High-mobility group box-1 protein promotes granulomatous nephritis in adenine-induced nephropathy. - Wikidata - awgldk - TriplyDB

High-mobility group box-1 protein promotes granulomatous nephritis in adenine-induced nephropathy.

High-mobility group box-1 protein promotes granulomatous nephritis in adenine-induced nephropathy.

http://www.wikidata.org/entity/Q34987778

about

The role of high mobility group box 1 (HMGB1) in the pathogenesis of kidney diseases Overexpression of receptor for advanced glycation end products and high-mobility group box 1 in human dental pulp inflammation.High-mobility group box-1 protein (HMGB1) is increased in antineutrophilic cytoplasmatic antibody (ANCA)-associated vasculitis with renal manifestations HMGB1 in health and disease.The role of necrotic cell death in the pathogenesis of immune mediated nephropathies.HMGB1 promotes the development of pulmonary arterial hypertension in rats.The high-mobility group box-1 nuclear factor mediates retinal injury after ischemia reperfusion.High mobility group box 1 (HMGB1) and anti-HMGB1 antibodies and their relation to disease characteristics in systemic lupus erythematosus.Urine levels of HMGB1 in Systemic Lupus Erythematosus patients with and without renal manifestations.Soluble Receptor for Advanced Glycation End Product Ameliorates Chronic Intermittent Hypoxia Induced Renal Injury, Inflammation, and Apoptosis via P38/JNK Signaling Pathways.Recombinant human soluble thrombomodulin prevents peripheral HMGB1-dependent hyperalgesia in rats.HMGB1 Promotes Intraoral Palatal Wound Healing through RAGE-Dependent Mechanisms.Capsaicin ameliorates cisplatin-induced renal injury through induction of heme oxygenase-1.Hematological, hepatic, and retinal phenotypes in mice deficient for prolyl hydroxylase domain proteins in the liver HMGB1 in development and diseases of the central nervous system.Role of GLP-1 and DPP-4 in diabetic nephropathy and cardiovascular disease.Regulation of wound healing and organ fibrosis by toll-like receptors.The alarmin functions of high-mobility group box-1 and IL-33 in the pathogenesis of systemic lupus erythematosus.Role of high mobility group box 1 and its signaling pathways in renal diseases.Therapeutic Opportunities in Damage-Associated Molecular Pattern-Driven Metabolic Diseases.Urinary MCP-1、HMGB1 increased in calcium nephrolithiasis patients and the influence of hypercalciuria on the production of the two cytokines.NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy.HMGB1, TGF-β and NF-κB are associated with chronic allograft nephropathy.Chronic kidney disease induces a systemic microangiopathy, tissue hypoxia and dysfunctional angiogenesis.

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P2860

High-mobility group box-1 protein promotes granulomatous nephritis in adenine-induced nephropathy.

http://www.wikidata.org/entity/Q34987778

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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type

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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High-mobility group box-1 prot ...... n adenine-induced nephropathy.

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LABINVEST.2010.64

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Laboratory Investigation

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High-mobility group box-1 prot ...... in adenine-induced nephropathy

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Ikuro Maruyama

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Kamal K Biswas

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Ko-Ichi Kawahara

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Martin Lotz

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Noboru Taniguchi

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Takao Nitanda

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Teruto Hashiguchi

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Tomonori Uchimura

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Yoko Oyama

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Yoshihisa Umekita

http://www.w3.org/2001/XMLSchema#string

P2860

Signaling danger: toll-like receptors and their potential roles in kidney disease

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

The lack of chromosomal protein Hmg1 does not disrupt cell growth but causes lethal hypoglycaemia in newborn mice

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

Real time quantitative PCR

Obstructive nephropathy: towards biomarker discovery and gene therapy.

Advanced glycation end-products induce tubular CTGF via TGF-beta-independent Smad3 signaling.

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labinvest.2010.64

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6

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Salunya Tancharoen

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41969445

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1000123347

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3096665

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