Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression - Wikidata - awgldk - TriplyDB

Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression

Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression

http://www.wikidata.org/entity/Q35783020

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Crosstalk of HNF4α with extracellular and intracellular signaling pathways in the regulation of hepatic metabolism of drugs and lipids Melanocortin receptors in rat liver cells: change of gene expression and intracellular localization during acute-phase response Hepatocyte expression of serum response factor is essential for liver function, hepatocyte proliferation and survival, and postnatal body growth in mice In vivo targeting of the growth hormone receptor (GHR) Box1 sequence demonstrates that the GHR does not signal exclusively through JAK2.STAT5 requires the N-domain for suppression of miR15/16, induction of bcl-2, and survival signaling in myeloproliferative disease STAT5-glucocorticoid receptor interaction and MTF-1 regulate the expression of ZnT2 (Slc30a2) in pancreatic acinar cells Minireview: mechanisms of growth hormone-mediated gene regulation.The different functions of Stat5 and chromatin alteration through Stat5 proteins Stat5 regulates cellular iron uptake of erythroid cells via IRP-2 and TfR-1.Alternative ways of modulating JAK-STAT pathway: Looking beyond phosphorylation.Diverse gene reprogramming events occur in the same spatial clusters of distal regulatory elements Context-Specific Growth Hormone Signaling through the Transcription Factor STAT5: Implications for the Etiology of Hepatosteatosis and Hepatocellular Carcinoma.Mapping the growth hormone--Stat5b--IGF-I transcriptional circuit.The biology of progesterone receptor in the normal mammary gland and in breast cancer Hepatic growth hormone and glucocorticoid receptor signaling in body growth, steatosis and metabolic liver cancer development.Growth hormone-STAT5 regulation of growth, hepatocellular carcinoma, and liver metabolism.STAT5 requires the N-domain to maintain hematopoietic stem cell repopulating function and appropriate lymphoid-myeloid lineage output.Interpretation of cytokine signaling through the transcription factors STAT5A and STAT5B GH/STAT5 signaling during the growth period in livers of mice overexpressing GH.Genomic dissection of the cytokine-controlled STAT5 signaling network in liver Hepatocyte nuclear factor-1alpha regulates glucocorticoid receptor expression to control postnatal body growth.The hormonal action of IGF1 in postnatal mouse growth.Dynamic trafficking of STAT5 depends on an unconventional nuclear localization signal The influence of estrogens on the biological and therapeutic actions of growth hormone in the liver.Interaction between the glucocorticoid and erythropoietin receptors in human erythroid cells Sex differences in the expression of hepatic drug metabolizing enzymes.Lineage-Specific and Non-specific Cytokine-Sensing Genes Respond Differentially to the Master Regulator STAT5.The dual role of LSD1 and HDAC3 in STAT5-dependent transcription is determined by protein interactions, binding affinities, motifs and genomic positions.An N-terminal splice variant of human Stat5a that interacts with different transcription factors is the dominant form expressed in invasive ductal carcinoma.Liver-specific GH receptor gene-disrupted (LiGHRKO) mice have decreased endocrine IGF-I, increased local IGF-I, and altered body size, body composition, and adipokine profiles Body size regulation and insulin-like growth factor signaling.Adipocyte STAT5 deficiency promotes adiposity and impairs lipid mobilisation in mice.The Interactome of the Glucocorticoid Receptor and Its Influence on the Actions of Glucocorticoids in Combatting Inflammatory and Infectious Diseases.Glucocorticoid augments lipopolysaccharide-induced activation of the IκBζ-dependent genes encoding the anti-microbial glycoproteins lipocalin 2 and pentraxin 3.STAT5-Interacting Proteins: A Synopsis of Proteins that Regulate STAT5 Activity.Impairment of hepatic growth hormone and glucocorticoid receptor signaling causes steatosis and hepatocellular carcinoma in mice.Disruption of the growth hormone--signal transducer and activator of transcription 5--insulinlike growth factor 1 axis severely aggravates liver fibrosis in a mouse model of cholestasis.Epo-induced erythroid maturation is dependent on Plcγ1 signaling.Lung epithelial tip progenitors integrate glucocorticoid- and STAT3-mediated signals to control progeny fate.Hepatic Deletion of Janus Kinase 2 Counteracts Oxidative Stress in Mice.

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Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression

http://www.wikidata.org/entity/Q35783020

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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name

Direct glucocorticoid receptor ...... ration-related gene expression

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Direct glucocorticoid receptor ...... ration-related gene expression

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type

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Direct glucocorticoid receptor ...... ration-related gene expression

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Direct glucocorticoid receptor ...... ration-related gene expression

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Direct glucocorticoid receptor ...... ration-related gene expression

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P1433

Genes & Development

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Direct glucocorticoid receptor ...... ration-related gene expression

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P2093

Andreas Reimann

http://www.w3.org/2001/XMLSchema#string

David Engblom

http://www.w3.org/2001/XMLSchema#string

Francois Tronche

http://www.w3.org/2001/XMLSchema#string

Günther Schütz

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Hartmut Beug

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Lukas Schwake

http://www.w3.org/2001/XMLSchema#string

P2860

MAPPFinder: using Gene Ontology and GenMAPP to create a global gene-expression profile from microarray data

Cellular localization and regulation of gene expression for components of the insulin-like growth factor ternary binding protein complex

Functional interactions between Stat5 and the glucocorticoid receptor

Stat5a and Stat5b proteins have essential and nonessential, or redundant, roles in cytokine responses

In vivo analysis of growth hormone receptor signaling domains and their associated transcripts.

Clarifying the role of Stat5 in lymphoid development and Abelson-induced transformation.

Circulating levels of IGF-1 directly regulate bone growth and density.

Stat5a/b are essential for normal lymphoid development and differentiation.

DNA binding of the glucocorticoid receptor is not essential for survival.

Liver-derived insulin-like growth factor I (IGF-I) is the principal source of IGF-I in blood but is not required for postnatal body growth in mice

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1157-1162

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10.1101/GAD.426007

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10

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21

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Richard Moriggl

Jan-Wilhelm Kornfeld

Lothar Hennighausen

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2007-05-01T00:00:00Z

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6327333

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17504935

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1865487

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