expression in human hepatocytes, but not in lymphocytes or monocytes.Induction of interferon-λ contributes to Toll-like receptor-3-activated hepatic stellate cell-mediated hepatitis C virus inhibition in hepatocytesSOCS1 abrogates IFN's antiviral effect on hepatitis C virus replication.Interaction of IFNL3 with insulin resistance, steatosis and lipid metabolism in chronic hepatitis C virus infection.The AMPK-related kinase SNARK regulates hepatitis C virus replication and pathogenesis through enhancement of TGF-β signaling.Kinetic differences in the induction of interferon stimulated genes by interferon-α and interleukin 28B are altered by infection with hepatitis C virus.
P2860
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P2860
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@ast
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@en
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@nl
type
label
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@ast
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@en
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@nl
prefLabel
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@ast
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@en
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@nl
P2093
P2860
P1476
IL28B inhibits hepatitis C virus replication through the JAK-STAT pathway.
@en
P2093
Dahlene N Fusco
Lee F Peng
Leiliang Zhang
Nikolaus Jilg
Run-Xuan Shao
Wen-Chi Chen
P2860
P304
P356
10.1016/J.JHEP.2010.11.019
P577
2010-12-13T00:00:00Z