Sulforaphane improves cognitive function administered following traumatic brain injury. - Wikidata - awgldk - TriplyDB

Sulforaphane improves cognitive function administered following traumatic brain injury.

Sulforaphane improves cognitive function administered following traumatic brain injury.

http://www.wikidata.org/entity/Q34986043

about

Sulforaphane as a potential protective phytochemical against neurodegenerative diseases Modulation of oxidative stress and mitochondrial function by the ketogenic diet Dietary Phytochemicals in Neuroimmunoaging: A New Therapeutic Possibility for Humans?Stress-activated cap'n'collar transcription factors in aging and human disease Transfusion of Old RBCs Induces Neuroinflammation and Cognitive Impairment.Supplements, nutrition, and alternative therapies for the treatment of traumatic brain injury.Beneficial Effects of Sulforaphane Treatment in Alzheimer's Disease May Be Mediated through Reduced HDAC1/3 and Increased P75NTR Expression Nrf2 upregulates ATP binding cassette transporter expression and activity at the blood-brain and blood-spinal cord barriers Temporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in mice.Antioxidant therapies in traumatic brain and spinal cord injury Neuroprotective effects of sulforaphane on cholinergic neurons in mice with Alzheimer's disease-like lesions.Sulforaphane protects primary cultures of cortical neurons against injury induced by oxygen-glucose deprivation/reoxygenation via antiapoptosis Reactive oxygen species in the regulation of synaptic plasticity and memory.Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.Dietary broccoli mildly improves neuroinflammation in aged mice but does not reduce lipopolysaccharide-induced sickness behavior The absence of Nrf2 enhances NF-κB-dependent inflammation following scratch injury in mouse primary cultured astrocytes.Neuroprotective effects of sulforaphane after contusive spinal cord injury.Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.Attenuation of β-amyloid-induced oxidative cell death by sulforaphane via activation of NF-E2-related factor 2.Activating the Nrf2-mediated antioxidant response element restores barrier function in the alveolar epithelium of HIV-1 transgenic rats.The role of Nrf2 signaling in the regulation of antioxidants and detoxifying enzymes after traumatic brain injury in rats and mice.The cytoprotective role of the Keap1-Nrf2 pathway.Modulation of Nrf2/ARE pathway by food polyphenols: a nutritional neuroprotective strategy for cognitive and neurodegenerative disorders Natural sulforaphane as a functional chemopreventive agent: including a review of isolation, purification and analysis methods.Novel therapeutic strategies for traumatic brain injury: acute antioxidant reinforcement.Pleiotropic role of PPARγ in intracerebral hemorrhage: an intricate system involving Nrf2, RXR, and NF-κB.DNA Microarray Highlights Nrf2-Mediated Neuron Protection Targeted by Wasabi-Derived Isothiocyanates in IMR-32 Cells.Sulforaphane Ameliorates Okadaic Acid-Induced Memory Impairment in Rats by Activating the Nrf2/HO-1 Antioxidant Pathway.Sulforaphane induces autophagy through ERK activation in neuronal cells.Targeting the NF-E2-Related Factor 2 Pathway: a Novel Strategy for Traumatic Brain Injury.Perspectives on molecular biomarkers of oxidative stress and antioxidant strategies in traumatic brain injury.Sulforaphane protects SK-N-SH cells against antipsychotic-induced oxidative stress.Fractalkine activates NRF2/NFE2L2 and heme oxygenase 1 to restrain tauopathy-induced microgliosis.Intranasal Administration of the Antisecretory Peptide AF-16 Reduces Edema and Improves Cognitive Function Following Diffuse Traumatic Brain Injury in the Rat.Sulforaphane epigenetically enhances neuronal BDNF expression and TrkB signaling pathways.Sulforaphane prevents quinolinic acid-induced mitochondrial dysfunction in rat striatum.A combination antioxidant therapy to inhibit NOX2 and activate Nrf2 decreases secondary brain damage and improves functional recovery after traumatic brain injury.Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.Sulforaphane ameliorates neurobehavioral deficits and protects the brain from amyloid β deposits and peroxidation in mice with Alzheimer-like lesions.Cognition Enhancing Activity of Sulforaphane Against Scopolamine Induced Cognitive Impairment in Zebra Fish (Danio rerio).

