Transcriptional regulation of Salmonella virulence: a PhoQ periplasmic domain mutation results in increased net phosphotransfer to PhoP.
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The pleiotropic two-component regulatory system PhoP-PhoQA PhoP-regulated outer membrane protease of Salmonella enterica serovar typhimurium promotes resistance to alpha-helical antimicrobial peptides.CD4+ T cells and toll-like receptors recognize Salmonella antigens expressed in bacterial surface organelles.Rapid and highly efficient method for scarless mutagenesis within the Salmonella enterica chromosome.Regulation of virulence gene transcripts by the Francisella novicida orphan response regulator PmrA: role of phosphorylation and evidence of MglA/SspA interaction.Mechanisms of intrinsic resistance to antimicrobial peptides of Edwardsiella ictaluri and its influence on fish gut inflammation and virulence.The nature of the attenuation of Salmonella typhimurium strains expressing human papillomavirus type 16 virus-like particles determines the systemic and mucosal antibody responses in nasally immunized mice.Coordinate intracellular expression of Salmonella genes induced during infection.A HilA-independent pathway to Salmonella typhimurium invasion gene transcription.InvF is required for expression of genes encoding proteins secreted by the SPI1 type III secretion apparatus in Salmonella typhimurium.Hha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activatorConstitutive mutations of the Salmonella enterica serovar Typhimurium transcriptional virulence regulator phoPGenetic and functional analysis of a PmrA-PmrB-regulated locus necessary for lipopolysaccharide modification, antimicrobial peptide resistance, and oral virulence of Salmonella enterica serovar typhimurium.Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.Virulence gene regulation in Salmonella enterica.Mechanisms and fitness costs of resistance to antimicrobial peptides LL-37, CNY100HL and wheat germ histonesProbing conservation of HAMP linker structure and signal transduction mechanism through analysis of hybrid sensor kinases.Colistin resistance mechanisms in Klebsiella pneumoniae strains from Taiwan.Immunogenicity against human papillomavirus type 16 virus-like particles is strongly enhanced by the PhoPc phenotype in Salmonella enterica serovar TyphimuriumPhoQ mutations promote lipid A modification and polymyxin resistance of Pseudomonas aeruginosa found in colistin-treated cystic fibrosis patientsEntry and survival of Salmonella typhimurium in dendritic cells and presentation of recombinant antigens do not require macrophage-specific virulence factorsDsbA and MgrB regulate steA expression through the two-component system PhoQ/PhoP in Salmonella enterica.Type III secretion system 1 genes in Vibrio parahaemolyticus are positively regulated by ExsA and negatively regulated by ExsD.Modulation of the regulatory activity of bacterial two-component systems by SlyA.Molecular characterization of the PhoP-PhoQ two-component system in Escherichia coli K-12: identification of extracellular Mg2+-responsive promoters.Characterization of the catalytic activities of the PhoQ histidine protein kinase of Salmonella enterica serovar Typhimurium.PhoP-PhoQ-regulated loci are required for enhanced bile resistance in Salmonella sppRoles of hilC and hilD in regulation of hilA expression in Salmonella enterica serovar TyphimuriumMultiple factors independently regulate hilA and invasion gene expression in Salmonella enterica serovar typhimurium.The ATP-dependent lon protease of Salmonella enterica serovar Typhimurium regulates invasion and expression of genes carried on Salmonella pathogenicity island 1Mutational analysis of the Escherichia coli PhoQ sensor kinase: differences with the Salmonella enterica serovar Typhimurium PhoQ protein and in the mechanism of Mg2+ and Ca2+ sensing.Integrating global regulatory input into the Salmonella pathogenicity island 1 type III secretion system.Mutational analysis of the residue at position 48 in the Salmonella enterica Serovar Typhimurium PhoQ sensor kinase.PhoP-PhoQ activates transcription of pmrAB, encoding a two-component regulatory system involved in Salmonella typhimurium antimicrobial peptide resistance.Identification and Characterization of Two Klebsiella pneumoniae lpxL Lipid A Late Acyltransferases and Their Role in Virulence.Genomic Characterization of Colistin Heteroresistance in Klebsiella pneumoniae during a Nosocomial Outbreak.HAMP Domain Rotation and Tilting Movements Associated with Signal Transduction in the PhoQ Sensor KinaseAntimicrobial peptides activate the Rcs regulon through the outer membrane lipoprotein RcsF.Transcriptional regulation of Salmonella enterica serovar Typhimurium genes by bile.PmrA-PmrB-regulated genes necessary for 4-aminoarabinose lipid A modification and polymyxin resistance.
P2860
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P2860
Transcriptional regulation of Salmonella virulence: a PhoQ periplasmic domain mutation results in increased net phosphotransfer to PhoP.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@ast
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@en
type
label
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@ast
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@en
prefLabel
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@ast
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@en
P2093
P2860
P1476
Transcriptional regulation of ...... d net phosphotransfer to PhoP.
@en
P2093
P2860
P304
P356
10.1128/JB.178.21.6369-6373.1996
P407
P577
1996-11-01T00:00:00Z