Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction. - Wikidata - awgldk - TriplyDB

Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.

Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.

http://www.wikidata.org/entity/Q34780382

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BMI1: A Biomarker of Hematologic Malignancies Interleukin-1 Family Cytokines in Liver Diseases Microglia-derived IL-1β promotes chemokine expression by Müller cells and RPE in focal retinal degeneration.Progesterone Attenuates Microglial-Driven Retinal Degeneration and Stimulates Protective Fractalkine-CX3CR1 Signaling.The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction.Fibroblast-myocyte coupling in the heart: Potential relevance for therapeutic interventions Interleukin 1α and the inflammatory process.Inflammation in cardiac injury, repair and regeneration.Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges.Interleukin-1 in cardiac injury, repair, and remodeling: pathophysiologic and translational concepts Targeting danger-associated molecular patterns after myocardial infarction.Pathophysiology of Myocardial Infarction.Translational failure of anti-inflammatory compounds for myocardial infarction: a meta-analysis of large animal models.miR-181a and miR-150 regulate dendritic cell immune inflammatory responses and cardiomyocyte apoptosis via targeting JAK1-STAT1/c-Fos pathway.11β-HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI.NLRP3 signaling drives macrophage-induced adaptive immune suppression in pancreatic carcinoma.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice.Tumor necrosis factor-alpha induces VCAM-1-mediated inflammation via c-Src-dependent transactivation of EGF receptors in human cardiac fibroblasts.Potential humoral mediators of remote ischemic preconditioning in patients undergoing surgical coronary revascularization.The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling.Function and regulation of IL-1α in inflammatory diseases and cancer.Exopolysaccharide Produced by Probiotic Strain Lactobacillus paraplantarum BGCG11 Reduces Inflammatory Hyperalgesia in Rats.Cardioprotective Effects of Exosomes and Their Potential Therapeutic Use.Anti-inflammatory therapies in myocardial infarction: failures, hopes, and challenges.A Review of Interleukin-1 in Heart Disease: Where Do We Stand Today?Alcohol attenuates myocardial ischemic injury.Immunothrombotic Activity of Damage-Associated Molecular Patterns and Extracellular Vesicles in Secondary Organ Failure Induced by Trauma and Sterile Insults.Properties and Immune Function of Cardiac Fibroblasts.Short-term MyD88 inhibition ameliorates cardiac graft rejection and promotes donor-specific hyporesponsiveness of skin grafts in mice.Immunomodulatory interventions in myocardial infarction and heart failure: a systematic review of clinical trials and meta-analysis of IL-1 inhibition

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P2860

Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.

http://www.wikidata.org/entity/Q34780382

description

2014 nî lūn-bûn

@nan

2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Cutting edge: IL-1α is a cruci ...... during myocardial infarction.

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Cutting edge: IL-1α is a cruci ...... during myocardial infarction.

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Cutting edge: IL-1α is a cruci ...... during myocardial infarction.

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Cutting edge: IL-1α is a cruci ...... during myocardial infarction.

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JIMMUNOL.1401948

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Journal of Immunology

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Cutting edge: IL-1α is a cruci ...... during myocardial infarction.

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Aubry Tardivel

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Bernard Waeber

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François Feihl

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Lucas Liaudet

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Marianne Zeller

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Olivier Müller

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Roumen Parapanov

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Stéphanie Rignault-Clerc

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P2860

A pilot study of IL-1 inhibition by anakinra in acute gout

Myocardial ischemic preconditioning in rodents is dependent on poly (ADP-ribose) synthetase.

FGF-2 controls the differentiation of resident cardiac precursors into functional cardiomyocytes.

Oxidants positively or negatively regulate nuclear factor kappaB in a context-dependent manner

IL-1α and IL-1β recruit different myeloid cells and promote different stages of sterile inflammation.

The interleukin-1 family: back to the future

Peroxynitrite induces HMGB1 release by cardiac cells in vitro and HMGB1 upregulation in the infarcted myocardium in vivo.

The inflammatory response in myocardial injury, repair, and remodelling.

Innate immune adaptor MyD88 mediates neutrophil recruitment and myocardial injury after ischemia-reperfusion in mice.

Targeting inflammatory pathways in myocardial infarction.

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499-503

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scholarly article

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10.4049/JIMMUNOL.1401948

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2

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194

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Pascal Schneider

Jérôme Lugrin

Catherine Vergely

Nathalie Rosenblatt

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2014-12-10T00:00:00Z

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269715562

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25505286

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4278196

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