Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates. - Wikidata - awgldk - TriplyDB

Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates.

Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates.

http://www.wikidata.org/entity/Q35082127

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Cancer as a metabolic disease Mitochondria-controlled signaling mechanisms of brain protection in hypoxia Ischemic A/D transition of mitochondrial complex I and its role in ROS generation Regulation of succinate-ubiquinone reductase and fumarate reductase activities in human complex II by phosphorylation of its flavoprotein subunit Mitochondrial alterations caused by defective peroxisomal biogenesis in a mouse model for Zellweger syndrome (PEX5 knockout mouse)Energetic determinants of tyrosine phosphorylation of focal adhesion proteins during hypoxia/reoxygenation of kidney proximal tubules Partial complex I deficiency due to the CNS conditional ablation of Ndufa5 results in a mild chronic encephalopathy but no increase in oxidative damage.The self-organizing fractal theory as a universal discovery method: the phenomenon of life Multiphoton imaging of the functioning kidney.Fumarate reductase activity maintains an energized membrane in anaerobic Mycobacterium tuberculosis.Cross-talk between mitochondrial malate dehydrogenase and the cytochrome bc1 complex Regulation of the mitochondrial permeability transition in kidney proximal tubules and its alteration during hypoxia-reoxygenation Oxidation of alpha-ketoglutarate is required for reductive carboxylation in cancer cells with mitochondrial defects.Anaerobic and aerobic pathways for salvage of proximal tubules from hypoxia-induced mitochondrial injury.Mitochondrial dysfunction in inherited renal disease and acute kidney injury SUCNR1-mediated chemotaxis of macrophages aggravates obesity-induced inflammation and diabetes.SRT1720 induces mitochondrial biogenesis and rescues mitochondrial function after oxidant injury in renal proximal tubule cells.Therapeutic targeting of the mitochondrial dysfunction in septic acute kidney injury.In vivo multiphoton imaging of mitochondrial structure and function during acute kidney injury.How wasting is saving: weight loss at altitude might result from an evolutionary adaptation.The succinate receptor as a novel therapeutic target for oxidative and metabolic stress-related conditions HIF-1α in the heart: remodeling nucleotide metabolism.Atpenins, potent and specific inhibitors of mitochondrial complex II (succinate-ubiquinone oxidoreductase).Lung tissue bioenergetics and caspase activity in rodents.Mechanisms of organ dysfunction in critical illness: report from a Round Table Conference held in Brussels.Maintenance of homeostasis in the aging hypothalamus: the central and peripheral roles of succinate.Cyclophilin D and the mitochondrial permeability transition in kidney proximal tubules after hypoxic and ischemic injury Substrate modulation of fatty acid effects on energization and respiration of kidney proximal tubules during hypoxia/reoxygenation.Clinical review: anticoagulation for continuous renal replacement therapy--heparin or citrate?Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy Mitochondrial function and disturbances in the septic kidney Manganese porphyrin reduces renal injury and mitochondrial damage during ischemia/reperfusion.Persistent disruption of mitochondrial homeostasis after acute kidney injury.Modeling of Mitochondrial Donut Formation.Visualization of in vivo metabolic flows reveals accelerated utilization of glucose and lactate in penumbra of ischemic heart.Adverse effects of α-ketoglutarate/malate in a rat model of acute kidney injury.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.Tolerance of the human kidney to isolated controlled ischemia.5-HT2 Receptor Regulation of Mitochondrial Genes: Unexpected Pharmacological Effects of Agonists and Antagonists CCAAT/enhancer binding protein-beta is a transcriptional regulator of peroxisome-proliferator-activated receptor-gamma coactivator-1alpha in the regenerating liver.

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P2860

Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates.

http://www.wikidata.org/entity/Q35082127

description

2000 nî lūn-bûn

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2000 թուականի Մարտին հրատարակուած գիտական յօդուած

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2000 թվականի մարտին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年论文

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Mitochondrial dysfunction duri ...... tric acid cycle intermediates.

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Mitochondrial dysfunction duri ...... tric acid cycle intermediates.

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Proceedings of the National Academy of Sciences of the United States of America

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Mitochondrial dysfunction duri ...... tric acid cycle intermediates.

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P2093

I Nissim

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J M Weinberg

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M A Venkatachalam

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N F Roeser

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P2860

From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state

The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

The mitochondrial death/life regulator in apoptosis and necrosis.

Cytoprotective effects of glycine and glutathione against hypoxic injury to renal tubules.

Oxidative phosphorylation and ultrastructural transformation in mitochondria in the intact ascites tumor cell.

Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide.

Mechanisms by which mitochondria transport calcium.

The mitochondrial permeability transition.

Mechanisms of myocardial protection by amino acids: facts and hypotheses.

Anaerobic rat heart. Effects of glucose and tricarboxylic acid-cycle metabolites on metabolism and physiological performance.

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2826-2831

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