Both influenza-induced neutrophil dysfunction and neutrophil-independent mechanisms contribute to increased susceptibility to a secondary Streptococcus pneumoniae infection.
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Role of Autophagy and Apoptosis in the Postinfluenza Bacterial PneumoniaThe role of respiratory viruses in the etiology of bacterial pneumonia: An ecological perspectiveModeling Influenza Virus Infection: A Roadmap for Influenza ResearchInfluenza and Bacterial Superinfection: Illuminating the Immunologic Mechanisms of DiseaseRegulating the adaptive immune response to respiratory virus infectionPlatelet-activating factor receptor plays a role in lung injury and death caused by Influenza A in miceInnate immunity to influenza virus: implications for future therapyThe immunology of influenza virus-associated bacterial pneumoniaThe host immune dynamics of pneumococcal colonization: implications for novel vaccine developmentLinezolid has unique immunomodulatory effects in post-influenza community acquired MRSA pneumoniaInteraction between influenza virus and Streptococcus pneumoniae in severe pneumonia.Protecting against post-influenza bacterial pneumonia by increasing phagocyte recruitment and ROS production.Influenza vaccines to control influenza-associated bacterial infection: where do we stand?Bench-to-bedside review: bacterial pneumonia with influenza - pathogenesis and clinical implications.Postinfluenza bacterial pneumonia: host defenses gone awry.Exogenous Activation of Invariant Natural Killer T Cells by α-Galactosylceramide Reduces Pneumococcal Outgrowth and Dissemination PostinfluenzaPreventing and treating secondary bacterial infections with antiviral agentsCharacterization of the nasopharyngeal microbiota in health and during rhinovirus challengeKinetics of coinfection with influenza A virus and Streptococcus pneumoniae.Naturally-acquired influenza-specific CD4+ T-cell proliferative responses are impaired in HIV-infected African adults.Increased susceptibility for superinfection with Streptococcus pneumoniae during influenza virus infection is not caused by TLR7-mediated lymphopenia.Respiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.Innate immunity against bacterial infection following hyperoxia exposure is impaired in NRF2-deficient mice.Influenza virus infection decreases tracheal mucociliary velocity and clearance of Streptococcus pneumoniaeViral and bacterial interactions in the upper respiratory tractCoinfection with Streptococcus pneumoniae negatively modulates the size and composition of the ongoing influenza-specific CD8⁺ T cell responseTranscriptional adaptation of pneumococci and human pharyngeal cells in the presence of a virus infection.Influenza virus primes mice for pneumonia from Staphylococcus aureus.The methyltransferase Setdb2 mediates virus-induced susceptibility to bacterial superinfection.Lethal coinfection of influenza virus and Streptococcus pneumoniae lowers antibody response to influenza virus in lung and reduces numbers of germinal center B cells, T follicular helper cells, and plasma cells in mediastinal lymph Node.Lowering the threshold of lung innate immune cell activation alters susceptibility to secondary bacterial superinfectionEarliest innate immune responses require macrophage RelA during pneumococcal pneumonia.The role of the local microbial ecosystem in respiratory health and disease.Deletions in the neuraminidase stalk region of H2N2 and H9N2 avian influenza virus subtypes do not affect postinfluenza secondary bacterial pneumonia.Influenza-induced type I interferon enhances susceptibility to gram-negative and gram-positive bacterial pneumonia in miceTRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.Extremes of Interferon-Stimulated Gene Expression Associate with Worse Outcomes in the Acute Respiratory Distress Syndrome.Pulmonary immunostimulation with MALP-2 in influenza virus-infected mice increases survival after pneumococcal superinfectionImpact of Type I and III Interferons on Respiratory Superinfections Due to Multidrug-Resistant Pathogens.Type I interferon induction during influenza virus infection increases susceptibility to secondary Streptococcus pneumoniae infection by negative regulation of γδ T cells
P2860
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P2860
Both influenza-induced neutrophil dysfunction and neutrophil-independent mechanisms contribute to increased susceptibility to a secondary Streptococcus pneumoniae infection.
description
2006 nî lūn-bûn
@nan
2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@ast
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@en
type
label
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@ast
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@en
prefLabel
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@ast
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@en
P2860
P356
P1476
Both influenza-induced neutrop ...... tococcus pneumoniae infection.
@en
P2093
Allen G Harmsen
Lynnelle A McNamee
P2860
P304
P356
10.1128/IAI.00789-06
P407
P577
2006-09-18T00:00:00Z