GPR48 increases mineralocorticoid receptor gene expression. - Wikidata - awgldk - TriplyDB

GPR48 increases mineralocorticoid receptor gene expression.

GPR48 increases mineralocorticoid receptor gene expression.

http://www.wikidata.org/entity/Q35715750

about

LGR4 and Its Role in Intestinal Protection and Energy Metabolism Lgr4/Gpr48 negatively regulates TLR2/4-associated pattern recognition and innate immunity by targeting CD14 expression Identification and characterization of novel renal sensory receptors.Soluble adenylyl cyclase in health and disease.Analysis of LGR4 receptor distribution in human and mouse tissues The multifaceted mineralocorticoid receptor Lgr4 in ocular development and glaucoma Renal and cardiovascular sensory receptors and blood pressure regulation.LGR4 and its ligands, R-spondin 1 and R-spondin 3, regulate food intake in the hypothalamus of male rats Lgr4 is a key regulator of prostate development and prostate stem cell differentiation.Lgr4 regulates mammary gland development and stem cell activity through the pluripotency transcription factor Sox2.Expression of mineralocorticoid and glucocorticoid receptors in preautonomic neurons of the rat paraventricular nucleus.Extra sensory perception: the role of Gpr receptors in the kidney.BMP-2 Enhances Lgr4 Gene Expression in Osteoblastic Cells.A novel SCFA receptor, the microbiota, and blood pressure regulation.Nonsense mutation in the LGR4 gene is associated with several human diseases and other traits.Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.LGR4 modulates breast cancer initiation, metastasis, and cancer stem cells.

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P2860

GPR48 increases mineralocorticoid receptor gene expression.

http://www.wikidata.org/entity/Q35715750

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2011 nî lūn-bûn

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2011年論文

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2011年论文

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2011年论文

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GPR48 increases mineralocorticoid receptor gene expression.

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GPR48 increases mineralocorticoid receptor gene expression.

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GPR48 increases mineralocorticoid receptor gene expression.

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GPR48 increases mineralocorticoid receptor gene expression.

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GPR48 increases mineralocorticoid receptor gene expression.

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Journal of the American Society of Nephrology

P1476

GPR48 increases mineralocorticoid receptor gene expression.

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P2093

Feng Wang

http://www.w3.org/2001/XMLSchema#string

Guang Ning

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Haiyan Sun

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Huijie Zhang

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Jiqiu Wang

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Jun Yang

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Lei Ye

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Mingyao Liu

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Nengguang Fan

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Ruixin Liu

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P2860

R-spondins function as ligands of the orphan receptors LGR4 and LGR5 to regulate Wnt/beta-catenin signaling

Lgr5 homologues associate with Wnt receptors and mediate R-spondin signalling

Mutations in the mineralocorticoid receptor gene cause autosomal dominant pseudohypoaldosteronism type I

A salt wasting syndrome in infancy

Requirement of human renal water channel aquaporin-2 for vasopressin-dependent concentration of urine

Molecular identification of the gene responsible for congenital nephrogenic diabetes insipidus

Epithelial sodium channel and the control of sodium balance: interaction between genetic and environmental factors

Type I pseudohypoaldosteronism includes two clinically and genetically distinct entities with either renal or multiple target organ defects

Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1

Up-regulation of GPR48 induced by down-regulation of p27Kip1 enhances carcinoma cell invasiveness and metastasis

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281-293

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10.1681/ASN.2011040351

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2

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23

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22135314

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