Chromosome breakage after G2 checkpoint release - Wikidata - awgldk - TriplyDB

Chromosome breakage after G2 checkpoint release

Chromosome breakage after G2 checkpoint release

http://www.wikidata.org/entity/Q24683383

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Both telomeric and non-telomeric DNA damage are determinants of mammalian cellular senescence Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2 Characterizing the DNA Damage Response by Cell Tracking Algorithms and Cell Features Classification Using High-Content Time-Lapse Analysis DNA Double-Strand Break Repair Pathway Choice Is Directed by Distinct MRE11 Nuclease Activities Asf1 facilitates dephosphorylation of Rad53 after DNA double-strand break repair.Non-homologous end-joining, a sticky affair A mitotic phosphorylation feedback network connects Cdk1, Plk1, 53BP1, and Chk2 to inactivate the G(2)/M DNA damage checkpoint Interference in DNA replication can cause mitotic chromosomal breakage unassociated with double-strand breaks Epstein-Barr virus-encoded latent membrane protein 1 impairs G2 checkpoint in human nasopharyngeal epithelial cells through defective Chk1 activation The HSP90 inhibitor NVP-AUY922 radiosensitizes by abrogation of homologous recombination resulting in mitotic entry with unresolved DNA damage microRNA-34a promotes DNA damage and mitotic catastrophe.Replication independent ATR signalling leads to G2/M arrest requiring Nbs1, 53BP1 and MDC1.Investigation of the functional link between ATM and NBS1 in the DNA damage response in the mouse cerebellum.A genetic screen identifies BRCA2 and PALB2 as key regulators of G2 checkpoint maintenance DNA lesions induced by replication stress trigger mitotic aberration and tetraploidy development G2-phase chromosomal radiosensitivity of primary fibroblasts from hereditary retinoblastoma family members and some apparently normal controls.Homeostatic control of polo-like kinase-1 engenders non-genetic heterogeneity in G2 checkpoint fidelity and timing.Vertebrate cells genetically deficient for Cdc14A or Cdc14B retain DNA damage checkpoint proficiency but are impaired in DNA repair.Escherichia coli induces DNA damage in vivo and triggers genomic instability in mammalian cells The multifunctional SNM1 gene family: not just nucleases Inducible response required for repair of low-dose radiation damage in human fibroblasts.Cul4A is essential for spermatogenesis and male fertility.Error-prone ZW pairing and no evidence for meiotic sex chromosome inactivation in the chicken germ line Opposite roles for p38MAPK-driven responses and reactive oxygen species in the persistence and resolution of radiation-induced genomic instability.Validation of the cell cycle G(2) delay assay in assessing ionizing radiation sensitivity and breast cancer risk.No DNA damage response and negligible genome-wide transcriptional changes in human embryonic stem cells exposed to terahertz radiation Visualisation of γH2AX foci caused by heavy ion particle traversal; distinction between core track versus non-track damage.Unrepaired clustered DNA lesions induce chromosome breakage in human cells.Heavy charged particle radiobiology: using enhanced biological effectiveness and improved beam focusing to advance cancer therapy.The p53 response in single cells is linearly correlated to the number of DNA breaks without a distinct threshold.Give me a break, but not in mitosis: the mitotic DNA damage response marks DNA double-strand breaks with early signaling events Fidelity of end joining in mammalian episomes and the impact of Metnase on joint processing.Bystander communication and cell cycle decisions after DNA damage Pre-exposure to ionizing radiation stimulates DNA double strand break end resection, promoting the use of homologous recombination repair SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells.DNA double-strand break repair is impaired in presenescent Syrian hamster fibroblasts Cellular automaton-based model for radiation-induced bystander effects.Combined haploinsufficiency and genetic control of the G2/M checkpoint in irradiated cells.Radiation-induced double-strand breaks require ATM but not Artemis for homologous recombination during S-phase.

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Chromosome breakage after G2 checkpoint release

http://www.wikidata.org/entity/Q24683383

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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type

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Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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prefLabel

Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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Chromosome breakage after G2 checkpoint release

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Journal of Cell Biology

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Chromosome breakage after G2 checkpoint release

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Andrea Beucher

http://www.w3.org/2001/XMLSchema#string

Andrea Krempler

http://www.w3.org/2001/XMLSchema#string

Dorothee Deckbar

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Julie Birraux

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Leopoldine Tchouandong

http://www.w3.org/2001/XMLSchema#string

Markus Löbrich

http://www.w3.org/2001/XMLSchema#string

Penny Jeggo

http://www.w3.org/2001/XMLSchema#string

Sarah Walker

http://www.w3.org/2001/XMLSchema#string

Tom Stiff

http://www.w3.org/2001/XMLSchema#string

P2860

CENP-F is a protein of the nuclear matrix that assembles onto kinetochores at late G2 and is rapidly degraded after mitosis

Artemis is a phosphorylation target of ATM and ATR and is involved in the G2/M DNA damage checkpoint response

Dynamic assembly of end-joining complexes requires interaction between Ku70/80 and XRCC4

Functional interaction between BLM helicase and 53BP1 in a Chk1-mediated pathway during S-phase arrest

Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

Megabase chromatin domains involved in DNA double-strand breaks in vivo

Structural basis for Chk1 inhibition by UCN-01

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

DNA repair protein Ku80 suppresses chromosomal aberrations and malignant transformation

Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development

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749-55

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scholarly article

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4765677

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10.1083/JCB.200612047

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6

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176

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2007-03-12T00:00:00Z

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6454446

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17353355

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2064048

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