Short-Form Ron Promotes Spontaneous Breast Cancer Metastasis through Interaction with Phosphoinositide 3-Kinase. - Wikidata - awgldk - TriplyDB

Short-Form Ron Promotes Spontaneous Breast Cancer Metastasis through Interaction with Phosphoinositide 3-Kinase.

Short-Form Ron Promotes Spontaneous Breast Cancer Metastasis through Interaction with Phosphoinositide 3-Kinase.

http://www.wikidata.org/entity/Q35556501

about

Pathogenesis of RON receptor tyrosine kinase in cancer cells: activation mechanism, functional crosstalk, and signaling addiction A novel RON splice variant lacking exon 2 activates the PI3K/AKT pathway via PTEN phosphorylation in colorectal carcinoma cells.RON is not a prognostic marker for resectable pancreatic cancer.Short-form RON overexpression augments benzyl isothiocyanate-induced apoptosis in human breast cancer cells Preclinical Efficacy of Ron Kinase Inhibitors Alone and in Combination with PI3K Inhibitors for Treatment of sfRon-Expressing Breast Cancer Patient-Derived Xenografts.A novel protein isoform of the RON tyrosine kinase receptor transforms human pancreatic duct epithelial cells.Biological evaluation of antibody-maytansinoid conjugates as a strategy of RON targeted drug delivery for treatment of non-small cell lung cancer Dietary chemopreventative benzyl isothiocyanate inhibits breast cancer stem cells in vitro and in vivo.Short-form Ron is a novel determinant of ovarian cancer initiation and progression.RON kinase isoforms demonstrate variable cell motility in normal cells.Characterization of RON protein isoforms in pancreatic cancer: implications for biology and therapeutics.The RON receptor tyrosine kinase promotes metastasis by triggering MBD4-dependent DNA methylation reprogramming.Recepteur d'origine nantais (RON), more than a kinase: Role in castrate-resistant prostate cancer.HRAS, EGFR, MET, and RON Genes Are Recurrently Activated by Provirus Insertion in Liver Tumors Induced by the Retrovirus Myeloblastosis-Associated Virus 2.The Tyrosine Kinome Dictates Breast Cancer Heterogeneity and Therapeutic Responsiveness.Role of Recepteur D'origine Nantais on Gastric Cancer Development and Progression Targeting PDK1 for Chemosensitization of Cancer Cells.RON Signaling Is a Key Mediator of Tumor Progression in Many Human Cancers.Inhibition of RON kinase potentiates anti-CTLA-4 immunotherapy to shrink breast tumors and prevent metastatic outgrowth mTORC1 is a key mediator of RON-dependent breast cancer metastasis with therapeutic potential

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Short-Form Ron Promotes Spontaneous Breast Cancer Metastasis through Interaction with Phosphoinositide 3-Kinase.

http://www.wikidata.org/entity/Q35556501

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

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Short-Form Ron Promotes Sponta ...... ith Phosphoinositide 3-Kinase.

@en

P2093

Alana L Welm

http://www.w3.org/2001/XMLSchema#string

Henok Eyob

http://www.w3.org/2001/XMLSchema#string

Leigh Neumayer

http://www.w3.org/2001/XMLSchema#string

Ling Zhao

http://www.w3.org/2001/XMLSchema#string

Magdalena Bieniasz

http://www.w3.org/2001/XMLSchema#string

Saundra S Buys

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Xuemei Liu

http://www.w3.org/2001/XMLSchema#string

Yi-Chun Lin

http://www.w3.org/2001/XMLSchema#string

Yoko S Derose

http://www.w3.org/2001/XMLSchema#string

P2860

The PI3K-PDK1 connection: more than just a road to PKB

STK/RON receptor tyrosine kinase mediates both apoptotic and growth signals via the multifunctional docking site conserved among the HGF receptor family

The macrophage stimulating protein/Ron pathway as a potential therapeutic target to impede multiple mechanisms involved in breast cancer progression.

c-Cbl is a critical modulator of the Ron tyrosine kinase receptor

A multifunctional docking site mediates signaling and transformation by the hepatocyte growth factor/scatter factor receptor family

Fv2 encodes a truncated form of the Stk receptor tyrosine kinase

Cancer statistics, 2004

Receptor tyrosine kinase coactivation networks in cancer

Met-related receptor tyrosine kinase Ron in tumor growth and metastasis.

A dominant mutant allele of the ING4 tumor suppressor found in human cancer cells exacerbates MYC-initiated mouse mammary tumorigenesis.

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7

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