Deficiency of endothelial nitric-oxide synthase confers susceptibility to diabetic nephropathy in nephropathy-resistant inbred mice. - Wikidata - awgldk - TriplyDB

Deficiency of endothelial nitric-oxide synthase confers susceptibility to diabetic nephropathy in nephropathy-resistant inbred mice.

Deficiency of endothelial nitric-oxide synthase confers susceptibility to diabetic nephropathy in nephropathy-resistant inbred mice.

http://www.wikidata.org/entity/Q35757153

about

Modulating DDAH/NOS Pathway to Discover Vasoprotective Insulin Sensitizers Nitric oxide synthase derangements and hypertension in kidney disease Role of T cells in type 2 diabetic nephropathy Renal endothelial dysfunction in diabetic nephropathy Effects of stimulation of soluble guanylate cyclase on diabetic nephropathy in diabetic eNOS knockout mice on top of angiotensin II receptor blockade Alterations of urinary metabolite profile in model diabetic nephropathy.Diabetic nephropathy induces alterations in the glomerular and tubule lipid profiles.eNOS knockout mice with advanced diabetic nephropathy have less benefit from renin-angiotensin blockade than from aldosterone receptor antagonists.Epidermal growth factor receptor inhibition slows progression of diabetic nephropathy in association with a decrease in endoplasmic reticulum stress and an increase in autophagy Phosphodiesterase 5 inhibition ameliorates angiontensin II-induced podocyte dysmotility via the protein kinase G-mediated downregulation of TRPC6 activity.Mouse models of diabetic nephropathy.Podocyte-specific VEGF-a gain of function induces nodular glomerulosclerosis in eNOS null mice.Diabetic nephropathy - complications and treatment.Role of endothelial nitric oxide synthase in diabetic nephropathy: lessons from diabetic eNOS knockout mice Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.New targets for treatment of diabetic nephropathy: what we have learned from animal models.High glucose causes dysfunction of the human glomerular endothelial glycocalyx.Reduction in basal nitric oxide activity causes albuminuria A modest decrease in endothelial NOS in mice comparable to that associated with human NOS3 variants exacerbates diabetic nephropathy Glomerular endothelial cell injury and damage precedes that of podocytes in adriamycin-induced nephropathy.A novel mouse model of advanced diabetic kidney disease.Long-term administration of the histone deacetylase inhibitor vorinostat attenuates renal injury in experimental diabetes through an endothelial nitric oxide synthase-dependent mechanism Endothelial dysfunction exacerbates renal interstitial fibrosis through enhancing fibroblast Smad3 linker phosphorylation in the mouse obstructed kidney.Diffuse glomerular nodular lesions in diabetic pigs carrying a dominant-negative mutant hepatocyte nuclear factor 1-alpha, an inheritant diabetic gene in humans.SOD1, but not SOD3, deficiency accelerates diabetic renal injury in C57BL/6-Ins2(Akita) diabetic mice Acute laminar shear stress reversibly increases human glomerular endothelial cell permeability via activation of endothelial nitric oxide synthase Increased nitric oxide availability attenuates high fat diet metabolic alterations and gene expression associated with insulin resistance.The effect of salt on renal damage in eNOS-deficient mice Tadalafil Integrates Nitric Oxide-Hydrogen Sulfide Signaling to Inhibit High Glucose-induced Matrix Protein Synthesis in Podocytes Hypohalous acids contribute to renal extracellular matrix damage in experimental diabetes The Short-Term Remission of Diabetic Nephropathy After Roux-en-Y Gastric Bypass in Chinese Patients of T2DM with Obesity Blockade of endothelial-mesenchymal transition by a Smad3 inhibitor delays the early development of streptozotocin-induced diabetic nephropathy Role of blood pressure and the renin-angiotensin system in development of diabetic nephropathy (DN) in eNOS-/- db/db mice.Hyperglycemia and renin-dependent hypertension synergize to model diabetic nephropathy.Integrin α1/Akita double-knockout mice on a Balb/c background develop advanced features of human diabetic nephropathy.Quantitative non-invasive cell characterisation and discrimination based on multispectral autofluorescence features Nicorandil as a novel therapy for advanced diabetic nephropathy in the eNOS-deficient mouse.Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathy Endothelial Nitric Oxide Synthase Prevents Heparanase Induction and the Development of Proteinuria.Generation of a conditional allele for the mouse endothelial nitric oxide synthase gene

