Lack of kinase-independent activity of PI3Kγ in locus coeruleus induces ADHD symptoms through increased CREB signaling.
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Phosphoinositide 3-kinase: friend and foe in cardiovascular diseaseThe Multifaceted Roles of PI3Kγ in Hypertension, Vascular Biology, and InflammationNoradrenergic Modulation of Cognition in Health and Disease.Scaffolding Function of PI3Kgamma Emerges from Enzyme's Shadow.Exploring the Validity of Proposed Transgenic Animal Models of Attention-Deficit Hyperactivity Disorder (ADHD).PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research.Melatonin inhibits attention-deficit/hyperactivity disorder caused by atopic dermatitis-induced psychological stress in an NC/Nga atopic-like mouse modelErbB4 deletion in noradrenergic neurons in the locus coeruleus induces mania-like behavior via elevated catecholamines
P2860
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P2860
Lack of kinase-independent activity of PI3Kγ in locus coeruleus induces ADHD symptoms through increased CREB signaling.
description
2015 nî lūn-bûn
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name
Lack of kinase-independent act ...... ough increased CREB signaling.
@ast
Lack of kinase-independent act ...... ough increased CREB signaling.
@en
type
label
Lack of kinase-independent act ...... ough increased CREB signaling.
@ast
Lack of kinase-independent act ...... ough increased CREB signaling.
@en
prefLabel
Lack of kinase-independent act ...... ough increased CREB signaling.
@ast
Lack of kinase-independent act ...... ough increased CREB signaling.
@en
P2093
P2860
P50
P356
P1476
Lack of kinase-independent act ...... rough increased CREB signaling
@en
P2093
Fabio Pallante
Ivana D'Andrea
Roberta Iacobucci
Stefania Fardella
Valentina Fardella
P2860
P304
P356
10.15252/EMMM.201404697
P577
2015-07-01T00:00:00Z