NMDA receptor antagonism in the lateral/basolateral but not central nucleus of the amygdala prevents the induction of facilitated learning in response to stress.
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Stress reduction correlates with structural changes in the amygdalaAn animal model of emotional blunting in schizophrenia.The bed nucleus of the stria terminalis is critically involved in enhancing associative learning after stressful experienceCritical brain circuits at the intersection between stress and learningMetabotropic glutamate receptor 5/Homer interactions underlie stress effects on fearThe effects of two forms of physical activity on eyeblink classical conditioning.Neuroprotective fractalkine in fetal alcohol syndrome.Basolateral amygdala noradrenergic influence enables enhancement of memory consolidation induced by hippocampal glucocorticoid receptor activation.Regulation of ERK Kinase by MEK1 Kinase Inhibition in the BrainAcute exposure to stress improves performance in trace eyeblink conditioning and spatial learning tasks in healthy menGlucocorticoids are necessary for enhancing the acquisition of associative memories after acute stressful experienceStressful experience and learning across the lifespan.Distinctive stress effects on learning during pubertyThe hippocampus is necessary for enhancements and impairments of learning following stressThe same antidepressant elicits contrasting patterns of synaptic changes in the amygdala vs hippocampusEarly life stress elicits visceral hyperalgesia and functional reorganization of pain circuits in adult rats.Formation of aversive memories associated with conditioned drug withdrawal requires BDNF expression in the amygdala in acute morphine-dependent rats.The bed nucleus of the stria terminalis modulates learning after stress in masculinized but not cycling femalesThe basolateral nucleus of the amygdala is necessary to induce the opposing effects of stressful experience on learning in males and females.Alterations in phosphorylated cyclic adenosine monophosphate response element of binding protein activity: a pathway for fetal alcohol syndrome-related neurotoxicity.Stress enhances fear by forming new synapses with greater capacity for long-term potentiation in the amygdala.Acute stress causes rapid synaptic insertion of Ca2+ -permeable AMPA receptors to facilitate long-term potentiation in the hippocampus.Factors that determine the non-linear amygdala influence on hippocampus-dependent memory.Previous stress attenuates the susceptibility to Midazolam's disruptive effect on fear memory reconsolidation: influence of pre-reactivation D-cycloserine administration.
P2860
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P2860
NMDA receptor antagonism in the lateral/basolateral but not central nucleus of the amygdala prevents the induction of facilitated learning in response to stress.
description
1998 nî lūn-bûn
@nan
1998 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
NMDA receptor antagonism in th ...... earning in response to stress.
@ast
NMDA receptor antagonism in th ...... earning in response to stress.
@en
type
label
NMDA receptor antagonism in th ...... earning in response to stress.
@ast
NMDA receptor antagonism in th ...... earning in response to stress.
@en
prefLabel
NMDA receptor antagonism in th ...... earning in response to stress.
@ast
NMDA receptor antagonism in th ...... earning in response to stress.
@en
P2860
P1433
P1476
NMDA receptor antagonism in th ...... earning in response to stress.
@en
P2093
P2860
P304
P577
1998-07-01T00:00:00Z