P2860

Q21285068-CD9AD36B-5386-46D0-BAC2-0F9F643E9113 Q26824903-F465FE65-CD6F-4013-801E-905B34A709F4 Q28075814-DC51EE4D-BE63-48B7-B281-4F65844C6B96 Q28384783-B00A3482-5238-42D2-A256-D9B4BABDF658 Q30300208-3C6F6E6E-DC43-4AEB-AFAA-B6B0D6FB4B3B Q30393744-42FB0D10-99C2-474A-9280-5FD1F94C0201 Q33619463-B24EBD9F-35C9-40BE-A3E6-8B0421648674 Q33770428-709F913E-945C-4035-AADF-0C3CD39A0025 Q33847548-944B1BFD-00C4-4BF4-A8B4-D11543FDCC39 Q34049949-5B957DF2-DCD6-4D51-8C55-F28F3F142675 Q34158616-FF7530F0-AF62-47C8-ADD3-F67C8DECB242 Q34440320-B72F807E-A5E1-4278-96FA-30089F4F4BB4 Q34805998-3C5C0053-1D89-43DE-A415-941C3FE210F1 Q35071145-35739BA1-C6D4-40D3-B0A5-AD76B726BB21 Q35597126-33C17535-FF5B-4D9E-A518-014DF8BA179D Q35865287-03D48496-0D9E-4169-AFE9-4F8E4A6B59EB Q36390152-6F60C47B-9432-4300-A836-5812CBF2AFC7 Q36674740-0CEA53F9-6B4C-4B93-B66F-232559DBBCA6 Q36995865-FDCF64B6-8A10-4D46-BBF9-418064524D61 Q37094206-54870E34-9CEB-4942-B8D2-1CE1C7E3880D Q37729858-A8E52B86-2731-495E-8B91-B9D9E417AE9E Q37848718-FFD84D07-DEA4-4C8B-8D41-B9BC1369A94A Q37866170-A0FCF74E-8DA6-47B4-8EB7-A309725204CF Q37937673-DB9D967B-3332-4B36-AA20-DDD49812B6EE Q38188467-21C59D2C-5755-4D21-BB69-4DA8EDFD85A2 Q38272127-B9829D7B-A236-4E19-86F1-C0D26C268635 Q38750815-D28FE863-AAD1-4C78-99DE-BA46E79084BA Q38830531-8114F997-95EC-4D02-BE12-1E00000D20B5 Q38982423-B775D9BE-DFBA-40AE-9222-A6323DF38378 Q39146237-32432FCA-6D56-43AD-896C-AA662972EAC6 Q39218449-88B99E39-9DAE-4DC5-8314-B06210D7B2C8 Q39472796-7DA8D1B4-371E-494B-B66C-ACB62BE3C324 Q42252979-45AA5C10-F20A-49F8-9F18-E819F0B72530 Q42321886-0468FAC5-DF88-4E3C-8ED1-C56A47558754 Q43650001-829ECEEB-6F72-49CB-A4A1-8DA8D86D04C2 Q45297224-5B445BAA-112C-44BB-ABC4-770A4D445996 Q46272420-BD312542-5DDC-403E-A38D-0A0D191D6971 Q48300905-E3465EE5-0363-451E-9AE3-02A10C1B9A74 Q48659626-A9C06A8D-524E-4E68-B9E5-B515920D504B Q50507255-BEC666D9-FE93-4B0D-90E8-BDFA496F02B5

P2860

Sulforaphane improves cognitive function administered following traumatic brain injury.

http://www.wikidata.org/entity/Q34986043

description

2009 nî lūn-bûn

@nan

2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Sulforaphane improves cognitive function administered following traumatic brain injury.

@ast

Sulforaphane improves cognitive function administered following traumatic brain injury.

@en

Sulforaphane improves cognitive function administered following traumatic brain injury.

@nl

type

label

Sulforaphane improves cognitive function administered following traumatic brain injury.

@ast

Sulforaphane improves cognitive function administered following traumatic brain injury.

@en

Sulforaphane improves cognitive function administered following traumatic brain injury.

@nl

prefLabel

Sulforaphane improves cognitive function administered following traumatic brain injury.

@ast

Sulforaphane improves cognitive function administered following traumatic brain injury.

@en

Sulforaphane improves cognitive function administered following traumatic brain injury.