P2860

Q26771754-6400E7FA-A10F-49C0-9EE8-7308C3A7BBE4 Q26849234-5544F999-91BA-424A-81BE-F0B0B5E5D989 Q26849691-2502CCF2-A3F0-49EA-9839-404CAA316468 Q27021318-783CCB69-91F0-449D-88FB-0DA786E99A19 Q28482185-6DBDAFAE-0100-407D-B20D-3640B041052A Q30369623-DFABBBC9-FC69-4CAF-B041-F61800D33BB3 Q30407920-660987A5-D927-44B5-8581-4C1CA800997F Q33597090-F2AE94ED-CEF4-4E02-AD19-16C1DC401425 Q33646704-CFEA5750-5A51-4DFF-A238-C42C77D5C6F0 Q33764566-E230626C-2E86-4E15-A11C-B0B898EB1779 Q33821186-9B3A5830-CD35-4046-9FA8-91D0E2925BCB Q33974608-F0FA22B9-C668-4D82-BC65-FD331A5BF762 Q34387952-3FEC7186-8CE4-4E52-9620-5F2FB949461E Q34412951-FBE792AE-1EB0-4A21-BBA9-ED6B4787880B Q34427632-CF6C961D-0116-45B5-9281-2E2FC958FAF0 Q34498964-B34A7E72-E09C-42CA-AB98-3759B3493EDE Q34501882-0EF077B7-0CE8-4BC7-B77E-D7BF784EA0CE Q34527146-CBD9DC8E-19A9-41B8-89B6-1E4C273A506E Q34549867-69C3A53B-DE84-4B84-826C-C93D49EBAEAE Q34566167-A78045D0-3F6E-447D-AAD5-829309319A2D Q34712527-73564A4A-22FF-4178-A51E-F89A47F86BE8 Q34839640-AB2D1C05-2E18-43D5-8BEE-F46D8401E11B Q35082779-9B301431-8642-4C5F-B35D-CC8817D3BA4D Q35125191-A811E34A-765C-42EB-BE0C-BEEC5E4AB839 Q35180432-B820CB50-F2F7-4E5D-BAD0-20CE84ED6AEA Q35326124-089E7063-4CB0-4FF0-9FC9-72F12F958848 Q35540671-6A12A44D-014A-482C-A8A0-2982F99B67CD Q35569538-AC838169-C7CB-4971-B20F-FAD7BE47AC32 Q35583071-1694A5B0-8B1C-468B-A087-4D3134DD6C62 Q35630838-B6059269-634B-4611-A74A-19F07A60AF83 Q35698046-7516228E-CC7F-42F9-B768-7977343D3092 Q35757154-85C288E2-DE50-4AE7-ABD1-ABF6D99896D1 Q35787422-95F537E0-3BC6-4A13-9CC5-B8AF7E0E6506 Q35804146-4E63519F-95F8-4513-A721-A0ED2A53D134 Q35937053-E548CC2A-14C7-4683-98F7-397019123720 Q35974515-26A7E4C8-42A9-4BEF-9E66-AA992FA031F5 Q35994519-1FEFC97A-9250-490E-8CE0-CBFE74E4F3DB Q36017619-5A0900E2-F8B4-4E97-B883-9D575C22411A Q36098881-486A9E01-7F5D-40DD-BB09-0F53402F8A5F Q36103630-ECC37C98-4FBB-4D76-8E75-A575C8CA3F37

P2860

Deficiency of endothelial nitric-oxide synthase confers susceptibility to diabetic nephropathy in nephropathy-resistant inbred mice.

