Homologous recombination is required for genome stability in the absence of DOG-1 in Caenorhabditis elegans - Wikidata - awgldk - TriplyDB

Homologous recombination is required for genome stability in the absence of DOG-1 in Caenorhabditis elegans

Homologous recombination is required for genome stability in the absence of DOG-1 in Caenorhabditis elegans

http://www.wikidata.org/entity/Q34895210

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A role for the malignant brain tumour (MBT) domain protein LIN-61 in DNA double-strand break repair by homologous recombination RTEL1 maintains genomic stability by suppressing homologous recombination Getting Ready for the Dance: FANCJ Irons Out DNA Wrinkles Role of DNA polymerase κ in the maintenance of genomic stability Replication of Structured DNA and its implication in epigenetic stability Molecular functions and cellular roles of the ChlR1 (DDX11) helicase defective in the rare cohesinopathy Warsaw breakage syndrome Involvement of global genome repair, transcription coupled repair, and chromatin remodeling in UV DNA damage response changes during development Caenorhabditis elegans HIM-18/SLX-4 interacts with SLX-1 and XPF-1 and maintains genomic integrity in the germline by processing recombination intermediates Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis Poly-G/poly-C tracts in the genomes of Caenorhabditis.Selection for the G4 DNA motif at the 5' end of human genes ZTF-8 interacts with the 9-1-1 complex and is required for DNA damage response and double-strand break repair in the C. elegans germline.Non-homologous end joining plays a key role in transgene concatemer formation in transgenic zebrafish embryos.Caenorhabditis elegans chromatin-associated proteins SET-2 and ASH-2 are differentially required for histone H3 Lys 4 methylation in embryos and adult germ cells.Spectrum of variations in dog-1/FANCJ and mdf-1/MAD1 defective Caenorhabditis elegans strains after long-term propagation.Having a direct look: analysis of DNA damage and repair mechanisms by next generation sequencing.Replication blocking lesions present a unique substrate for homologous recombination.Evidence for the kinetic partitioning of polymerase activity on G-quadruplex DNA.DOG-1 is the Caenorhabditis elegans BRIP1/FANCJ homologue and functions in interstrand cross-link repair FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability Promotion of Homologous Recombination by SWS-1 in Complex with RAD-51 Paralogs in Caenorhabditis elegans.Welcome the family of FANCJ-like helicases to the block of genome stability maintenance proteins.DNA damage leads to progressive replicative decline but extends the life span of long-lived mutant animals.FANCJ helicase operates in the Fanconi Anemia DNA repair pathway and the response to replicational stress.CX-5461 is a DNA G-quadruplex stabilizer with selective lethality in BRCA1/2 deficient tumours A DOG's View of Fanconi Anemia: Insights from C. elegans.The yin and yang of repair mechanisms in DNA structure-induced genetic instability.Genome integrity is regulated by the Caenorhabditis elegans Rad51D homolog rfs-1 Nucleotide Excision Repair in Caenorhabditis elegans.Epigenetic instability due to defective replication of structured DNA.FANCJ is a structure-specific DNA helicase associated with the maintenance of genomic G/C tracts.Mutagenic consequences of a single G-quadruplex demonstrate mitotic inheritance of DNA replication fork barriers.Human Translesion Polymerase κ Exhibits Enhanced Activity and Reduced Fidelity Two Nucleotides from G-Quadruplex DNA.Genomic stability: FANCJ-dependent G4 DNA repair.FANCJ promotes DNA synthesis through G-quadruplex structures.A Polymerase Theta-dependent repair pathway suppresses extensive genomic instability at endogenous G4 DNA sites.MinION-based long-read sequencing and assembly extends the Caenorhabditis elegans reference genome.

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Homologous recombination is required for genome stability in the absence of DOG-1 in Caenorhabditis elegans

http://www.wikidata.org/entity/Q34895210

description

2006 nî lūn-bûn

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2006 թվականի մարտին հրատարակված գիտական հոդված

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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GENETICS.106.056879

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Homologous recombination is re ...... OG-1 in Caenorhabditis elegans

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Ann M Rose

http://www.w3.org/2001/XMLSchema#string

Jillian L Youds

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The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases

The structure-specific endonuclease Ercc1-Xpf is required for targeted gene replacement in embryonic stem cells

Highly prevalent putative quadruplex sequence motifs in human DNA

The genetics of Caenorhabditis elegans

Localization of DNA polymerases eta and iota to the replication machinery is tightly co-ordinated in human cells.

The Bloom's syndrome gene product promotes branch migration of holliday junctions

RAD1, an excision repair gene of Saccharomyces cerevisiae, is also involved in recombination

RAD10, an excision repair gene of Saccharomyces cerevisiae, is involved in the RAD1 pathway of mitotic recombination.

G-quadruplex DNA structures--variations on a theme

The processing of Holliday junctions by BLM and WRN helicases is regulated by p53

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697-708

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scholarly article

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10.1534/GENETICS.106.056879

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2

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173

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Caenorhabditis elegans

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1526509

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