Targeting inflammatory pathways in myocardial infarction.
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Targeting the Innate Immune Response to Improve Cardiac Graft Recovery after Heart Transplantation: Implications for the Donation after Cardiac DeathDiacerein improves left ventricular remodeling and cardiac function by reducing the inflammatory response after myocardial infarctionA strategy for the identification of combinatorial bioactive compounds contributing to the holistic effect of herbal medicines.Cyclovirobuxinum D suppresses lipopolysaccharide-induced inflammatory responses in murine macrophages in vitro by blocking JAK-STAT signaling pathway.Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.The inflammatory response in myocardial injury, repair, and remodelling.Role of S100A1 in hypoxia-induced inflammatory response in cardiomyocytes via TLR4/ROS/NF-κB pathway.Reduced acute myocardial ischemia-reperfusion injury in IL-6-deficient mice employing a closed-chest model.Towards comprehensive cardiac repair and regeneration after myocardial infarction: Aspects to consider and proteins to deliver.Emerging Concepts in Paracrine Mechanisms in Regenerative Cardiovascular Medicine and Biology.PEDF Inhibits the Activation of NLRP3 Inflammasome in Hypoxia Cardiomyocytes through PEDF Receptor/Phospholipase A2.Macrophage activation and polarization in post-infarction cardiac remodeling.Fibroblasts in myocardial infarction: a role in inflammation and repair.The Notch pathway: a novel target for myocardial remodelling therapy?Post-myocardial infarct inflammation and the potential role of cell therapy.Inflammation in cardiac injury, repair and regeneration.Non-invasive Macrophage Tracking Using Novel Porphysome Nanoparticles in the Post-myocardial Infarction Murine Heart.Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges.Interleukin-1 in cardiac injury, repair, and remodeling: pathophysiologic and translational conceptsPathophysiology of Myocardial Infarction.Translational failure of anti-inflammatory compounds for myocardial infarction: a meta-analysis of large animal models.Integrins and integrin-related proteins in cardiac fibrosis.Echocardiographic epicardial fat thickness is a predictor for target vessel revascularization in patients with ST-elevation myocardial infarction.Cellular recruitment in myocardial ischaemia/reperfusion injury.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Advanced glycation end-product (AGE)-albumin from activated macrophage is critical in human mesenchymal stem cells survival and post-ischemic reperfusion injury.PRDX2 in Myocyte Hypertrophy and Survival is Mediated by TLR4 in Acute Infarcted MyocardiumSoy isoflavones in nutritionally relevant amounts have varied nutrigenomic effects on adipose tissue.The cardiac microenvironment uses non-canonical WNT signaling to activate monocytes after myocardial infarction.Cardiac-specific YAP activation improves cardiac function and survival in an experimental murine MI model.Myocardial Reparative Properties of Cardiac Mesenchymal Cells Isolated on the Basis of AdherenceIdentification of long non-coding RNAs biomarkers for early diagnosis of myocardial infarction from the dysregulated coding-non-coding co-expression network.FoxO4 promotes early inflammatory response upon myocardial infarction via endothelial Arg1.Elevated plasma levels of interleukin-16 in patients with acute myocardial infarction.Prevention of export of anoxia/reoxygenation injury from ischemic to nonischemic cardiomyocytes via inhibition of endocytosis.MR Assessment of Acute Pathologic Process after Myocardial Infarction in a Permanent Ligation Mouse Model: Role of Magnetic Nanoparticle-Contrasted MRI.Umbelliferone Alleviates Myocardial Ischemia: the Role of Inflammation and Apoptosis.CpG oligodeoxynucleotide preconditioning improves cardiac function after myocardial infarction via modulation of energy metabolism and angiogenesis.GDF-15 and TRAIL-R2 are powerful predictors of long-term mortality in patients with acute myocardial infarction.
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Targeting inflammatory pathways in myocardial infarction.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 June 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Targeting inflammatory pathways in myocardial infarction.
@en
Targeting inflammatory pathways in myocardial infarction.
@nl
type
label
Targeting inflammatory pathways in myocardial infarction.
@en
Targeting inflammatory pathways in myocardial infarction.
@nl
prefLabel
Targeting inflammatory pathways in myocardial infarction.
@en
Targeting inflammatory pathways in myocardial infarction.
@nl
P2860
P356
P1476
Targeting inflammatory pathways in myocardial infarction
@en
P2093
Panagiota Christia
P2860
P304
P356
10.1111/ECI.12118
P577
2013-06-17T00:00:00Z