Evidence that vasopressin V1b receptors mediate the transition to excessive drinking in ethanol-dependent rats. - Wikidata - awgldk - TriplyDB

Evidence that vasopressin V1b receptors mediate the transition to excessive drinking in ethanol-dependent rats.

Evidence that vasopressin V1b receptors mediate the transition to excessive drinking in ethanol-dependent rats.

http://www.wikidata.org/entity/Q35228123

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The relationship of appetitive, reproductive and posterior pituitary hormones to alcoholism and craving in humans Individual differences in gene expression of vasopressin, D2 receptor, POMC and orexin: vulnerability to relapse to heroin-seeking in rats Self-administration of ethanol, cocaine, or nicotine does not decrease the soma size of ventral tegmental area dopamine neurons A Phase 2, Double-Blind, Placebo-Controlled Randomized Trial Assessing the Efficacy of ABT-436, a Novel V1b Receptor Antagonist, for Alcohol Dependence.Operant alcohol self-administration in dependent rats: focus on the vapor model Applications of schedule-induced polydipsia in rodents for the study of an excessive ethanol intake phenotype.The dark side of emotion: the addiction perspective Dendritic remodeling of hippocampal neurons is associated with altered NMDA receptor expression in alcohol dependent rats Chronic nicotine activates stress/reward-related brain regions and facilitates the transition to compulsive alcohol drinking.Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.Chronic treatment with the vasopressin 1b receptor antagonist SSR149415 prevents the dysphoria associated with nicotine withdrawal in rats Glucocorticoid receptor antagonism decreases alcohol seeking in alcohol-dependent individuals.Divergent regulation of distinct glucocorticoid systems in alcohol dependence.Adolescent alcohol exposure alters the rat adult hypothalamic-pituitary-adrenal axis responsiveness in a sex-specific manner.Addiction is a Reward Deficit and Stress Surfeit Disorder.Addiction as a stress surfeit disorder.Optogenetic stimulation of VTA dopamine neurons reveals that tonic but not phasic patterns of dopamine transmission reduce ethanol self-administration.Escalation of drug self-administration as a hallmark of persistent addiction liability.Neuronal extracellular signal-regulated kinase (ERK) activity as marker and mediator of alcohol and opioid dependence.Postdependent state in rats as a model for medication development in alcoholism.Emerging pharmacotherapies for alcohol use disorder.The Epigenetic Regulation of GATA4-Dependent Brain Natriuretic Peptide Expression during Alcohol Withdrawal.Alcohol Dependence Disrupts Amygdalar L-Type Voltage-Gated Calcium Channel Mechanisms.Antireward, compulsivity, and addiction: seminal contributions of Dr. Athina Markou to motivational dysregulation in addiction.Differential Roles for L-Type Calcium Channel Subtypes in Alcohol Dependence.Dysregulation of Brain Stress Systems Mediates Compulsive Alcohol Drinking.Persistent increase in hypothalamic arginine vasopressin gene expression during protracted withdrawal from chronic escalating-dose cocaine in rodents.Genetic and Pharmacologic Manipulation of TLR4 Has Minimal Impact on Ethanol Consumption in Rodents.Exploring the Neurochemical Basis of Alcohol Addiction-Related Behaviors: Translational Research.Increased anxiety, voluntary alcohol consumption and ethanol-induced place preference in mice following chronic psychosocial stress.Cluster and meta-analyses on factors influencing stress-induced alcohol drinking and relapse in rodents.Advances in Pharmacotherapy Development: Human Clinical Studies.Oxytocin Reduces Ethanol Self-Administration in Mice.Oxytocin Reduces Alcohol Cue-Reactivity in Alcohol-Dependent Rats and Humans.V1b Receptor Antagonist SSR149415 and Naltrexone Synergistically Decrease Excessive Alcohol Drinking in Male and Female Mice.Bidirectional relationship between alcohol intake and sensitivity to social defeat: association with Tacr1 and Avp expression.Single-Dose Interaction Study of the Arginine Vasopressin Type 1B Receptor Antagonist ABT-436 and Alcohol in Moderate Alcohol Drinkers.Involvement of activated brain stress responsive systems in excessive and "relapse" alcohol drinking in rodent models: implications for therapeutics.Targeting Stress Pathophysiology to Improve Alcoholism Relapse Outcomes.The influence of adolescent nicotine exposure on ethanol intake and brain gene expression.

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Evidence that vasopressin V1b receptors mediate the transition to excessive drinking in ethanol-dependent rats.

http://www.wikidata.org/entity/Q35228123

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

@ast

Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

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type

label

Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

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Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

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prefLabel

Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

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Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

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J.1369-1600.2010.00291.X

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Addiction Biology

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Evidence that vasopressin V1b ...... ing in ethanol-dependent rats.

@en

P2093

Edward Roberts

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George F Koob

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Miguel Guerrero

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Ola M Ghoneim

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Scott Edwards

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P2860

Vasopressin-induced von Willebrand factor secretion from endothelial cells involves V2 receptors and cAMP

Vasopressin and Oxytocin Excite Distinct Neuronal Populations in the Central Amygdala

Synthesis, transport, and release of posterior pituitary hormones

Anxiolytic- and antidepressant-like effects of the non-peptide vasopressin V1b receptor antagonist, SSR149415, suggest an innovative approach for the treatment of stress-related disorders.

A role for brain stress systems in addiction.

Pharmacological evidence for a motivational role of kappa-opioid systems in ethanol dependence

The hypothalamic-pituitary-adrenal axis response to stress in mice lacking functional vasopressin V1b receptors

Stress and disorders of the stress system

Transcriptional regulation of the pituitary vasopressin V1b receptor involves a GAGA-binding protein

The vasopressin receptors colocalize with vasopressin in the magnocellular neurons of the rat supraoptic nucleus and are modulated by water balance

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