Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma. - Wikidata - awgldk - TriplyDB

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

http://www.wikidata.org/entity/Q35588829

about

Current and future trials of targeted therapies in cutaneous melanoma Anticancer activity of cationic porphyrins in melanoma tumour-bearing mice and mechanistic in vitro studies.Small-molecule antagonists for CXCR2 and CXCR1 inhibit human melanoma growth by decreasing tumor cell proliferation, survival, and angiogenesis Upregulated ankyrin repeat-rich membrane spanning protein contributes to tumour progression in cutaneous melanoma Expression of phosphorylated extracellular signal-regulated kinase at the invasive front of hepatic colorectal metastasis Biomarkers: the useful and the not so useful--an assessment of molecular prognostic markers for cutaneous melanoma High expression of cytoplasmic phosphorylated CSE1L in malignant melanoma but not in benign nevi: phosphorylated CSE1L for the discrimination between melanoma and benign nevi.BRAF, KIT and NRAS mutations and expression of c-KIT, phosphorylated extracellular signal-regulated kinase and phosphorylated AKT in Japanese melanoma patients.Plexin B1 is repressed by oncogenic B-Raf signaling and functions as a tumor suppressor in melanoma cells Proteasome inhibition blocks NF-κB and ERK1/2 pathways, restores antigen expression, and sensitizes resistant human melanoma to TCR-engineered CTLs DW-F5: A novel formulation against malignant melanoma from Wrightia tinctoria Coexpression of SOX10/CD271 (p75(NTR)) and β-Galactosidase in Large to Giant Congenital Melanocytic Nevi of Pediatric Patients.Growth factors and oncogenes as targets in melanoma: lost in translation?Modeling Melanoma In Vitro and In Vivo.Increased cyclin D1 expression can mediate BRAF inhibitor resistance in BRAF V600E-mutated melanomas Choice of fixative is crucial to successful immunohistochemical detection of phosphoproteins in paraffin-embedded tumor tissues.CXCR1 and CXCR2 enhances human melanoma tumourigenesis, growth and invasion Tissue biomarkers for prognosis in cutaneous melanoma: a systematic review and meta-analysis.Small molecules and targeted therapies in distant metastatic disease.Melanoma biomarkers: current status and utility in diagnosis, prognosis, and response to therapy.Targeting the MAPK pathway in melanoma: why some approaches succeed and other fail.PLX4032: does it keep its promise for metastatic melanoma treatment?The role of mitogen- and stress-activated protein kinase pathways in melanoma.In vitro three-dimensional tumor microenvironment models for anticancer drug discovery.Melanocytic nevi and melanoma: unraveling a complex relationship.Metastatic melanoma moves on: translational science in the era of personalized medicine.Real-time cell cycle imaging during melanoma growth, invasion, and drug response.Specifically targeting ERK1 or ERK2 kills melanoma cells Increased levels of DUSP6 phosphatase stimulate tumourigenesis in a molecularly distinct melanoma subtype.Etlingera elatior Extract promotes cell death in B16 melanoma cells via down-regulation of ERK and Akt signaling pathways.CD24 induces the invasion of cholangiocarcinoma cells by upregulating CXCR4 and increasing the phosphorylation of ERK1/2 Extracellular signal-regulated protein kinase is activated in cervical intraepithelial neoplasms but inactivated in invasive cervical carcinoma.Effects of microRNA-708 on Epithelial-Mesenchymal Transition, Cell Proliferation and Apoptosis in Melanoma Cells by Targeting LEF1 through the Wnt Signaling Pathway.Extracellular signal-regulated kinase (ERK) expression and activation in mobile tongue squamous cell carcinoma: associations with clinicopathological parameters and patients survival.Modulation of human melanoma cell proliferation and apoptosis by hydatid cyst fluid of Echinococcus granulosus.Gefitinib or lapatinib with foretinib synergistically induce a cytotoxic effect in melanoma cell lines.Immunohistochemical analysis of molecular drivers in melanoma identifies p16 as an independent prognostic biomarker.Perspectives for cancer immunotherapy mediated by p19Arf plus interferon-beta gene transfer

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P2860

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

http://www.wikidata.org/entity/Q35588829

description

2005 nî lūn-bûn

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name

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

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Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

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type

label

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

@ast

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

@en

prefLabel

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

@ast

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma.

@en

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JCP.2005.025957

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Journal of Clinical Pathology

P1476

Activation of the extracellular signal regulated kinase (ERK) pathway in human melanoma

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P2093

A A Palmer

http://www.w3.org/2001/XMLSchema#string

C S Lee

http://www.w3.org/2001/XMLSchema#string

J F Thompson

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L P Bron

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L Zhuang

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P Hersey

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S W McCarthy

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P2860

Constitutive activation of the Ras-Raf signaling pathway in metastatic melanoma is associated with poor prognosis

Mutations of the BRAF gene in human cancer

The Bcl2 family: regulators of the cellular life-or-death switch

BAY 43-9006: early clinical data in patients with advanced solid malignancies.

Prognosis for patients with thin cutaneous melanoma: long-term survival data from New South Wales Central Cancer Registry and the Sydney Melanoma Unit.

MAP kinase pathways.

Signal transduction pathway targets for anticancer drug discovery.

Prognostic factors analysis of 17,600 melanoma patients: validation of the American Joint Committee on Cancer melanoma staging system.

High frequency of BRAF mutations in nevi.

Blocking oncogenic Ras signaling for cancer therapy.

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