Crescentic glomerulonephritis in CD4- and CD8-deficient mice. Requirement for CD4 but not CD8 cells
about
Neutrophil-Mediated Regulation of Innate and Adaptive Immunity: The Role of MyeloperoxidaseCD4(+) T cells specific to a glomerular basement membrane antigen mediate glomerulonephritis.CCR7 deficiency exacerbates injury in acute nephritis due to aberrant localization of regulatory T cellsT cells, adhesion molecules and modulation of apoptosis in visceral leishmaniasis glomerulonephritis.P-selectin deficiency exacerbates experimental glomerulonephritis: a protective role for endothelial P-selectin in inflammation.Lack of chemokine receptor CCR1 enhances Th1 responses and glomerular injury during nephrotoxic nephritis.Toll-like receptor 9 enhances nephritogenic immunity and glomerular leukocyte recruitment, exacerbating experimental crescentic glomerulonephritis.The IL-27 receptor has biphasic effects in crescentic glomerulonephritis mediated through Th1 responses.Identification of a bone marrow-derived CD8αα+ dendritic cell-like population in inflamed autoimmune target tissue with capability of inducing T cell apoptosisLeptin-deficient mice are protected from accelerated nephrotoxic nephritisConditional ablation of macrophages halts progression of crescentic glomerulonephritis.Natural recovery from antiglomerular basement membrane glomerulonephritis is associated with glomeruli-infiltrating CD8α+CD11c+MHC class II+ cells.Local IL-17 Production Exerts a Protective Role in Murine Experimental Glomerulonephritis.Inflammation-Induced IL-6 Functions as a Natural Brake on Macrophages and Limits GN.Protease-activated receptor-2 augments experimental crescentic glomerulonephritisAntibodies against linear epitopes on the Goodpasture autoantigen and kidney injury.Protease-activated receptor 1 mediates thrombin-dependent, cell-mediated renal inflammation in crescentic glomerulonephritis.Major histocompatibility complex class II expression by intrinsic renal cells is required for crescentic glomerulonephritisImmature renal dendritic cells recruit regulatory CXCR6(+) invariant natural killer T cells to attenuate crescentic GN.T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.Renal dendritic cells stimulate IL-10 production and attenuate nephrotoxic nephritis.Spontaneous recovery from early glomerular inflammation is associated with resistance to anti-GBM glomerulonephritis: tolerance and autoimmune tissue injury.Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritisEfficient renal recruitment of macrophages and T cells in mice lacking the duffy antigen/receptor for chemokines.Loss of endogenous thymosin β4 accelerates glomerular disease.Inhibition of indoleamine 2, 3-dioxygenase-mediated tryptophan catabolism accelerates crescentic glomerulonephritis.Signal transducer and activation of transcription 6 (STAT6) regulates T helper type 1 (Th1) and Th17 nephritogenic immunity in experimental crescentic glomerulonephritis.
P2860
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P2860
Crescentic glomerulonephritis in CD4- and CD8-deficient mice. Requirement for CD4 but not CD8 cells
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@ast
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@en
type
label
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@ast
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@en
prefLabel
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@ast
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@en
P2093
P2860
P1476
Crescentic glomerulonephritis ...... ment for CD4 but not CD8 cells
@en
P2093
P2860
P304
P407
P577
1998-06-01T00:00:00Z