http://www.wikidata.org/entity/Q35757153

description

2007 nî lūn-bûn

@nan

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

2007年论文

@zh

2007年论文

@zh-cn

name

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@ast

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@en

type

label

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@ast

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@en

prefLabel

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@ast

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@en

P1433

Q35757153-CF085187-768D-4EFC-969F-8A5BE5E4830A

P1476

Q35757153-60256757-CBF1-4E23-A332-BD06C9C7A098

P2093

Q35757153-0584BA05-12D8-42B1-9AF4-F84DD1F094DC

Q35757153-1B64B1DE-7433-48DD-BB1B-6C75EAD442FD

Q35757153-3482F9BD-8262-4ADA-AB07-DF984C92E761

Q35757153-7BF6C0A7-0B0E-4E1E-813C-981FA2DAE238

Q35757153-D2EAAC06-1DAC-44AF-A763-003AF6D22541

Q35757153-DCD4B267-1254-4FB8-ABFD-FEF5E8427D86

Q35757153-F94E183A-3A83-480F-80EA-8CF98AD506DB

P2860

Q35757153-07BD3464-D721-4607-9E41-02B2AB99E8F3

Q35757153-0B10DBD3-C528-49EA-B42D-5FF3430B49C0

Q35757153-26C2F899-78D9-4CD2-9AA8-A665915383C5

Q35757153-29297074-3F47-4358-A2C7-34D0CD94ECF6

Q35757153-2C91672D-58BE-4A95-95DA-ABA703C5BC9D

Q35757153-2D4135D4-23CD-4FAD-A9CA-961C2F53ECE3

Q35757153-348FF0DC-8FC0-41A8-9A10-4D7CC4DF9936

Q35757153-38ABAE24-F8CF-4C19-A3DB-415586EFF9FB

Q35757153-3B1FDBDE-C95B-4A95-AED0-9B06BF639981

Q35757153-3DF616F9-9975-4755-800A-9C7587DD56F7

P304

Q35757153-9ABEEEE4-2072-419B-AD23-EA57D2D301EF

P31

Q35757153-0E971255-32A9-480E-A101-AB0DB7CF9A64

P356

Q35757153-33C8D060-22A0-49B6-8E0A-41777B401BF1

P407

Q35757153-C5D537E8-7158-4877-B84E-B3BEF3172A96

P433

Q35757153-84EB92C0-CEF8-480A-9D60-558BCA36C3C0

P478

Q35757153-767E1C5B-BB3A-4FCE-B014-858C859D3FE0

P577

Q35757153-6E81C73A-DB7A-4950-A461-F36F7F787101

P5875

Q35757153-D59D2D48-64F6-4033-BD2A-7BFA82616793

P698

Q35757153-66895632-4888-4460-928E-8799D53864E1

P921

Q35757153-E4607E11-9790-4677-B3A5-824CB8B553CF

P932

Q35757153-0323E5E5-2482-4B4D-BC22-2848D76EDD83

P356

AJPATH.2007.060481

P1433

The American Journal of Pathology

P1476

Deficiency of endothelial nitr ...... ropathy-resistant inbred mice.

@en

P2093

Keiko Takahashi

http://www.w3.org/2001/XMLSchema#string

Matthew D Breyer

http://www.w3.org/2001/XMLSchema#string

Maureen A Gannon

http://www.w3.org/2001/XMLSchema#string

Michio Nagata

http://www.w3.org/2001/XMLSchema#string

Raymond C Harris

http://www.w3.org/2001/XMLSchema#string

Takamune Takahashi

http://www.w3.org/2001/XMLSchema#string

Yukiko Kanetsuna

http://www.w3.org/2001/XMLSchema#string

P2860

Augmented BH4 by gene transfer restores nitric oxide synthase function in hyperglycemic human endothelial cells

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Protective role of endothelial nitric oxide synthase

The regulation and pharmacology of endothelial nitric oxide synthase

Hyperglycaemia-induced superoxide production decreases eNOS expression via AP-1 activation in aortic endothelial cells.

NAD(P)H oxidase and uncoupled nitric oxide synthase are major sources of glomerular superoxide in rats with experimental diabetic nephropathy.

Mechanisms underlying endothelial dysfunction in diabetes mellitus.

Impairment of vascular endothelial nitric oxide synthase activity by advanced glycation end products.

Charge selectivity of the glomerular filtration barrier in healthy and nephrotic humans.

Impaired nitric oxide-dependent cyclic guanosine monophosphate generation in glomeruli from diabetic rats. Evidence for protein kinase C-mediated suppression of the cholinergic response.

P304

1473-1484

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.2353/AJPATH.2007.060481

http://www.w3.org/2001/XMLSchema#string

P407

P433

5

http://www.w3.org/2001/XMLSchema#string

P478

170

http://www.w3.org/2001/XMLSchema#string

P577

2007-05-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

6371920

http://www.w3.org/2001/XMLSchema#string

P698

17456755

http://www.w3.org/2001/XMLSchema#string

P921

P932

1854944

http://www.w3.org/2001/XMLSchema#string