Hyperglycemia-induced O-GlcNAcylation and truncation of 4E-BP1 protein in liver of a mouse model of type 1 diabetes. - Wikidata - awgldk - TriplyDB

Hyperglycemia-induced O-GlcNAcylation and truncation of 4E-BP1 protein in liver of a mouse model of type 1 diabetes.

Hyperglycemia-induced O-GlcNAcylation and truncation of 4E-BP1 protein in liver of a mouse model of type 1 diabetes.

http://www.wikidata.org/entity/Q35310496

about

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction mTORC1 and JNK coordinate phosphorylation of the p70S6K1 autoinhibitory domain in skeletal muscle following functional overloading.Regulated in development and DNA damage 1 is necessary for hyperglycemia-induced vascular endothelial growth factor expression in the retina of diabetic rodents Mechanistic target of rapamycin complex 1 (mTORC1)-mediated phosphorylation is governed by competition between substrates for interaction with raptor.Hyperglycemia mediates a shift from cap-dependent to cap-independent translation via a 4E-BP1-dependent mechanism.The role of the unfolded protein response in diabetes mellitus.Induction of REDD1 gene expression in the liver in response to endoplasmic reticulum stress is mediated through a PERK, eIF2α phosphorylation, ATF4-dependent cascade.Emerging roles of RNA-binding proteins in diabetes and their therapeutic potential in diabetic complications.Real Talk: The Inter-play Between the mTOR, AMPK, and Hexosamine Biosynthetic Pathways in Cell Signaling

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P2860

Hyperglycemia-induced O-GlcNAcylation and truncation of 4E-BP1 protein in liver of a mouse model of type 1 diabetes.

http://www.wikidata.org/entity/Q35310496

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

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Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

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type

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Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

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Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

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Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

@ast

Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

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P356

JBC.M111.259457

P1433

Journal of Biological Chemistry

P1476

Hyperglycemia-induced O-GlcNAc ...... ouse model of type 1 diabetes.

@en

P2093

Leonard S Jefferson

http://www.w3.org/2001/XMLSchema#string

Michael D Dennis

http://www.w3.org/2001/XMLSchema#string

Scot R Kimball

http://www.w3.org/2001/XMLSchema#string

Tabitha L Schrufer

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance

Crosstalk between O-GlcNAcylation and proteolytic cleavage regulates the host cell factor-1 maturation pathway

Hexosamines, insulin resistance, and the complications of diabetes: current status.

Mapping sites of O-GlcNAc modification using affinity tags for serine and threonine post-translational modifications

Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes

Glucagon represses signaling through the mammalian target of rapamycin in rat liver by activating AMP-activated protein kinase

4E-BP1, a repressor of mRNA translation, is phosphorylated and inactivated by the Akt(PKB) signaling pathway

Modification of p53 with O-linked N-acetylglucosamine regulates p53 activity and stability.

Dynamic interplay between O-linked N-acetylglucosaminylation and glycogen synthase kinase-3-dependent phosphorylation.

O-linked N-acetylglucosamine modification of insulin receptor substrate-1 occurs in close proximity to multiple SH2 domain binding motifs.

P304

34286-34297

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scholarly article

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10.1074/JBC.M111.259457

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39

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286

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Sarah K. Bronson

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2011-08-12T00:00:00Z

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P698

21840999

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P921

type-1 diabetes

P932

3190771

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