@nl

P1433

Q34986043-A5A9E53F-8C88-47D2-8AAB-E14DA1723FDB

P1476

Q34986043-9C38CFFB-4B12-46CE-82E2-782FA23EFDC4

P2093

Q34986043-3DC97297-00F2-4B1B-85F2-3D5D62F8B6A1

Q34986043-4D8CE376-857A-43CD-8065-AF5D3A65CD19

Q34986043-57C99427-2676-4BA3-97FF-50595099B0FF

Q34986043-C1EB9819-B0E7-4A05-9BEA-67FE71862E81

Q34986043-E8B44746-0D0F-461D-BBCC-44E6C9813AF9

P2860

Q34986043-0003A1E9-F754-4205-B386-754252DAEB73

Q34986043-148628F6-982B-463E-9EE8-62A4E1600F64

Q34986043-32FA1077-64A3-466C-A66E-B4A5885A0F27

Q34986043-37CBED0A-12AE-4F77-B78B-563E454EFF64

Q34986043-3941FC48-A94E-45DD-ACC5-657797269D54

Q34986043-47DA2D1E-DA89-4BA3-A7E6-389DF9CCE80D

Q34986043-4F8A2B5C-0379-436F-B79A-B45D44FF6993

Q34986043-62FB6363-7B44-4FCA-BE6D-7A694E9E582C

Q34986043-7379764A-FDA9-414D-AD33-9455469F1BE4

Q34986043-7DA08AC7-829C-4D29-A7A4-CE16DB9DCF97

P304

Q34986043-C857ADDC-753F-49C1-8962-EAAE966B6307

P31

Q34986043-58627A57-AB41-441D-B0FE-AE83B552591D

P356

Q34986043-F5373ED7-B634-4F56-9585-DBFCA236B7B3

P407

Q34986043-FD844002-56A5-4A02-AE65-DFB9B77626F9

P433

Q34986043-08D942FB-8223-4239-8397-FF7F9BB04FF5

P478

Q34986043-D259003C-FD34-4DC0-8B8C-A87F49B16294

P577

Q34986043-ACD83C33-6732-453B-86D9-88F96A456A8B

P5875

Q34986043-65F0A151-A914-4222-831E-F40E1A5418FF

P698

Q34986043-D2496E9A-9201-4CF4-A623-B5555041212F

P921

Q34986043-052D3E2C-65EC-43DD-96EA-BDC829D542D3

P932

Q34986043-8B15ED20-E830-4276-AEDA-3AB1A9FFD242

P356

J.NEULET.2009.04.028

P1433

Neuroscience Letters

P1476

Sulforaphane improves cognitive function administered following traumatic brain injury.

@en

P2093

Anthony N Moore

http://www.w3.org/2001/XMLSchema#string

Jing Zhao

http://www.w3.org/2001/XMLSchema#string

Min Zhang

http://www.w3.org/2001/XMLSchema#string

Pramod K Dash

http://www.w3.org/2001/XMLSchema#string

Sara A Orsi

http://www.w3.org/2001/XMLSchema#string

P2860

Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex.

Identification of Nrf2-regulated genes induced by the chemopreventive agent sulforaphane by oligonucleotide microarray

Nrf2-Keap1 defines a physiologically important stress response mechanism

Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression

Cell death mechanisms following traumatic brain injury.

Neurotrophin-mediated neuroprotection of hippocampal neurons following traumatic brain injury is not associated with acute recovery of hippocampal function

Enhancing expression of Nrf2-driven genes protects the blood brain barrier after brain injury.

Identification of the NF-E2-related factor-2-dependent genes conferring protection against oxidative stress in primary cortical astrocytes using oligonucleotide microarray analysis.

HSP70 heat shock gene regulation during ischemia.

Cognitive deficits following traumatic brain injury produced by controlled cortical impact.

P304

103-107

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.NEULET.2009.04.028

http://www.w3.org/2001/XMLSchema#string

P407

P433

2

http://www.w3.org/2001/XMLSchema#string

P478

460

http://www.w3.org/2001/XMLSchema#string

P577

2009-04-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

26281695

http://www.w3.org/2001/XMLSchema#string

P698

19515491

http://www.w3.org/2001/XMLSchema#string

P921

traumatic brain injury

P932

2700200

http://www.w3.org/2001/XMLSchema#string