Direct observation of substance P-induced internalization of neurokinin 1 (NK1) receptors at sites of inflammation.
about
Neurokinin-1 receptor desensitization to consecutive microdialysis infusions of substance P in human skinDelineation of the endocytic pathway of substance P and its seven-transmembrane domain NK1 receptorThe role of Transient Receptor Potential Vanilloid 1 (TRPV1) channels in pancreatitisAgonists of proteinase-activated receptor 1 induce plasma extravasation by a neurogenic mechanismEndothelin-converting enzyme 1 promotes re-sensitization of neurokinin 1 receptor-dependent neurogenic inflammationAn antiangiogenic neurokinin-B/thromboxane A2 regulatory axisThe neuropeptide Y Y1 receptor is a somatic receptor on dorsal root ganglion neurons and a postsynaptic receptor on somatostatin dorsal horn neurons.Airway vasculature after mycoplasma infection: chronic leakiness and selective hypersensitivity to substance P.Delivery of bone morphogenetic protein-2 and substance P using graphene oxide for bone regenerationRespiratory syncytial virus infection and G and/or SH protein expression contribute to substance P, which mediates inflammation and enhanced pulmonary disease in BALB/c mice.Mechanisms of desensitization and resensitization of proteinase-activated receptor-2.The substance P/NK-1 receptor system: NK-1 receptor antagonists as anti-cancer drugs.Rapid endocytosis of a G protein-coupled receptor: substance P evoked internalization of its receptor in the rat striatum in vivo.PET-scan shows peripherally increased neurokinin 1 receptor availability in chronic tennis elbow: visualizing neurogenic inflammation?Substance P mediates inflammatory oedema in acute pancreatitis via activation of the neurokinin-1 receptor in rats and miceSubstance P increases neutrophil adhesion to human umbilical vein endothelial cellsBlockade of neurokinin-1 receptors in the ventral respiratory column does not affect breathing but alters neurochemical release.Effect of mitogen-activated protein kinases on chemokine synthesis induced by substance P in mouse pancreatic acinar cells.Substance P in the corneal stroma regulates the severity of herpetic stromal keratitis lesions.Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance PInflammation-induced abnormalities in the subcellular localization and trafficking of the neurokinin 1 receptor in the enteric nervous system.Botulinum toxin in migraine: Role of transport in trigemino-somatic and trigemino-vascular afferents.Angiogenesis in mice with chronic airway inflammation: strain-dependent differences.Endothelin-converting enzyme 1 degrades neuropeptides in endosomes to control receptor recycling.Substance P affects growth factors in Pseudomonas aeruginosa-infected mouse cornea.Role of substance P and the neurokinin 1 receptor in acute pancreatitis and pancreatitis-associated lung injury.Pathophysiology of pulmonary complications of acute pancreatitis.Role of calcium in substance P-induced chemokine synthesis in mouse pancreatic acinar cells.TNF signaling contributes to the development of nociceptive sensitization in a tibia fracture model of complex regional pain syndrome type IDesensitization of the neurokinin-1 receptor (NK1-R) in neurons: effects of substance P on the distribution of NK1-R, Galphaq/11, G-protein receptor kinase-2/3, and beta-arrestin-1/2Pentoxifylline attenuates nociceptive sensitization and cytokine expression in a tibia fracture rat model of complex regional pain syndrome.Decreased Brain Neurokinin-1 Receptor Availability in Chronic Tennis Elbow.Agonist-selective endocytosis of mu opioid receptor by neurons in vivo.Increased liposome extravasation in selected tissues: effect of substance P.Genetic construction and properties of a diphtheria toxin-related substance P fusion protein: in vitro destruction of cells bearing substance P receptors.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Involvement of substance P and the NK-1 receptor in pancreatic cancer.Arresting inflammation: contributions of plasma membrane and endosomal signalling to neuropeptide-driven inflammatory disease.Evidence for a role of mitogen-activated protein kinases in the treatment of experimental acute pancreatitis.Direct observation of endocytosis of gastrin releasing peptide and its receptor.
P2860
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P2860
Direct observation of substance P-induced internalization of neurokinin 1 (NK1) receptors at sites of inflammation.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Direct observation of substanc ...... tors at sites of inflammation.
@ast
Direct observation of substanc ...... tors at sites of inflammation.
@en
type
label
Direct observation of substanc ...... tors at sites of inflammation.
@ast
Direct observation of substanc ...... tors at sites of inflammation.
@en
prefLabel
Direct observation of substanc ...... tors at sites of inflammation.
@ast
Direct observation of substanc ...... tors at sites of inflammation.
@en
P2093
P2860
P356
P1476
Direct observation of substanc ...... tors at sites of inflammation.
@en
P2093
A M Garland
D M McDonald
J J Bowden
N W Bunnett
P2860
P304
P356
10.1073/PNAS.91.19.8964
P407
P577
1994-09-01T00:00:00Z