fzr-1 and lin-35/Rb function redundantly to control cell proliferation in C. elegans as revealed by a nonbiased synthetic screen
about
The THAP domain of THAP1 is a large C2CH module with zinc-dependent sequence-specific DNA-binding activityStructure-function analysis of the THAP zinc finger of THAP1, a large C2CH DNA-binding module linked to Rb/E2F pathwaysDifferential expression, localization and activity of two alternatively spliced isoforms of human APC regulator CDH1Involvement of an SCFSlmb complex in timely elimination of E2F upon initiation of DNA replication in DrosophilaCoordinated regulation of intestinal functions in C. elegans by LIN-35/Rb and SLR-2A new role for the GARP complex in microRNA-mediated gene regulationAPC/C and retinoblastoma interaction: cross-talk of retinoblastoma protein with the ubiquitin proteasome pathwayNuclear localization of the cell cycle regulator CDH1 and its regulation by phosphorylationDiverse chromatin remodeling genes antagonize the Rb-involved SynMuv pathways in C. elegansPotential conservation of circadian clock proteins in the phylum Nematoda as revealed by bioinformatic searchesCell growth suppression by thanatos-associated protein 11(THAP11) is mediated by transcriptional downregulation of c-MycSpindle assembly checkpoint genes reveal distinct as well as overlapping expression that implicates MDF-2/Mad2 in postembryonic seam cell proliferation in Caenorhabditis elegansInvasive Cell Fate Requires G1 Cell-Cycle Arrest and Histone Deacetylase-Mediated Changes in Gene ExpressionLarge-scale RNAi screens identify novel genes that interact with the C. elegans retinoblastoma pathway as well as splicing-related components with synMuv B activity.The lin-35/Rb and RNAi pathways cooperate to regulate a key cell cycle transition in C. elegans.Cyclin E and CDK2 repress the terminal differentiation of quiescent cells after asymmetric division in C. elegans.A mechanistic basis for the coordinated regulation of pharyngeal morphogenesis in Caenorhabditis elegans by LIN-35/Rb and UBC-18-ARI-1.A complex regulatory network coordinating cell cycles during C. elegans development is revealed by a genome-wide RNAi screen.Cancer models in Caenorhabditis elegans.Dissection of genetic pathways in C. elegansThe miR-35-41 family of microRNAs regulates RNAi sensitivity in Caenorhabditis elegans.gon-14 functions with class B and class C synthetic multivulva genes to control larval growth in Caenorhabditis elegans.The identities of sym-2, sym-3 and sym-4, three genes that are synthetically lethal with mec-8 in Caenorhabditis elegans.Analysis of genetic interaction networks shows that alternatively spliced genes are highly versatile.lin-8, which antagonizes Caenorhabditis elegans Ras-mediated vulval induction, encodes a novel nuclear protein that interacts with the LIN-35 Rb proteinCaenorhabditis elegans lin-35/Rb, efl-1/E2F and other synthetic multivulva genes negatively regulate the anaphase-promoting complex gene mat-3/APC8.The LIN-15A and LIN-56 transcriptional regulators interact to negatively regulate EGF/Ras signaling in Caenorhabditis elegans vulval cell-fate determination.The cyclin-dependent kinase inhibitors, cki-1 and cki-2, act in overlapping but distinct pathways to control cell cycle quiescence during C. elegans development.The C. elegans NR4A nuclear receptor gene nhr-6 promotes cell cycle progression in the spermatheca lineageOvercoming redundancy: an RNAi enhancer screen for morphogenesis genes in Caenorhabditis elegans.Developmental defects observed in hypomorphic anaphase-promoting complex mutants are linked to cell cycle abnormalitiesAnaphase-promoting complex in Caenorhabditis elegans.DPL-1 DP, LIN-35 Rb and EFL-1 E2F act with the MCD-1 zinc-finger protein to promote programmed cell death in Caenorhabditis elegans.lin-35/Rb and the CoREST ortholog spr-1 coordinately regulate vulval morphogenesis and gonad development in C. elegansThe SynMuv genes of Caenorhabditis elegans in vulval development and beyondmir-35 is involved in intestine cell G1/S transition and germ cell proliferation in C. elegans.A network of genes antagonistic to the LIN-35 retinoblastoma protein of Caenorhabditis elegans.The decapping scavenger enzyme DCS-1 controls microRNA levels in Caenorhabditis elegans.C. elegans AMPKs promote survival and arrest germline development during nutrient stress.toca-1 is in a novel pathway that functions in parallel with a SUN-KASH nuclear envelope bridge to move nuclei in Caenorhabditis elegans.
P2860
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P2860
fzr-1 and lin-35/Rb function redundantly to control cell proliferation in C. elegans as revealed by a nonbiased synthetic screen
description
2002 nî lūn-bûn
@nan
2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
name
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@ast
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@en
type
label
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@ast
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@en
prefLabel
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@ast
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@en
P2093
P2860
P356
P1433
P1476
fzr-1 and lin-35/Rb function r ...... y a nonbiased synthetic screen
@en
P2093
P2860
P304
P356
10.1101/GAD.952302
P577
2002-02-01T00:00:00Z