Substrate selectivity of epidermal growth factor-receptor ligand sheddases and their regulation by phorbol esters and calcium influx. - Wikidata - awgldk - TriplyDB

Substrate selectivity of epidermal growth factor-receptor ligand sheddases and their regulation by phorbol esters and calcium influx.

Substrate selectivity of epidermal growth factor-receptor ligand sheddases and their regulation by phorbol esters and calcium influx.

http://www.wikidata.org/entity/Q35545249

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TRP channel regulates EGFR signaling in hair morphogenesis and skin barrier formation Epidermal growth factor receptor ligands as new extracellular targets for the metastasis-promoting S100A4 protein Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling Staphylococcus aureus α-toxin: nearly a century of intrigue Targeting EGFR signalling in chronic lung disease: therapeutic challenges and opportunities Phosphatidylserine exposure is required for ADAM17 sheddase function Cytoplasmic relaxation of active Eph controls ephrin shedding by ADAM10 Rhomboids, signalling and cell biology A Disintegrin and Metalloprotease 17 in the Cardiovascular and Central Nervous Systems ADAM9 is involved in pathological retinal neovascularization Proteomic identification of ADAM12 as a regulator for TGF-β1-induced differentiation of human mesenchymal stem cells to smooth muscle cells Phenotypic diversity of breast cancer-related mutations in metalloproteinase-disintegrin ADAM12 PKCα and PKCδ regulate ADAM17-mediated ectodomain shedding of heparin binding-EGF through separate pathways.Characterization of the catalytic activity of the membrane-anchored metalloproteinase ADAM15 in cell-based assays.Active-site determinants of substrate recognition by the metalloproteinases TACE and ADAM10 Ectodomain Shedding by ADAM17: Its Role in Neutrophil Recruitment and the Impairment of This Process during Sepsis SAR Studies of Exosite-Binding Substrate-Selective Inhibitors of A Disintegrin And Metalloprotease 17 (ADAM17) and Application as Selective in Vitro Probes.Phosphorylation of iRhom2 at the plasma membrane controls mammalian TACE-dependent inflammatory and growth factor signalling.ADAM12 and ADAM17 gene expression in laser-capture microdissected and non-microdissected breast tumors.Stimulation of platelet-derived growth factor receptor beta (PDGFRbeta) activates ADAM17 and promotes metalloproteinase-dependent cross-talk between the PDGFRbeta and epidermal growth factor receptor (EGFR) signaling pathways.TACE/ADAM17 is essential for oligodendrocyte development and CNS myelination.Interleukin-1 stimulates ADAM17 through a mechanism independent of its cytoplasmic domain or phosphorylation at threonine 735 iRhom2 regulation of TACE controls TNF-mediated protection against Listeria and responses to LPS CD23 Sheddase A disintegrin and metalloproteinase 10 (ADAM10) is also required for CD23 sorting into B cell-derived exosomes.Induction of RAGE shedding by activation of G protein-coupled receptors.Characterization of oxygen-induced retinopathy in mice carrying an inactivating point mutation in the catalytic site of ADAM15.Selective inhibition of ADAM12 catalytic activity through engineering of tissue inhibitor of metalloproteinase 2 (TIMP-2).Membrane-enabled dimerization of the intrinsically disordered cytoplasmic domain of ADAM10.Systemic overexpression of TNFα-converting enzyme does not lead to enhanced shedding activity in vivo Conditional inactivation of TNFα-converting enzyme in chondrocytes results in an elongated growth plate and shorter long bones.TACE activation by MAPK-mediated regulation of cell surface dimerization and TIMP3 association.TRPV1 activation is required for hypertonicity-stimulated inflammatory cytokine release in human corneal epithelial cells.Membrane-anchored growth factors, the epidermal growth factor family: beyond receptor ligands.The ADAM17-amphiregulin-EGFR axis in mammary development and cancer.Migration of growth factor-stimulated epithelial and endothelial cells depends on EGFR transactivation by ADAM17.Epidermal growth factor (EGF) ligand release by substrate-specific a disintegrin and metalloproteases (ADAMs) involves different protein kinase C (PKC) isoenzymes depending on the stimulus The Coxsackievirus and Adenovirus Receptor (CAR) undergoes ectodomain shedding and regulated intramembrane proteolysis (RIP).Alternative mRNA splicing generates two distinct ADAM12 prodomain variants.Differential surface expression of ADAM10 and ADAM17 on human T lymphocytes and tumor cells.ADAM-mediated amphiregulin shedding and EGFR transactivation.

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P2860

Substrate selectivity of epidermal growth factor-receptor ligand sheddases and their regulation by phorbol esters and calcium influx.

http://www.wikidata.org/entity/Q35545249

description

2006 nî lūn-bûn

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2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Substrate selectivity of epide ...... bol esters and calcium influx.

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Substrate selectivity of epide ...... bol esters and calcium influx.

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type

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Substrate selectivity of epide ...... bol esters and calcium influx.

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Substrate selectivity of epide ...... bol esters and calcium influx.

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Substrate selectivity of epide ...... bol esters and calcium influx.

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Substrate selectivity of epide ...... bol esters and calcium influx.

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MBC.E06-01-0014

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Molecular Biology of the Cell

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Substrate selectivity of epide ...... bol esters and calcium influx.

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Carl P Blobel

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Dieter Hartmann

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Gillian Murphy

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Karina Reiss

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Marc Schulte

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Paul Saftig

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Sylvain Le Gall

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Takafumi Yamaguchi

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Yoshiaki Toyama

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P2860

A role for a PDZ protein in the early secretory pathway for the targeting of proTGF-alpha to the cell surface

TACE cleavage of proamphiregulin regulates GPCR-induced proliferation and motility of cancer cells

The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts

Extracellular signal-regulated kinase phosphorylates tumor necrosis factor alpha-converting enzyme at threonine 735: a potential role in regulated shedding.

A metalloprotease-disintegrin, MDC9/meltrin-gamma/ADAM9 and PKCdelta are involved in TPA-induced ectodomain shedding of membrane-anchored heparin-binding EGF-like growth factor.

Mammary ductal morphogenesis requires paracrine activation of stromal EGFR via ADAM17-dependent shedding of epithelial amphiregulin

Untangling the ErbB signalling network

A secreted form of ADAM9 promotes carcinoma invasion through tumor-stromal interactions

ADAMs: key components in EGFR signalling and development

Rapid redistribution of Golgi proteins into the ER in cells treated with brefeldin A: evidence for membrane cycling from Golgi to ER

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10.1091/MBC.E06-01-0014

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1

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18

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Keisuke Horiuchi

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2006-11-01T00:00:00Z

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6716424

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17079736

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1751309

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