Role of matrix metalloproteinases in the acquisition and reconsolidation of cocaine-induced conditioned place preference. - Wikidata - awgldk - TriplyDB

Role of matrix metalloproteinases in the acquisition and reconsolidation of cocaine-induced conditioned place preference.

Role of matrix metalloproteinases in the acquisition and reconsolidation of cocaine-induced conditioned place preference.

http://www.wikidata.org/entity/Q35721080

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Caught in the Net: Perineuronal Nets and Addiction Activity dependent CAM cleavage and neurotransmission Synaptic circuit remodelling by matrix metalloproteinases in health and disease Anisomycin in the medial prefrontal cortex reduces reconsolidation of cocaine-associated memories in the rat self-administration model MMPs and soluble ICAM-5 increase neuronal excitability within in vitro networks of hippocampal neurons Soluble ICAM-5, a product of activity dependent proteolysis, increases mEPSC frequency and dendritic expression of GluA1 Contributions of matrix metalloproteinases to neural plasticity, habituation, associative learning and drug addiction.Extracellular matrix plasticity and GABAergic inhibition of prefrontal cortex pyramidal cells facilitates relapse to heroin seeking.Genetic variations within metalloproteinases impact on the prophylaxis of depressive phases in bipolar patients.The Hepatocyte Growth Factor/c-Met Antagonist, Divalinal-Angiotensin IV, Blocks the Acquisition of Methamphetamine Dependent Conditioned Place Preference in Rats.A role for matrix metalloproteinases in nicotine-induced conditioned place preference and relapse in adolescent female rats Learning to forget: manipulating extinction and reconsolidation processes to treat addiction Synaptic plasticity mediating cocaine relapse requires matrix metalloproteinases.Matrix metalloproteinases, synaptic injury, and multiple sclerosis Metzincin proteases and their inhibitors: foes or friends in nervous system physiology?Aberrant learning and memory in addiction.Plasticity associated with escalated operant ethanol self-administration during acute withdrawal in ethanol-dependent rats requires intact matrix metalloproteinase systems Removal of perineuronal nets in the medial prefrontal cortex impairs the acquisition and reconsolidation of a cocaine-induced conditioned place preference memory New medications for drug addiction hiding in glutamatergic neuroplasticity.Integrins modulate relapse to cocaine-seeking.The extracellular protease matrix metalloproteinase-9 is activated by inhibitory avoidance learning and required for long-term memory.The tetrapartite synapse: Extracellular matrix remodeling contributes to corticoaccumbens plasticity underlying drug addiction The MMP-1/PAR-1 Axis Enhances Proliferation and Neuronal Differentiation of Adult Hippocampal Neural Progenitor Cells Matrix metalloproteinase-dependent shedding of intercellular adhesion molecule-5 occurs with long-term potentiation.Conceptualizing withdrawal-induced escalation of alcohol self-administration as a learned, plasticity-dependent process.Early postnatal expression and localization of matrix metalloproteinases-2 and -9 during establishment of rat hippocampal synaptic circuitry.The NMDA antagonist MK-801 disrupts reconsolidation of a cocaine-associated memory for conditioned place preference but not for self-administration in rats.Targeting extinction and reconsolidation mechanisms to combat the impact of drug cues on addiction.Inhibition of matrix metalloproteinase activity disrupts reconsolidation but not consolidation of a fear memory Basolateral amygdala involvement in memory reconsolidation processes that facilitate drug context-induced cocaine seeking.Differential effects of propranolol on conditioned hyperactivity and locomotor sensitization induced by morphine in rats.Common influences of non-competitive NMDA receptor antagonists on the consolidation and reconsolidation of cocaine-cue memory.Reconsolidation of drug memories.Matrix metalloproteinase dependent cleavage of cell adhesion molecules in the pathogenesis of CNS dysfunction with HIV and methamphetamine.Implication of matrix metalloproteinases in regulating neuronal disorder.MMP-9 in translation: from molecule to brain physiology, pathology, and therapy.Role of perineuronal nets in the anterior dorsal lateral hypothalamic area in the acquisition of cocaine-induced conditioned place preference and self-administration.Dopamine increases NMDA-stimulated calcium flux in striatopallidal neurons through a matrix metalloproteinase-dependent mechanism.Integrins in synapse regulation.Methamphetamine-associated cleavage of the synaptic adhesion molecule intercellular adhesion molecule-5.

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P2860

Role of matrix metalloproteinases in the acquisition and reconsolidation of cocaine-induced conditioned place preference.

http://www.wikidata.org/entity/Q35721080

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Role of matrix metalloproteina ...... conditioned place preference.

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Role of matrix metalloproteina ...... conditioned place preference.

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Role of matrix metalloproteina ...... conditioned place preference.

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Learning and Memory

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Role of matrix metalloproteina ...... d conditioned place preference

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Davelle L Cocking

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Heiko T Jansen

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Joseph W Harding

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Melissa R Forquer

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Travis E Brown

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P2860

Matrix metalloproteinases: old dogs with new tricks

Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval

Matrix metalloproteinases: structures, evolution, and diversification

Acute and repeated administration of cocaine differentially regulates expression of PSA-NCAM-positive neurons in the rat hippocampus

The reinstatement model of drug relapse: history, methodology and major findings

Directly reactivated, but not indirectly reactivated, memories undergo reconsolidation in the amygdala.

Memory consolidation for the discrimination of frequency-modulated tones in mongolian gerbils is sensitive to protein-synthesis inhibitors applied to the auditory cortex.

Matrix metalloproteinase-9 is required for hippocampal late-phase long-term potentiation and memory.

Cocaine self-administration alters the morphology of dendrites and dendritic spines in the nucleus accumbens and neocortex.

Cocaine-induced expression of the tetraspanin CD81 and its relation to hypothalamic function.

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10.1101/LM.476207

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3

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14

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17353546

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1838561

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