Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
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SOD2 Mediates Amifostine-Induced Protection against Glutamate in PC12 Cells.Neuroinflammation in animal models of traumatic brain injury.Targeting the NF-E2-Related Factor 2 Pathway: a Novel Strategy for Traumatic Brain Injury.Edaravone attenuates oxidative stress induced by chronic cerebral hypoperfusion injury: role of ERK/Nrf2/HO-1 signaling pathway.Prognostic value of HMGB1 and oxidative stress markers in multiple trauma patients: A single-centre prospective study.NADPH oxidases in traumatic brain injury - Promising therapeutic targets?
P2860
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@en
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@nl
type
label
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@en
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@nl
prefLabel
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@en
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@nl
P2093
P2860
P1476
Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
@en
P2093
P2860
P2888
P304
P356
10.1007/S10571-015-0167-9
P577
2015-03-03T00:00:00Z