The role of the c-Jun N-terminal kinases 1/2 and receptor-interacting protein kinase 3 in furosemide-induced liver injury
about
Role of endoplasmic reticulum stress in drug-induced toxicityPathophysiological significance of c-jun N-terminal kinase in acetaminophen hepatotoxicityMechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.Increased hepatic receptor interacting protein kinase 3 expression due to impaired proteasomal functions contributes to alcohol-induced steatosis and liver injury.New insights into the role and mechanism of c-Jun-N-terminal kinase signaling in the pathobiology of liver diseases.
P2860
The role of the c-Jun N-terminal kinases 1/2 and receptor-interacting protein kinase 3 in furosemide-induced liver injury
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2014 nî lūn-bûn
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2014年の論文
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2014年論文
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name
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@ast
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@en
type
label
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@ast
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@en
prefLabel
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@ast
The role of the c-Jun N-termin ...... urosemide-induced liver injury
@en
P2093
P2860
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The role of the c-Jun N-termin ...... urosemide-induced liver injury
@en
P2093
Hartmut Jaeschke
Mary Lynn Bajt
Wen-Xing Ding
Yuchao Xie
P2860
P304
P356
10.3109/00498254.2014.986250
P407
P577
2014-11-25T00:00:00Z