Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB. - Wikidata - awgldk - TriplyDB

Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB.

Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB.

http://www.wikidata.org/entity/Q35781348

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Breaking the mold: transcription factors in the anucleate platelet and platelet-derived microparticles The aryl hydrocarbon receptor complex and the control of gene expression Neu-164 and Neu-107, two novel antioxidant and anti-myeloperoxidase compounds, inhibit acute cigarette smoke-induced lung inflammation Adverse effects of wood smoke PM(2.5) exposure on macrophage functions Activation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering Mechanisms SIRT1 is a redox-sensitive deacetylase that is post-translationally modified by oxidants and carbonyl stress TCDD and a putative endogenous AhR ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts.Protein kinase C zeta mediates cigarette smoke/aldehyde- and lipopolysaccharide-induced lung inflammation and histone modifications.Cigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.The glutaredoxin/S-glutathionylation axis regulates interleukin-17A-induced proinflammatory responses in lung epithelial cells in association with S-glutathionylation of nuclear factor κB family proteins.Dioxin may promote inflammation-related development of endometriosis.Aryl hydrocarbon receptor targets pathways extrinsic to bone marrow cells to enhance neutrophil recruitment during influenza virus infection Effects of cigarette smoke on the activation of oxidative stress-related transcription factors in female A/J mouse lung.Alterations in the expression of the NF-κB family member RelB as a novel marker of cardiovascular outcomes during acute exacerbations of chronic obstructive pulmonary disease.Cigarette smoke induced airway inflammation is independent of NF-κB signalling A novel anti-inflammatory and pro-resolving role for resolvin D1 in acute cigarette smoke-induced lung inflammation Cigarette smoke exacerbates mouse allergic asthma through Smad proteins expressed in mast cells.Aryl hydrocarbon receptor-dependent retention of nuclear HuR suppresses cigarette smoke-induced cyclooxygenase-2 expression independent of DNA-binding.Lung-targeted overexpression of the NF-κB member RelB inhibits cigarette smoke-induced inflammation AhR deficiency impairs expression of LPS-induced inflammatory genes in mice Impact of cigarette smoke on the human and mouse lungs: a gene-expression comparison study.Ocular fibroblast diversity: implications for inflammation and ocular wound healing Deletion of the aryl hydrocarbon receptor enhances the inflammatory response to Leishmania major infection Decreased expression of the NF-κB family member RelB in lung fibroblasts from Smokers with and without COPD potentiates cigarette smoke-induced COX-2 expression.Genetic ablation of the aryl hydrocarbon receptor causes cigarette smoke-induced mitochondrial dysfunction and apoptosis Aryl hydrocarbon receptor is necessary to protect fetal human pulmonary microvascular endothelial cells against hyperoxic injury: Mechanistic roles of antioxidant enzymes and RelB Aryl Hydrocarbon Receptor-Mediated Perturbations in Gene Expression during Early Stages of CD4(+) T-cell Differentiation.Resolvins attenuate inflammation and promote resolution in cigarette smoke-exposed human macrophages.Omeprazole Attenuates Pulmonary Aryl Hydrocarbon Receptor Activation and Potentiates Hyperoxia-Induced Developmental Lung Injury in Newborn Mice Leflunomide Induces Pulmonary and Hepatic CYP1A Enzymes via Aryl Hydrocarbon Receptor.Aryl Hydrocarbon Receptor Protects Lungs from Cockroach Allergen-Induced Inflammation by Modulating Mesenchymal Stem Cells.VEGFR-2 inhibition augments cigarette smoke-induced oxidative stress and inflammatory responses leading to endothelial dysfunction Omeprazole does not Potentiate Acute Oxygen Toxicity in Fetal Human Pulmonary Microvascular Endothelial Cells Exposed to Hyperoxia.Resolvin D1 Reduces Emphysema and Chronic Inflammation.Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infection.Genetic ablation of NADPH oxidase enhances susceptibility to cigarette smoke-induced lung inflammation and emphysema in mice.Functional deficiency of aryl hydrocarbon receptor augments oxygen toxicity-induced alveolar simplification in newborn mice Aryl Hydrocarbon Receptor Plays Protective Roles against High Fat Diet (HFD)-induced Hepatic Steatosis and the Subsequent Lipotoxicity via Direct Transcriptional Regulation of Socs3 Gene Expression High-dose but not low-dose mainstream cigarette smoke suppresses allergic airway inflammation by inhibiting T cell function MicroRNA-375 regulation of thymic stromal lymphopoietin by diesel exhaust particles and ambient particulate matter in human bronchial epithelial cells

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Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB.

http://www.wikidata.org/entity/Q35781348

description

2007 nî lūn-bûn

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2007年論文

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2007年論文

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Aryl hydrocarbon receptor-defi ...... factor-kappaB component RelB.

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The American Journal of Pathology

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Aryl hydrocarbon receptor-defi ...... factor-kappaB component RelB.

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Carolyn J Baglole

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Heather F Lakatos

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Patricia J Sime

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Richard P Phipps

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Sanjay B Maggirwar

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Thomas A Gasiewicz

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Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

The RelA NF-kappaB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c-myc promoter in mammary cells

LPS induces pulmonary intravascular macrophages producing inflammatory mediators via activating NF-kappaB

The anti-inflammatory sesquiterpene lactone helenalin inhibits the transcription factor NF-kappaB by directly targeting p65

Differential activation of MAP kinase signaling pathways and nuclear factor-kappaB in bronchoalveolar cells of smokers and nonsmokers

Neurotrophins prevent HIV Tat-induced neuronal apoptosis via a nuclear factor-kappaB (NF-kappaB)-dependent mechanism

Regulation of tumor necrosis factor alpha transcription in macrophages: involvement of four kappa B-like motifs and of constitutive and inducible forms of NF-kappa B

Signaling to NF-kappaB

Aryl hydrocarbon receptor signaling plays a significant role in mediating benzo[a]pyrene- and cigarette smoke condensate-induced cytogenetic damage in vivo.

The aromatic hydrocarbon receptor, transcription, and endocrine aspects of dioxin action.

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