Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
about
The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damageEvaluating biomarkers to model cancer risk post cosmic ray exposureVisualization of complex DNA double-strand breaks in a tumor treated with carbon ion radiotherapy.Establishment of a γ-H2AX foci-based assay to determine biological dose of radon to red bone marrow in ratsDependence and independence of survival parameters on linear energy transfer in cells and tissues.RAD51 interconnects between DNA replication, DNA repair and immunity.Genomic stability in response to high versus low linear energy transfer radiation in Arabidopsis thaliana.Carbon Ion Radiotherapy: A Review of Clinical Experiences and Preclinical Research, with an Emphasis on DNA Damage/RepairIntegrated molecular analysis indicates undetectable change in DNA damage in mice after continuous irradiation at ~ 400-fold natural background radiation.Upregulation of miR-24 is associated with a decreased DNA damage response upon etoposide treatment in highly differentiated CD8(+) T cells sensitizing them to apoptotic cell death.Persistent DNA damage after high dose in vivo gamma exposure of minipig skin.A stochastic model of DNA fragments rejoiningDistinct roles of Ape1 protein, an enzyme involved in DNA repair, in high or low linear energy transfer ionizing radiation-induced cell killing.The role of ATM in the deficiency in nonhomologous end-joining near telomeres in a human cancer cell line.Spatiotemporal dynamics of DNA repair proteins following laser microbeam induced DNA damage - when is a DSB not a DSB?Visualisation of γH2AX foci caused by heavy ion particle traversal; distinction between core track versus non-track damage.Mechanisms of DNA damage response to targeted irradiation in organotypic 3D skin cultures.Modeling damage complexity-dependent non-homologous end-joining repair pathway.Bcl2 inhibits recruitment of Mre11 complex to DNA double-strand breaks in response to high-linear energy transfer radiationEffects of X-ray and carbon ion beam irradiation on membrane permeability and integrity in Saccharomyces cerevisiae cellsDNA end resection is needed for the repair of complex lesions in G1-phase human cells.ATRX contributes to epigenetic asymmetry and silencing of major satellite transcripts in the maternal genome of the mouse embryoStress-induced DNA damage biomarkers: applications and limitations.VE-821, an ATR inhibitor, causes radiosensitization in human tumor cells irradiated with high LET radiationTargeted DNA damage at individual telomeres disrupts their integrity and triggers cell death.Both Complexity and Location of DNA Damage Contribute to Cellular Senescence Induced by Ionizing Radiation.Direct measurement of the 3-dimensional DNA lesion distribution induced by energetic charged particles in a mouse model tissue.FANCD2 influences replication fork processes and genome stability in response to clustered DSBs.Mechanisms of resistance to high and low linear energy transfer radiation in myeloid leukemia cells.Impact of Charged Particle Exposure on Homologous DNA Double-Strand Break Repair in Human Blood-Derived Cells.Mechanistic studies on histone catalyzed cleavage of apyrimidinic/apurinic sites in nucleosome core particlesTranscriptional elongation requires DNA break-induced signalling.HPV16 E6 and E7 proteins induce a chronic oxidative stress response via NOX2 that causes genomic instability and increased susceptibility to DNA damage in head and neck cancer cells.Replication stress induced site-specific phosphorylation targets WRN to the ubiquitin-proteasome pathwayAdenomatous polyposis coli-mediated accumulation of abasic DNA lesions lead to cigarette smoke condensate-induced neoplastic transformation of normal breast epithelial cells.Artemis is required to improve the accuracy of repair of double-strand breaks with 5'-blocked termini generated from non-DSB-clustered lesions.Mechanisms of Cell Killing Response from Low Linear Energy Transfer (LET) Radiation Originating from (177)Lu Radioimmunotherapy Targeting Disseminated Intraperitoneal Tumor XenograftsClustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PKTorin2 Suppresses Ionizing Radiation-Induced DNA Damage RepairThe potential value of the neutral comet assay and γH2AX foci assay in assessing the radiosensitivity of carbon beam in human tumor cell lines.
P2860
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P2860
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@ast
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@en
type
label
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@ast
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@en
prefLabel
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@ast
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@en
P2093
P2860
P356
P1476
Unrepaired clustered DNA lesions induce chromosome breakage in human cells.
@en
P2093
Aroumougame Asaithamby
David J Chen
P2860
P304
P356
10.1073/PNAS.1016045108
P407
P577
2011-04-28T00:00:00Z