Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration. - Wikidata - awgldk - TriplyDB

Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration.

Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration.

http://www.wikidata.org/entity/Q35160651

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Inflammation and its role in age-related macular degeneration Parainflammation, chronic inflammation, and age-related macular degeneration Molecular pathology of macrophages and interleukin-17 in age-related macular degeneration Genetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Systemic frequencies of T helper 1 and T helper 17 cells in patients with age-related macular degeneration: A case-control study.Association between aqueous humor and vitreous fluid levels of Th17 cell-related cytokines in patients with proliferative diabetic retinopathy.Immune responses in age-related macular degeneration and a possible long-term therapeutic strategy for prevention Genetics of age-related macular degeneration: application to drug design Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.FPR1 interacts with CFH, HTRA1 and smoking in exudative age-related macular degeneration and polypoidal choroidal vasculopathy.Prevalence of anti-retinal autoantibodies in different stages of Age-related macular degeneration.Inflammation and Cell Death in Age-Related Macular Degeneration: An Immunopathological and Ultrastructural Model.T cells and macrophages responding to oxidative damage cooperate in pathogenesis of a mouse model of age-related macular degeneration Complement activation and choriocapillaris loss in early AMD: implications for pathophysiology and therapy.Interleukin-17 retinotoxicity is prevented by gene transfer of a soluble interleukin-17 receptor acting as a cytokine blocker: implications for age-related macular degeneration.CFH Y402H polymorphism is associated with elevated vitreal GM-CSF and choroidal macrophages in the postmortem human eye.A comparative analysis of C57BL/6J and 6N substrains; chemokine/cytokine expression and susceptibility to laser-induced choroidal neovascularization.Inflammatory Mechanisms of Age-related Macular Degeneration.Immunoactivation at the crossroads of human disease Basic biology and role of interleukin-17 in immunity and inflammation Connecting the innate and adaptive immune responses in mouse choroidal neovascularization via the anaphylatoxin C5a and γδT-cells.Anti-inflammatory recombinant TSG-6 stabilizes the progression of focal retinal degeneration in a murine model.Immune mechanisms in inflammatory and degenerative eye disease.T-helper-associated cytokines expression by peripheral blood mononuclear cells in patients with polypoidal choroidal vasculopathy and age-related macular degeneration Choroidal Neovascularization Is Inhibited in Splenic-Denervated or Splenectomized Mice with a Concomitant Decrease in Intraocular Macrophage Responses of Multipotent Retinal Stem Cells to IL-1β, IL-18, or IL-17 Alterations in Circulating Immune Cells in Neovascular Age-Related Macular Degeneration.Suggestive association between PLA2G12A single nucleotide polymorphism rs2285714 and response to anti-vascular endothelial growth factor therapy in patients with exudative age-related macular degeneration.Hypomethylation of the IL17RC promoter associates with age-related macular degeneration.Functional single nucleotide polymorphism in IL-17A 3' untranslated region is targeted by miR-4480 in vitro and may be associated with age-related macular degeneration.Regulatory effects of C5a on IL-17A, IL-17F, and IL-23 Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration: Complement activation in AMD.Interrelationship Between Periapical Lesion and Systemic Metabolic Disorders.The role of the immune response in age-related macular degeneration Genetic and Epigenetic Regulation in Age-related Macular Degeneration.Idiopathic Choroidal Neovascularization: Intraocular Inflammatory Cytokines and the Effect of Intravitreal Ranibizumab Treatment.Aging is not a disease: distinguishing age-related macular degeneration from aging.Pigment epithelium-derived factor reduces apoptosis and pro-inflammatory cytokine gene expression in a murine model of focal retinal degeneration.Evaluating Potential Therapies in a Mouse Model of Focal Retinal Degeneration with Age-related Macular Degeneration (AMD)-Like Lesions.An Ocular Protein Triad Can Classify Four Complex Retinal Diseases.

P2860

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P2860

Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration.

http://www.wikidata.org/entity/Q35160651

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2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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Complement component C5a promo ...... -related macular degeneration.

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Complement component C5a promo ...... -related macular degeneration.

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type

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Complement component C5a promo ...... -related macular degeneration.

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Complement component C5a promo ...... -related macular degeneration.

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Complement component C5a promo ...... -related macular degeneration.

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Complement component C5a promo ...... -related macular degeneration.

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Journal of Translational Medicine

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Complement component C5a promo ...... -related macular degeneration.

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Baoying Liu

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Catherine Meyerle

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Frederick Ferris

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Jingsheng Tuo

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Lai Wei

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Michael L Klein

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Sagarika Chakrabarty

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Zhiyu Li

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P2860

Drusen complement components C3a and C5a promote choroidal neovascularization

A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration

Complement factor H polymorphism in age-related macular degeneration

Ranibizumab and bevacizumab for neovascular age-related macular degeneration

Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Systemic complement activation in age-related macular degeneration

Complement factor H polymorphism and age-related macular degeneration

Development, cytokine profile and function of human interleukin 17-producing helper T cells

Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

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10.1186/1479-5876-9-111

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Robert B Nussenblatt

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