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Brain injury impairs dentate gyrus inhibitory efficacy

Brain injury impairs dentate gyrus inhibitory efficacy

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Traumatic Brain Injury and Neuronal Functionality Changes in Sensory Cortex Making Waves in the Brain: What Are Oscillations, and Why Modulating Them Makes Sense for Brain Injury Mechanisms of GABAergic homeostatic plasticity Modulation of neuronal activity by phosphorylation of the K-Cl cotransporter KCC2 Abnormal chloride homeostasis in the substancia nigra pars reticulata contributes to locomotor deficiency in a model of acute liver injury Sensory cortex underpinnings of traumatic brain injury deficits Disrupted dentate granule cell chloride regulation enhances synaptic excitability during development of temporal lobe epilepsy.Exendin-4, a glucagon-like peptide-1 receptor agonist prevents mTBI-induced changes in hippocampus gene expression and memory deficits in mice Excitatory and inhibitory synaptic connectivity to layer V fast-spiking interneurons in the freeze lesion model of cortical microgyria.Dietary branched chain amino acids ameliorate injury-induced cognitive impairment.Novel repression of Kcc2 transcription by REST-RE-1 controls developmental switch in neuronal chloride.Traumatic brain injury-induced ependymal ciliary loss decreases cerebral spinal fluid flow.Ovariectomy modulation of morphine analgesia of neuropathic pain is associated with the change of K(+)-Cl(-) cotransporter 2 protein level in spinal dorsal horn.Expression of GABA receptor subunits in the hippocampus and thalamus after experimental traumatic brain injury Cation-chloride cotransporters in neuronal development, plasticity and disease Imipramine treatment improves cognitive outcome associated with enhanced hippocampal neurogenesis after traumatic brain injury in mice Loss of cation-chloride cotransporter expression in preterm infants with white matter lesions: implications for the pathogenesis of epilepsy Efficacy, dosage, and duration of action of branched chain amino Acid therapy for traumatic brain injury.Calcium-activated chloride channels (CaCCs) regulate action potential and synaptic response in hippocampal neurons.Traumatic alterations in GABA signaling disrupt hippocampal network activity in the developing brain.Prenatal Hypoxia-Ischemia Induces Abnormalities in CA3 Microstructure, Potassium Chloride Co-Transporter 2 Expression and Inhibitory Tone.Rapamycin down-regulates KCC2 expression and increases seizure susceptibility to convulsants in immature rats.Detrimental effect of post Status Epilepticus treatment with ROCK inhibitor Y-27632 in a pilocarpine model of temporal lobe epilepsy.Cortical excitation and inhibition following focal traumatic brain injury.Proteomics: in pursuit of effective traumatic brain injury therapeutics.Decoding hippocampal signaling deficits after traumatic brain injury Intracellular zinc inhibits KCC2 transporter activity Feed-forward inhibition as a buffer of the neuronal input-output relation.Imaging and serum biomarkers reflecting the functional efficacy of extended erythropoietin treatment in rats following infantile traumatic brain injury.Diffuse traumatic brain injury and the sensory brain.The K(+)-Cl(-) Cotransporter KCC2 and Chloride Homeostasis: Potential Therapeutic Target in Acute Central Nervous System Injury.Chloride co-transporters as possible therapeutic targets for stroke.Pathophysiology and Treatment of Memory Dysfunction After Traumatic Brain Injury.The Small-Molecule TrkB Agonist 7, 8-Dihydroxyflavone Decreases Hippocampal Newborn Neuron Death After Traumatic Brain Injury.Primary blast injury causes cognitive impairments and hippocampal circuit alterations.Chronic stress shifts the GABA reversal potential in the hippocampus and increases seizure susceptibility.Moderate traumatic brain injury promotes proliferation of quiescent neural progenitors in the adult hippocampus.Post-Injury Treatment of 7,8-Dihydroxyflavone Promotes Neurogenesis in the Hippocampus of the Adult Mouse.Network dynamics during the progression of seizure-like events in the hippocampal-parahippocampal regions.Alterations in Hippocampal Network Activity after In Vitro Traumatic Brain Injury.

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Brain injury impairs dentate gyrus inhibitory efficacy

http://www.wikidata.org/entity/Q35242333

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2006 nî lūn-bûn

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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Brain injury impairs dentate gyrus inhibitory efficacy

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Brain injury impairs dentate gyrus inhibitory efficacy

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Brain injury impairs dentate gyrus inhibitory efficacy

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Brain injury impairs dentate gyrus inhibitory efficacy

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J.NBD.2006.09.002

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Neurobiology of Disease

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Brain injury impairs dentate gyrus inhibitory efficacy

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Akiva S Cohen

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David P Bonislawski

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Elizabeth P Schwarzbach

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P2860

Loss of recent memory after bilateral hippocampal lesions

The K+/Cl- co-transporter KCC2 renders GABA hyperpolarizing during neuronal maturation

Reduction of KCC2 expression and GABAA receptor-mediated excitation after in vivo axonal injury

Functional characterization of the neuronal-specific K-Cl cotransporter: implications for [K+]o regulation

The dentate gyrus as a control point for seizures in the hippocampus and beyond.

Theta rhythm of hippocampal CA1 neuron activity: gating by GABAergic synaptic depolarization.

Early and late posttraumatic seizures in traumatic brain injury rehabilitation patients: brain injury factors causing late seizures and influence of seizures on long-term outcome.

Human amnesia and the medial temporal region: enduring memory impairment following a bilateral lesion limited to field CA1 of the hippocampus.

Three cases of enduring memory impairment after bilateral damage limited to the hippocampal formation.

A population-based study of seizures after traumatic brain injuries.

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