Met receptor signaling is required for sensory nerve development and HGF promotes axonal growth and survival of sensory neurons.
about
The Gab1 PH domain is required for localization of Gab1 at sites of cell-cell contact and epithelial morphogenesis downstream from the met receptor tyrosine kinaseBiology of MET: a double life between normal tissue repair and tumor progressionHGF/SF-met signaling in the control of branching morphogenesis and invasionMET receptor tyrosine kinase controls dendritic complexity, spine morphogenesis, and glutamatergic synapse maturation in the hippocampusCombined drug action of 2-phenylimidazo[2,1-b]benzothiazole derivatives on cancer cells according to their oncogenic molecular signaturesViable hypomorphic signaling mutant of the Met receptor reveals a role for hepatocyte growth factor in postnatal cerebellar developmentMeteorin: a secreted protein that regulates glial cell differentiation and promotes axonal extensionStromal factors SDF1α, sFRP1, and VEGFD induce dopaminergic neuron differentiation of human pluripotent stem cellsHepatocyte growth factor and c-Met promote dendritic maturation during hippocampal neuron differentiation via the Akt pathwayMolecular basis of semaphorin-mediated axon guidanceExpression of c-Met in developing rat hippocampus: evidence for HGF as a neurotrophic factor for calbindin D-expressing neurons.Src homology 2-containing inositol 5-phosphatase 1 binds to the multifunctional docking site of c-Met and potentiates hepatocyte growth factor-induced branching tubulogenesis.Magic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis.Hepatocyte growth factor, a versatile signal for developing neurons.The tyrosine phosphatase SHP-2 is required for sustained activation of extracellular signal-regulated kinase and epithelial morphogenesis downstream from the met receptor tyrosine kinase.Emerging molecular targets in oncology: clinical potential of MET/hepatocyte growth-factor inhibitors.Prediction of regulatory networks in mouse abdominal wallPlasticity versus specificity in RTK signalling modalities for distinct biological outcomes in motor neurons.A tandem repeat of a fragment of Listeria monocytogenes internalin B protein induces cell survival and proliferationGonadotropin-releasing hormone (GnRH) neuron migration: initiation, maintenance and cessation as critical steps to ensure normal reproductive functionMembrane targeting of Grb2-associated binder-1 (Gab1) scaffolding protein through Src myristoylation sequence substitutes for Gab1 pleckstrin homology domain and switches an epidermal growth factor response to an invasive morphogenic program.Spatial phosphoprotein profiling reveals a compartmentalized extracellular signal-regulated kinase switch governing neurite growth and retractionEnhanced neuronal Met signalling levels in ALS mice delay disease onset.Motor neuron trophic factors: therapeutic use in ALS?Intracellular control of developmental and regenerative axon growth.Localization of Magic-F1 transgene, involved in muscular hypertrophy, during early myogenesisDouble-blind, placebo-controlled study of HGF gene therapy in diabetic neuropathy.Tissue-Specific Gain of RTK Signalling Uncovers Selective Cell Vulnerability during Embryogenesis.Hepatocyte Growth Factor and MET Support Mouse Enteric Nervous System Development, the Peristaltic Response, and Intestinal Epithelial Proliferation in Response to Injury.Resistance to targeted cancer drugs through hepatocyte growth factor signalingNecrosis- and apoptosis-related Met cleavages have divergent functional consequencesPhase 1/2 open-label dose-escalation study of plasmid DNA expressing two isoforms of hepatocyte growth factor in patients with painful diabetic peripheral neuropathy.The autism-associated MET receptor tyrosine kinase engages early neuronal growth mechanism and controls glutamatergic circuits development in the forebrainAge dependent forebrain structural changes in mice deficient in the autism associated gene Met tyrosine kinase.Down-regulation of the met receptor tyrosine kinase by presenilin-dependent regulated intramembrane proteolysis.Vascular endothelial growth factor controls neuronal migration and cooperates with Sema3A to pattern distinct compartments of the facial nerve.MET receptor variant R970C favors calpain-dependent generation of a fragment promoting epithelial cell scattering.MET receptor tyrosine kinase as an autism genetic risk factor.MET and Small-Cell Lung Cancer.Promise and challenges on the horizon of MET-targeted cancer therapeutics
P2860
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P2860
Met receptor signaling is required for sensory nerve development and HGF promotes axonal growth and survival of sensory neurons.
description
1997 nî lūn-bûn
@nan
1997 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Met receptor signaling is requ ...... d survival of sensory neurons.
@ast
Met receptor signaling is requ ...... d survival of sensory neurons.
@en
type
label
Met receptor signaling is requ ...... d survival of sensory neurons.
@ast
Met receptor signaling is requ ...... d survival of sensory neurons.
@en
prefLabel
Met receptor signaling is requ ...... d survival of sensory neurons.
@ast
Met receptor signaling is requ ...... d survival of sensory neurons.
@en
P2093
P2860
P356
P1433
P1476
Met receptor signaling is requ ...... nd survival of sensory neurons
@en
P2093
P2860
P304
P356
10.1101/GAD.11.24.3341
P50
P577
1997-12-01T00:00:00Z